جست‌وجوی استاد و پژوهشگر

• Neurodegenerative Diseases
• Protein Aggregation
• Molecular Biology
• +۳ مورد دیگر

Sonali Vishal is an Associate Research Scientist in the Department of Pathology at Yale School of Medicine, Yale University. Her primary appointment is in Pathology with affiliations to the Gopal Lab. She focuses on neurodegenerative diseases, protein aggregation mechanisms, and cellular signaling pathways. Her work integrates molecular biology techniques with cellular models to study TDP-43 condensates, mRNA transport dysfunction, and aging-related protein kinases. Research interests center on neurodegenerative disorders like ALS and frontotemporal dementia, with emphasis on RNP condensate dynamics, axonal transport disruptions, and phase separation phenomena. Her studies also explore anti-aging mechanisms involving AMPK and sirtuins, alongside foundational work on desmosome assembly regulated by 14-3-3γ proteins. Recent publications analyze pathological transitions in neuronal RNP condensates and their role in disease progression. Her work bridges molecular mechanisms with clinical implications for neurodegenerative diseases. Active collaborations include studies with Pallavi Gopal and other Yale investigators in neuroscience and cell biology. Lab affiliations include the Gopal Lab, where she contributes to interdisciplinary research combining biochemistry, cell biology, and neurology. Her research portfolio reflects deep engagement with both fundamental cellular processes and translational disease mechanisms.

• Chondrocyte Biology
• High Fat Food Research
• Sirtuin Research
• +۵ مورد دیگر

Dawid Makosa is a Doctor of Philosophy Student and Research Associate at the UWA Medical School, University of Western Australia, affiliated with the UWA Centre for Medical Research and the Harry Perkins Institute of Medical Research. His research focuses on chondrocyte biology, osteoarthritis pathogenesis, and metabolic effects of high-fat diets on joint health. His primary research interests include: Chondrocyte Biology (100% research focus) High Fat Food effects (92% research focus) Sirtuin proteins (92% research focus) Osteoarthritis mechanisms (74% research focus) Mitochondrial Respiration in joint tissues (51% research focus) Makosa's publication record reveals a strong emphasis on understanding how sirtuin proteins regulate chondrocyte function and how metabolic disorders contribute to osteoarthritis development. His work employs mouse models to investigate sexually dimorphic responses to high-fat diets and examines molecular mechanisms linking obesity to joint degeneration. Recent publications in the Journal of Bone and Mineral Research and Cartilage demonstrate significant impact in the field, with his 2022 paper on Sirt3 accumulating 28 Scopus citations. His doctoral thesis, 'Development of new single-cell genomics tools to reveal the epigenome and transcriptome dynamics in the human brain across ageing' (2025, unpublished), indicates expanding research interests into neuroscience. Makosa's work contributes to UN Sustainable Development Goals related to good health and well-being, with datasets openly shared through Figshare to promote scientific reproducibility.

Sebastian Greissمشاهده پروفایل

پژوهشگر ارشد

• Genetic Code Expansion
• Neurobiology
• Unnatural Amino Acids
• +۴ مورد دیگر

Dr. Sebastian Greiss serves as a Chancellor's Fellow at the Centre for Integrative Physiology within the College of Medicine and Veterinary Medicine at the University of Edinburgh. His research focuses on developing and applying unnatural amino acid technology to study neuronal circuits in C. elegans , addressing fundamental questions about nervous system function. PhD, University of Dundee (2003-2008) Diploma in Biochemistry, University of Bayreuth (2002) Greiss pioneers genetic code expansion in multicellular organisms, creating light-activatable proteins through photo-caged amino acids to precisely control neuronal activity in freely moving C. elegans . His work bridges molecular engineering and systems neuroscience , using the worm's compact 302-neuron nervous system to decode how sensory inputs transform into behavioral outputs. Key innovations include tissue-specific proteomics and optogenetic control at single-protein resolution. His publication record shows consistent advancement in genetic code expansion from 2008-2014, transitioning from foundational work on sirtuins and apoptosis to breakthroughs in C. elegans and Drosophila genetic engineering. The research demonstrates increasing technical sophistication, moving from in vitro studies to in vivo applications in complex organisms with featured publications in Nature Biotechnology and Nature Chemical Biology . Greiss leads an active research group funded by the European Research Council, mentoring PhD students and postdoctoral fellows. His collaborative network spans the MRC Laboratory of Molecular Biology in Cambridge, Technical University Munich, and multiple Edinburgh research centers. Current work focuses on applying unnatural amino acid technology to dissect neuronal circuit function with unprecedented precision. Zhiyan Xi (PhD Student) Lloyd Davis (Postdoctoral Fellow) Andromachi Xypnitou (Research Associate)

Dr. Mirjam Knopمشاهده پروفایل

پژوهشگر

• Gut-microbiome interactions
• Immunity in Drosophila
• FoxO-dependent effects on gut renewal
• +۲ مورد دیگر

Dr. Mirjam Knop is a postdoctoral researcher at the Department of Molecular Physiology at Kiel University , Germany. She works in the laboratory of Prof. Dr. Thomas Roeder and focuses on gut-microbiome interactions , immunity , and FoxO-dependent effects on gut renewal using Drosophila melanogaster as a model organism. Her research connects: Gut epithelial homeostasis Microbiome dynamics FoxO signaling pathways Stress response mechanisms Host-microbe interactions Organ remodeling processes Recent publications highlight her work on: Mitochondrial sirtuin regulation of microbiota Tracheal system molecular architecture Sex-specific lifespan extension Bacteria-temperature interactions in Hydra Environmental adaptation in metaorganisms Notable awards include: DFG funding for the Excellence Cluster „Inflammation at interfaces“ She has developed key methodologies like Drosophila fecal sampling and contributed to understanding gene-environment interactions in invertebrates. Her work spans immunology , microbial ecology , and developmental physiology across multiple model systems.

B. Mark Eversمشاهده پروفایل

استاد

• Gastrointestinal Surgery
• Oncology
• Cancer Biology
• +۳ مورد دیگر

B. Mark Evers is a Professor of Surgery at the University of Kentucky , serving as the Director of the Markey Cancer Center and Mark McDowell Cancer Foundation Chair. He specializes in gastrointestinal surgery and oncology, with a focus on cancer biology and metabolic disorders. Dr. Evers holds the prestigious Fellow of the American College of Surgeons (FACS) distinction. Education: MD from the University of Tennessee, Memphis (summa cum laude). Completed residency at the University of Louisville and a fellowship in surgical research at the University of Texas Medical Branch. Research: Dr. Evers' work spans tumor metabolism, intestinal cell biology, and the role of neurotensin in cancer progression. Key areas include glycogen-driven lung adenocarcinoma initiation, neurotensin's impact on atherosclerosis, and radiosensitization strategies for pancreatic neuroendocrine tumors. His studies also address geographic disparities in cancer outcomes, particularly in Appalachian Kentucky. Leadership Roles: Physician-In-Chief, Oncology Service Director, Markey Cancer Center Member of the MD/PhD Program Internal Advisory Committee Key Contributions: Over 160 peer-reviewed publications, including high-impact studies on tumor microenvironment modulation and metabolic reprogramming in cancer therapy. Active in translational research to bridge molecular insights with clinical practice.

Bjoern Schwerمشاهده پروفایل

استاد پژوهشی

• DNA Repair
• Chromatin Regulation
• Transcriptional Regulation
• +۶ مورد دیگر

Bjoern Schwer is a Research Professor in the Department of Medicine at the University of California San Francisco (UCSF). His research focuses on the interplay between DNA repair, chromatin regulation, and transcriptional control in neural stem/progenitor cells, with a particular emphasis on neurodevelopment, brain function, and diseases such as medulloblastoma. He leads a laboratory investigating genome instability and chromosomal rearrangements in neural progenitors. Education: MD from University of Heidelberg (2003), PhD in Tumor Virology from University of Heidelberg (2005) Postdoctoral training: Mitochondrial metabolism and Sirtuin biology at UCSF Gladstone Institute (2007), DNA repair and Chromatin biology at Harvard/Boston Children's Hospital (2010) Research Interests Mechanisms of DNA double-strand breaks in neural cells Chromatin accessibility and gene expression in brain tumors Role of PRDM6 in medulloblastoma formation Genomic instability in neurodegeneration and aging Publication Trends Recent work highlights PRDM6 as a chromatin repressor in medulloblastoma, DNA repair mechanisms in neural aging, and innovative models for studying brain tumor development. His studies span neuro-oncology, epigenetics, and DNA damage responses. Scientific Awards DAAD Biomedical Exchange Fellowship (2001) Heidelberg Doctoral 'summa cum laude' (2005) Sandler Postdoctoral Fellowship (2006) Ellison Medical Foundation Fellowship (2008) NIH Career Development Award (2012) V Foundation Scholar Award (2013) UCSF Brain Tumor SPORE Award (2016-2017) Haderle Endowed Chair (2017) Kimmel Scholar (2019) Ludwig Award for Early Career Research (2021) Bakar Aging Research Institute Investigator (2021) Labs & Teams Schwer leads a research group at UCSF Department of Medicine, collaborating with neuro-oncology and chromatin biology teams. His lab uses advanced models like neural blastocyst complementation to study brain development and tumorigenesis.

• Bone Biology
• Skeletal Aging
• Osteoporosis
• +۵ مورد دیگر

Maria Schuller De Almeida is a Professor at the University of Arkansas for Medical Sciences in the College of Medicine. She holds dual appointments in the Department of Internal Medicine (Endocrinology Division) and the Department of Orthopaedics, reflecting her interdisciplinary research focus on bone biology and skeletal health. Dr. Almeida's research primarily focuses on the molecular mechanisms underlying skeletal aging, osteoporosis, and the effects of sex hormones on bone metabolism. Her work investigates how oxidative stress, cellular senescence, and mitochondrial dysfunction contribute to age-related bone loss, with particular emphasis on the role of NAD+ metabolism and FoxO transcription factors in bone cells. She explores how estrogen signaling regulates osteoclast and osteoblast function through mitochondrial pathways. Analysis of Dr. Almeida's recent publications reveals a strong focus on the intersection of bone biology, endocrinology, and aging. Her research consistently examines how estrogen signaling affects bone cell function, with increasing attention to mitochondrial metabolism and NAD+ pathways in skeletal health. This work has important implications for understanding and treating osteoporosis and other age-related bone disorders, particularly in postmenopausal women. R01AR082418: "Antagonism of RANKL signaling by estrogen in osteoclasts" (2024-2029) 2R56AR056679: "Mechanisms of decreased bone formation with aging" (2021-2024) 1R01AG068449: "Different consequences of cellular aging in cortical versus cancellous bone" (2021-2025) R01AR056679: "Role of FoxOs in Skeletal Homeostasis" (2008-2022) Dr. Almeida serves as Principal Investigator on multiple NIH R01 grants and as Co-Investigator on the Center for Musculoskeletal Disease Research (CMDR), a P20 grant from NIGMS (2023-2028). Her laboratory investigates how sex steroids protect bone through mitochondrial mechanisms and how aging affects bone formation at cellular and molecular levels.

Antiño R. Allenمشاهده پروفایل

استاد

• Radiation Biology
• Neuroscience
• Cognitive Neuroscience
• +۵ مورد دیگر

Antiño R. Allen is a Professor in the Department of Pharmaceutical Sciences at the University of Arkansas for Medical Sciences (UAMS) College of Pharmacy. He serves as the Associate Dean of Pipeline and Career Development for the Graduate School and is a member of the Division of Radiation Health. His work bridges radiation biology and neuroscience, focusing on the impact of radiation and trauma on cognitive function. Education: BS, Jackson State University, Jackson, MS MA, Indiana University, Bloomington, IN PhD in Evolution, Ecology & Behavior, Indiana University, Bloomington, IN Post-Doctoral Fellow, University of California, San Francisco Dr. Allen's research program investigates the mechanisms by which inflammation and oxidative stress alter neuronal structure and cognitive performance after exposure to ionizing radiation (including space-relevant heavy ions and galactic cosmic rays) or traumatic brain injury. His recent work also explores: Cardiac and bone effects of multi-ion space radiation Mitochondrial dysfunction in radiation-induced cognitive deficits Neuroprotective strategies against chemotherapy-induced cognitive impairment Analysis of his 2021-2023 publications shows a cohesive research trajectory in space radiation biology, utilizing murine models to assess multi-organ impacts (brain, heart, bone) and identify molecular targets for countermeasures. His work increasingly integrates mitochondrial metabolism and oxidative stress pathways. Dr. Allen leads an active research laboratory that has mentored summer students and produced alumni researchers. He contributes to graduate education through the UAMS Initiative for Maximizing Student Development (IMSD) and teaches courses in drug action and molecular biotechnology.

Lindsay Edward Wuمشاهده پروفایل

دانشیار

• Biology of Ageing
• Cell Metabolism
• NAD+ Biochemistry
• +۴ مورد دیگر

Associate Professor Lindsay Edward Wu is based at the University of New South Wales within the Faculty of Medicine & Health. He leads the Laboratory for Ageing Research, which focuses on fundamental mechanisms and interventions to maintain healthy ageing with immediate applications in fertility preservation, cancer treatment side effects, and understanding age-related susceptibility to infections like coronavirus. Faculty of Medicine & Health, University of New South Wales Principal Investigator, Laboratory for Ageing Research NHMRC Career Development Fellow Dr. Wu's educational background includes a BSc (Hons I) from the University of Sydney and a PhD from UNSW. His research bridges basic science with clinical applications, particularly in the areas of NAD+ metabolism, reproductive ageing, and age-related disease susceptibility. His research interests span multiple interconnected fields including the biology of ageing, cell metabolism, NAD+ biochemistry, reproductive ageing, and age-related infertility. The laboratory employs molecular biology, animal physiology, metabolomics and bioinformatics to investigate fundamental mechanisms of ageing and develop interventions. Current projects focus on new treatments to maintain female fertility during biological ageing and following chemotherapy treatment, the role of the pentose phosphate pathway in reproductive ageing, DNA transposons in genome instability, and NAD+ metabolism in age-related susceptibility to coronavirus infection. Analysis of Dr. Wu's publication record reveals a strong focus on translational ageing research with particular emphasis on NAD+ metabolism, reproductive ageing, and interventions to preserve health during ageing. His work consistently connects fundamental biochemical mechanisms with potential clinical applications, particularly in fertility preservation and cancer treatment side effects. NHMRC Career Development Fellowship (RD Wright Biomedical Fellowship) APP1127821 (2017-2020) NHMRC Project Grant APP1139763 "Translating new advances in oncofertility" (2018-2021) NHMRC Development Grant APP1122484 "NAD+ raising agents for improving oocyte quality" (2017-2019) Dr. Wu has maintained consistent NHMRC funding since 2015 and has successfully supervised students in areas including biology of ageing, age-related infertility, cell metabolism, and NAD+ biochemistry. His Laboratory for Ageing Research employs multidisciplinary approaches to address complex questions about the biological mechanisms of ageing and develop practical interventions. The lab's work has significant implications for improving health outcomes in older populations, particularly regarding reproductive health and resilience to diseases like cancer and viral infections.

Roman V. Kondratovمشاهده پروفایل

استاد

• Circadian Biology
• Aging Research
• Molecular Biology
• +۳ مورد دیگر

Roman V. Kondratov is a Professor and Associate Vice President for Research at Cleveland State University, leading the Kondratov Laboratory within the Center for Gene Regulation in Health and Disease (GRHD). His research establishes critical links between circadian biology and aging mechanisms. Research Focus: Dr. Kondratov pioneered the discovery that BMAL1 deficiency causes accelerated aging in mice (9 vs. 28-month lifespan), manifesting as sarcopenia, osteoporosis, and cataracts. His current work investigates circadian-metabolic crosstalk through mTOR, Sirtuin, and IGF/IGFR pathways, dietary restriction timing effects, and circadian-stem cell interactions in osteoporosis. His lab employs genetic models and molecular techniques to dissect aging mechanisms. Publication Trends: Recent work (2016-2021) reveals how caloric restriction reprograms diurnal metabolism, with time-of-day-dependent effects on glucose homeostasis, acetyl-CoA metabolism, and mTOR signaling. His research demonstrates that functional circadian clocks are essential for dietary restriction's lifespan-extending effects. Advising and Lab: Dr. Kondratov mentors active trainees including Oghogho Patrick Ebeigbe, Artem Astafev, and Jonathan Fritz. Alumni such as Rohini Khapre (Cleveland Clinic) and Sonal Patel (Research Scientist) have advanced to prominent research roles. His lab operates in the Science and Research Building (SR 270/PH 210), focusing on circadian regulation of aging and metabolism.

Mohd Adnanمشاهده پروفایل

دانشیار

• Phytomedicine
• Biofilms
• Drug Discovery
• +۱۲ مورد دیگر

Mohd Adnan is an Associate Professor, Principal Investigator, and Head of Academic Committee at the Department of Biology, College of Science, University of Ha'il, Saudi Arabia. With over 14 years of research, teaching, and administrative experience, he has established himself as a prominent researcher in natural products and microbiology. Dr. Adnan earned his PhD from University of Central Lancashire, UK; Master's and Bachelor's degrees from Bangalore University, India; and a Post Graduate Diploma in Bioinformatics from SJAIT, India. His research focuses on phytomedicine, biofilms, drug discovery, natural products, nutraceuticals, and functional foods. He specializes in plant-based antibiofilm and anticancer agents, microbial biosurfactants, biofilms in food industry and medical settings, probiotics, and cancer biology. His work spans ecology, marine biology, microbiology, green chemistry, agricultural science, food science, biotechnology, and molecular biology. Analysis of his recent publications reveals a strong focus on computational approaches to drug discovery, cancer biology, and antimicrobial research. His work frequently combines traditional natural product chemistry with modern bioinformatics and machine learning techniques, particularly in identifying novel therapeutic compounds and understanding their mechanisms of action. Travel grants as Principal Investigator Observership grants Research grants from prestigious organizations Dr. Adnan has successfully published over 250 papers in internationally recognized journals and has reviewed more than 400 manuscripts for over 70 peer-reviewed journals. He serves in various editorial capacities including Associate, Academic, Guest, and Review Editor positions in esteemed journals, having edited over 500 manuscripts. He is a member of the Society for Applied Microbiology (SFAM), UK; the European Society of Clinical Microbiology and Infectious Diseases (ESCMID), Switzerland; and an Elected Member of the Royal Society of Biology, UK.

Andrew C. Hsiehمشاهده پروفایل

استاد

• Protein synthesis in cancer
• Prostate cancer
• Bladder cancer
• +۵ مورد دیگر

Dr. Andrew C. Hsieh is a distinguished physician-scientist holding dual appointments at Fred Hutch Cancer Center and University of Washington. At Fred Hutch, he serves as Professor and Associate Director of the Human Biology Division, Professor of the Clinical Research Division, and maintains a professorship in the Division of Hematology and Oncology at the University of Washington School of Medicine. His clinical practice focuses on genitourinary cancers at Fred Hutch Cancer Center at UW Medical Center – Montlake, where he treats patients with prostate and bladder cancers while balancing clinical care with laboratory research. Dr. Hsieh earned his medical degree from Albert Einstein College of Medicine and completed both residency and fellowship training at University of California, San Francisco. He is board-certified in Medical Oncology and Internal Medicine by the American Board of Internal Medicine. His educational background provided the foundation for his dual career path as both clinician and researcher, allowing him to bridge discoveries from bench to bedside. Dr. Hsieh's research program centers on understanding how cancer cells hijack protein synthesis mechanisms to drive tumor growth and progression. The Hsieh laboratory investigates the deregulation of protein synthesis control in epithelial cells during the multi-step process of tumor initiation and progression, with particular emphasis on prostate and bladder cancers. His team aims to merge fundamental discoveries in translational control biology with the clinical needs of cancer patients, developing novel therapeutic approaches that selectively target cancer-specific protein synthesis pathways while sparing normal cells. This work has significant implications for overcoming therapy resistance in advanced cancers. Analysis of Dr. Hsieh's extensive publication record reveals a consistent research trajectory focused on translational control mechanisms in cancer biology. His work spans from basic molecular mechanisms of protein synthesis regulation to clinical applications in cancer treatment, with an increasing emphasis on precision medicine approaches for genitourinary malignancies. Key research themes include understanding therapy resistance mechanisms, identifying novel druggable targets in protein synthesis pathways, and developing biomarker-driven treatment strategies for prostate and bladder cancers. His collaborative approach is evident in numerous multidisciplinary studies involving clinical data analysis, molecular biology techniques, and translational research methodologies. $1 million grant for London-Seattle collaboration to develop new therapies for drug-resistant prostate cancer Multiple high-impact publications in journals including Nature, Cell, Cancer Cell, and Nature Communications Active participation in clinical trials for novel cancer therapeutics Recognition as a leading expert in translational control mechanisms in cancer As a physician-scientist, Dr. Hsieh mentors numerous trainees and collaborates extensively with both basic science and clinical researchers. His laboratory at Fred Hutch employs a multidisciplinary approach that integrates molecular biology, genomics, and clinical research to address fundamental questions about how cancer cells co-opt protein synthesis machinery. The Hsieh lab has developed innovative methodologies including CRISPR screening approaches to understand RNA-binding proteins in cancer, and has pioneered research on how transcriptional-translational conflicts serve as barriers to cellular transformation. Dr. Hsieh's commitment to translating basic discoveries into clinical applications is evident in his active involvement in clinical trials and his focus on developing novel therapeutic strategies for patients with advanced genitourinary cancers.

John M Seubertمشاهده پروفایل

دانشیار مدعو

• Cardioprotection
• Epoxyeicosatrienoic Acids (EETs)
• Mitochondrial Dynamics
• +۴ مورد دیگر

John M Seubert serves as an Adjunct Associate Professor in the Department of Pharmacology at the University of Alberta's Faculty of Pharmacy and Pharmaceutical Sciences. His research program investigates cytochrome P450-derived metabolites of arachidonic acid, particularly epoxyeicosatrienoic acids (EETs), in cardiac protection and mitochondrial regulation. His educational background includes: BSc Biology (Toxicology) and Psychology Minor, Simon Fraser University (1992) MSc Biology - Aquatic Toxicology, Simon Fraser University (1997) PhD Pharmacology and Toxicology, University of Western Ontario (2002) Dr. Seubert's research focuses on two primary areas: (1) The cardioprotective mechanisms of EETs in ischemia-reperfusion injury, utilizing transgenic mouse models with elevated EET levels and cardiomyocyte experiments; (2) EET-mediated regulation of mitochondrial dynamics and energetics in cellular survival pathways. His laboratory employs techniques including murine cardiac injury models, mitochondrial function assays, and lipid mediator analysis. Analysis of his recent publications reveals strong emphasis on translational cardioprotection, with recurring themes in EET pharmacology, mitochondrial integrity during stress, and therapeutic targeting of soluble epoxide hydrolase. His work bridges molecular cardiology with pharmacological intervention strategies. Professional activities include teaching PHARM367 and PHARM566 courses, with laboratory operations based in the Katz Group Centre (Room 2-020M). Current research directions involve sEH inhibition strategies and mitochondrial protection pathways.

• Exercise Physiology
• Muscle Biology
• Aging and Oxidative Stress
• +۳ مورد دیگر

Summary John Lawler is an Associate Professor in the Department of Health and Kinesiology at Texas A&M University since 1997. His research focuses on exercise physiology, muscle biology, and aging-related mechanisms, with particular interest in oxidative stress, skeletal muscle atrophy, and cardiac remodeling. He holds a Ph.D. in Exercise Physiology from the University of Florida (1991), M.S. from Louisiana State University (1987), and B.S. in Biomedical Engineering from Duke University (1981). Lawler’s work spans over 100 peer-reviewed publications, including studies on NADPH oxidase (Nox2) regulation in muscle dystrophy, exercise countermeasures for disuse atrophy, and aging heart protection. He has secured over $10M in federal grants (NASA, NIH, DOD) and serves as a Fellow of the American College of Sports Medicine. Key Research: Muscle atrophy mechanisms, oxidative stress pathways, exercise interventions, cardiac aging Recent Projects: Targeted antioxidant therapies for microgravity muscle loss, Nox2 inhibition in dystrophic muscles Leadership: ACSM Research Advisory Council, multiple grant review panels

Rommel Mathiasمشاهده پروفایل

استاد مدعو

• Virology
• Molecular Biology
• Proteomics
• +۳ مورد دیگر

Rommel Mathias is an Adjunct Professor in the Department of Microbiology at Monash University, affiliated with the Monash Biomedicine Discovery Institute. His research focuses on the pathogenesis of human cytomegalovirus (HCMV), particularly the mechanisms of viral assembly, egress, and host organelle remodelling. He employs a multidisciplinary approach combining virology, molecular biology, proteomics, and bioinformatics to elucidate critical host-viral interactions and viral strategies for immune evasion. Research Interests: HCMV latency and reactivation, antiviral therapeutic targets, host-pathogen interactions, and molecular mechanisms of viral egress. Grants: Blocking Human Cytomegalovirus: Targeting Host Organelle Remodelling and the Viral Assembly Complex (NHMRC, 2016–2018) Sirtuins and Pathogenesis of Human Cytomegalovirus (NHMRC, 2012–2016) Labs/Teams: Leads the Viral Pathogenesis and Host Cellular Defense Group, collaborating internationally on HCMV research and bacterial pathogenesis.