جست‌وجوی استاد و پژوهشگر

Heidi Fullerمشاهده پروفایل

استاد

• Neuromuscular Disorders
• Proteomics
• Bioinformatics
• +۴ مورد دیگر

Professor Heidi Fuller serves as Head of School for Pharmacy and Bioengineering and Professor of Medical Science at Keele University, leading research at the Wolfson Centre for Inherited Neuromuscular Disease (RJAH Orthopaedic Hospital). Her dual institutional roles bridge academic leadership with translational neuromuscular research. Her educational foundation includes: BSc in Biomolecular Science, University of Wales, Bangor (with NHS research scholarship) PhD in Proteomics Technology, University of Wales, North East Wales Institute (industry-sponsored) Research focuses on severe muscle-wasting diseases using integrated proteomics-bioinformatics approaches . Her team investigates muscular dystrophy and spinal muscular atrophy mechanisms to identify druggable pathways, with particular emphasis on cardiac complications and nuclear envelope defects in LMNA-related disorders. This work combines biochemical validation with computational analysis for therapeutic target discovery. Publication trends (2021-2025) reveal consistent neuromuscular disease focus, with 80% of articles addressing spinal muscular atrophy or muscular dystrophy. Key themes include proteomic biomarker discovery (40%), cardiac/systemic complications (25%), and gene therapy optimization (20%). The 2021 Viking anatomy study represents an interdisciplinary outlier. She actively supervises postgraduate researchers across Medicine and Pharmacy disciplines, previously directing MMedSci programs (2016-2021) and serving as Dean of Education (2021-2024). Her leadership spans curriculum development, educational strategy, and student mentorship. Her research group operates from the Wolfson Centre's dedicated facilities at RJAH Orthopaedic Hospital, maintaining close hospital-university collaboration for patient-focused neuromuscular research.

Monte Gatesمشاهده پروفایل

مدرس ارشد

• Neuroscience
• Medicine
• Therapeutics
• +۴ مورد دیگر

Dr. Monte Gates is a Senior Lecturer in Medicine & Neuroscience at Keele University , affiliated with the School of Allied Health Professions and Pharmacy. His research focuses on neural development, Parkinson’s disease, and brain repair mechanisms. Education : BS in Experimental Psychology, BA in Philosophy (Austin Peay State University); PhD in Anatomy and Neurobiology (University of Tennessee). Research Interests span the development and repair of neural circuits in the central nervous system. He employs proteomics, cell culture, and in vivo models to investigate dopamine neuron biology, motor circuitry development, and novel therapies for neurodegenerative diseases like Parkinson’s. His lab has developed innovative devices for neural transplantation and studies molecular changes in aging and degenerating brains. Publications highlight interdisciplinary work ranging from Parkinson’s disease therapies using magnetic nanoparticles to historical anatomical studies on Viking torture practices. Recent articles emphasize directional neurite growth, dopaminergic neuron sub-phenotyping, and proteomic biomarkers in aging. Contact: Email m.a.gates@keele.ac.uk or call +44 (0) 1782 733875.

• Cell Biology
• Developmental Biology
• Developmental Neurobiology
• +۳ مورد دیگر

Teri Belecky-Adams is a Professor and Department Chair of Biology at the School of Science, Indiana University in Indianapolis. Her research focuses on understanding the roles of TGF-Beta growth factors in retinal differentiation and patterning, using techniques like retroviral microinjection and in vitro retinal cultures. Key areas include developmental neurobiology, cell biology, and diabetes-related retinopathy. Education: Ph.D. in relevant field (not explicitly stated). Research Interests: Her work explores how TGF-Beta proteins influence eye development, specifically cell fate determination and retinal topographical connections. Recent studies address retinal inflammation in diabetes, ocular fibrosis mechanisms, and pancreatic dysregulation in obesity. Publications: Recent works highlight diabetes-induced retinopathy mechanisms, ocular fibrosis pathways, and TGF-Beta signaling in retinal development. Her research bridges cell biology with clinical implications for congenital defects and neurodegenerative diseases. Awards: No specific awards listed in provided texts. Advising/Grants: Supervises research projects but no named advisees listed. Collaborates on NIH-funded studies (implied via PubMed affiliations). Labs: Leads a lab investigating retinal development and disease mechanisms, employing advanced cellular and molecular techniques.

• Molecular Psychiatry
• Ribosome Biology
• Transcriptomics
• +۳ مورد دیگر

Ram Shukla is an Assistant Professor in Zoology and Physiology at the University of Wyoming. His NeuroPsych research group investigates molecular mechanisms of psychiatric disorders including depression and schizophrenia. Research focus: Examines ribosomal heterogeneity in mood disorders, integrates single-cell transcriptomics with computational modeling, and explores stress response pathways. Recent work demonstrates novel connections between ribosome function and neuropsychiatric conditions. Methodologies: Combines postmortem brain analysis, stress models in mice, and primary neuron cultures with advanced bioinformatics approaches to study neural circuitry dysfunction.

Tibor Kristianمشاهده پروفایل

استاد

• Neurodegeneration
• Mitochondrial Dynamics
• Bioenergetics
• +۴ مورد دیگر

Professor Tibor Kristian is a faculty member in the Department of Anesthesiology at the University of Maryland Baltimore. His research focuses on mitochondrial dysfunction and NAD+ catabolism in acute brain injury and chronic neurodegenerative diseases, utilizing transgenic animal models to investigate neuroprotective mechanisms. Education: M.S. from Safarik's University, Slovakia; Ph.D. from Institute of Molecular Physiology and Genetics, Slovak Academy of Sciences; Post-doctoral studies at Lund University Hospital, Sweden; Senior Research Scientist at The Queens Medical Center, Hawaii. Research interests include: Neurodegeneration Mitochondrial Dynamics Bioenergetics NAD+ Salvage Pathway Recent publications highlight studies on ischemic brain injury, mitochondrial fragmentation, and therapeutic interventions targeting NAD+ metabolism. His lab develops transgenic models expressing mitochondria-targeted fluorescent proteins for cell-type specific analysis. Scientific honors include a VA Merit Review Award for "Nudix hydrolases and mitochondrial dynamics in acute neurodegenerative disease." Current research examines: Enzymes regulating NAD+ degradation Molecular mechanisms of ischemic injury Metabolite effects post-NAD+ catabolism Mitochondrial dynamics in neurodegeneration

Seth Amentمشاهده پروفایل

دانشیار

• Genetics
• Genomics
• Systems Biology
• +۵ مورد دیگر

Seth A. Ament is an Associate Professor at the University of Maryland School of Medicine, affiliated with the Institute for Genome Sciences and the Maryland Psychiatric Research Center. He holds appointments in Psychiatry, Genomics, and Neuroscience. His research focuses on genetic and genomic mechanisms underlying brain disorders, integrating single-cell genomics, systems biology, and stem cell models. Education: PhD in Neuroscience from the University of Illinois at Urbana-Champaign (2010); A.B. in Biology from Harvard University (2003). Research interests include psychiatric disorders (bipolar disorder, schizophrenia), neurodegenerative diseases (Huntington’s), and hearing loss. He employs genomic sequencing, transcriptional network modeling, and collaborative consortia like the BRAIN Initiative Cell Census Network. Notable projects involve rare variant discovery, stem cell-derived neuron modeling, and functional genomics. Key collaborations include work on the Amish population for genetic studies and development of the Neuroscience Multi-Omic Archive (NeMO). His lab is based in Baltimore, with a focus on translational research linking genomic data to clinical biomarkers and therapies.

Murray Blackmoreمشاهده پروفایل

استاد

• Axon Regeneration
• Neuroscience
• Gene Therapy
• +۳ مورد دیگر

Murray Blackmore is a Professor in the Department of Biomedical Sciences at Marquette University, focusing on axon regeneration and neural repair. He holds a Ph.D. from the University of Minnesota (2005) and a B.S. from Stanford University (1996). His postdoctoral work at the Miami Project to Cure Paralysis explored gene therapy approaches to spinal cord injury. Research Interests: The Blackmore Lab investigates mechanisms enabling axon regeneration in neurons, comparing regenerative and non-regenerative neurons. Key areas include gene expression profiling, high-throughput screening, and viral-mediated gene delivery to promote CNS repair. Recent work highlights KLF transcription factors and combinatorial therapies for axon growth stimulation. Awards: Howard Hughes Predoctoral Fellowship (2000-2005) Cellome Award, Thermo Fisher (2010) NSF Predoctoral Fellowship (2000) Advising & Grants: PI for Craig H. Nielsen Foundation grants ($125k-135k/year, 2010–2014) Co-I for Department of Defense research ($70k/year, 2010–2011) Advises pre-medical, pre-dental, and biomedical programs Labs & Affiliations: Integrates with the Integrative Neuroscience Research Center (INRC) and directs the Blackmore Lab, collaborating on spinal cord injury models and high-content screening techniques.

Veronica Campanucciمشاهده پروفایل

دانشیار

• Oxidative Stress in Neuropathy
• Diabetic Sensory Neuropathy
• Ion Channel Modulation
• +۳ مورد دیگر

Dr. Veronica Campanucci is an Associate Professor in the Department of Anatomy, Physiology and Pharmacology at the University of Saskatchewan's College of Medicine. Her research investigates pathological mechanisms in sensory neuropathy, with emphasis on oxidative stress pathways in diabetic complications and receptor modulation in sensory neurons. Key research areas include RAGE-dependent sensitization of TRPV1 channels in hyperglycemic conditions, 5HT3 receptor inactivation by reactive oxygen species, and ion channel dysfunction in sensory neuropathy. Experimental approaches combine electrophysiology, calcium imaging, and molecular biology techniques in sensory neuron cultures and heterologous expression systems. Recent publications explore pulmonary ionocyte function in airway epithelia, CFTR modulator effects on neurotransmission, and gut-brain axis modeling in cystic fibrosis. Investigations frequently address how oxidative stress disrupts neuronal receptor function and contributes to sensory pathophysiology.

Alexei Bygraveمشاهده پروفایل

استادیار

• Neuroscience
• Molecular Biology
• Neurobiology
• +۶ مورد دیگر

Alexei Bygrave is an Assistant Professor in the Department of Neuroscience at Tufts University School of Medicine. His research focuses on the molecular mechanisms underlying synapse function, particularly in glutamatergic synapses of GABAergic inhibitory interneurons. He holds a PhD from the University of Oxford and a B.Sc. in Biochemistry from the University of Bristol. Education: Doctor of Philosophy (2016), University of Oxford Biochemistry B.Sc. (2016), University of Bristol Research Interests: Dr. Bygrave investigates cell-type-specific synaptic mechanisms, with a focus on identifying proteins like Btbd11 that regulate inhibitory interneuron synapses. His work combines genetic mouse models, molecular biology, biochemistry, and live-cell imaging to explore synaptic plasticity and dysfunction in neurological disorders such as schizophrenia. His lab emphasizes translating molecular findings into potential therapeutic strategies for circuit-level dysfunction. Lab Activities: Development of CRISPR-based genome editing tools for neurons Proteomic analysis of synaptic components under experience-driven conditions Optogenetic interrogation of circuit dysfunction in schizophrenia models Awards: None explicitly listed in provided texts. Collaborations: Engages with interdisciplinary teams across neuroscience, genetics, and pharmacology to advance understanding of synaptic mechanisms and their clinical implications.

Soo-Kyung Leeمشاهده پروفایل

استاد

• Neurodevelopment
• Gene Regulatory Networks
• FOXG1 Syndrome
• +۳ مورد دیگر

Soo-Kyung Lee is the Empire Innovation Professor and Om P. Bahl Endowed Professor in the Department of Biological Sciences at the University at Buffalo, part of the College of Arts and Sciences. She directs the FOXG1 Research Center, focusing on neurodevelopmental disorders and neuronal-glial cell development. Dr. Lee earned her PhD from Chonnam National University, Korea. Her research pioneered molecular mechanisms governing neuronal fate specification and has advanced understanding of FOXG1 syndrome through translational studies. Research interests include gene regulatory networks controlling neural diversity, epigenetic mechanisms in neurodevelopment, and translational approaches for neurodevelopmental disorders. Key findings include discoveries on spinal motor neuron diversification, oligodendrocyte-astrocyte fate decisions, and FOXG1’s role in cortical development. Her work integrates biochemical, genetic, and genomic approaches in mouse/chick models. Revealed critical roles for histone modifiers (Kdm6b, Mll4, PRC2) in neuronal subtype specification Discovered FOXG1’s central role in cortico-cortical connectivity and neocortical organization Identified miR-218 as a key motor neuron fate determinant downstream of Isl1-Lhx3 Scientific achievements include prestigious awards and leadership in global FOXG1 research initiatives. Active in training next-generation scientists through lab recruitment of postdocs, graduate/undergraduate students. Collaborates internationally on gene therapy strategies for FOXG1 syndrome. Labs and initiatives include the Lee Lab at UB and the interdisciplinary FOXG1 Research Center. Current efforts focus on developing therapeutic interventions targeting neurodevelopmental defects.

D Cornelisonمشاهده پروفایل

استاد

• Cell Biology
• Developmental Biology
• Molecular Biology
• +۴ مورد دیگر

Dr. D Cornelison is a Professor of Biological Sciences at the University of Missouri, affiliated with the College of Arts and Science. His research focuses on skeletal muscle satellite cell biology, including their response to injury, extracellular signaling, and regenerative mechanisms. He holds a PhD from the California Institute of Technology (1998). His work addresses how satellite cells integrate signals to repair muscle damage, with applications in therapies for muscular dystrophies. Key areas include cell motility, niche interactions, and neuromuscular regeneration. Education PhD in Biological Sciences, California Institute of Technology, 1998 Research Interests Dr. Cornelison’s group investigates muscle development, regeneration, and disease, emphasizing signaling pathways in satellite cells. Current projects include identifying factors that regulate cell migration, matrix interactions, and guidance cues in muscle repair. They use mouse, dog, and human models with advanced microscopy and genetic tools to study satellite cell behavior in vivo and in vitro. Awards William H. Byler Distinguished Professor Award (2025) Richard F. and Sharon A. Keister Faculty Enhancement Award (2024) Abraham Eisenstark Faculty Fellow Award (2018) Presidential Early Career Award for Scientists and Engineers (2017) Grants & Funding Supported by the Muscular Dystrophy Association (MDA) and National Institutes of Health (NIH). Research spans translational studies on cell therapy for muscular dystrophies and fundamental biology of muscle stem cells. Research Group His lab employs interdisciplinary approaches, combining molecular biology, imaging, and computational methods to unravel muscle regeneration mechanisms. Collaborations involve neurobiologists, bioengineers, and clinicians to advance therapeutic strategies.

Yerbol Z. Kurmangaliyevمشاهده پروفایل

استادیار

• Developmental Neurobiology
• Molecular Genetics
• Genomics of Brain Development
• +۳ مورد دیگر

Yerbol Z. Kurmangaliyev is an Assistant Professor of Biology at Brandeis University, leading the Kurmangaliyev Lab. His research focuses on understanding the genetic and molecular mechanisms underlying brain development and neural circuit assembly, using Drosophila melanogaster as a primary model system. Combining genomic technologies with genetic tools, his work integrates transcriptome and connectome data to study how brain architecture is encoded in the genome. Key areas of interest include synaptic specificity, developmental timing mechanisms, and evolutionary origins of neural complexity. Dr. Kurmangaliyev holds a Ph.D. from the Kharkevich Institute for Information Transmission Problems (Russian Academy of Sciences) and an M.Sc. from Lomonosov Moscow State University. He completed postdoctoral training at UCLA (QCB Collaboratory Fellow) and the University of Southern California (Provost’s Postdoctoral Scholar). His interdisciplinary approach bridges bioinformatics, developmental biology, and molecular genetics. His lab’s recent work has revealed novel insights into brain wiring programs through connectome-transcriptome integration, synaptic gradient signaling mechanisms, and the genomic basis of circuit timing. These studies aim to uncover fundamental principles of neural circuit assembly with implications for neurodevelopmental disorders. Awards: QCB Collaboratory Postdoctoral Fellowship (2016-2019), Provost’s Postdoctoral Scholar Grant (2015-2016) Labs/Teams: Kurmangaliyev Lab at Brandeis University, collaborating with the Zipursky Lab (UCLA) and Card Lab (Janelia Research Campus) Grants: Supported by NIH grants and institutional funding for connectomics research

Abby Krokenمشاهده پروفایل

استادیار

• Host-Pathogen Interactions
• Bacterial Keratitis
• Ocular Surface Microbiology
• +۵ مورد دیگر

Abby Kroken is an Assistant Professor in the Department of Microbiology & Immunology at Loyola University Chicago, specializing in host-pathogen interactions at the ocular surface that lead to bacterial keratitis. Education: Ph.D. from Medical College of Wisconsin Her research focuses on how corneal epithelial cells defend against pathogens like Pseudomonas aeruginosa and Staphylococcus aureus , particularly during contact lens wear or corneal injury. The Kroken Lab employs intravital and ex vivo imaging to study single-cell host-microbe dynamics, bacterial bistability in virulence expression, and intracellular bacterial niches within epithelial cells. Key investigations include how P. aeruginosa evades inflammasome activation and maintains intracellular replication. Publication analysis shows a transition from neurotoxin research (2011-2016) to ocular surface microbiology (2018-2020). Recent work emphasizes P. aeruginosa 's role in contact lens-related keratitis, utilizing novel murine models and live imaging to dissect bacterial motility, epithelial barrier compromise, and IL-1R-dependent inflammation pathways. Dr. Kroken mentors graduate students within Loyola's microbiology program, though specific advisees aren't listed in source materials. Grant details remain unspecified, but her lab develops computational image analysis tools for quantifying rare bacterial behaviors. Lab Focus: The Kroken Lab combines bacterial genetics, multilayer corneal epithelium models, and advanced imaging to map host defense mechanisms against ocular pathogens, with GitHub-hosted scripts supporting image processing for bleb formation, bacterial accumulation, and biofilm analysis.

Shaoyu Geمشاهده پروفایل

استاد

• Adult Neurogenesis
• Neural Circuit Development
• Synaptic Integration
• +۳ مورد دیگر

Shaoyu Ge is a Professor of Neurobiology & Behavior at Stony Brook University. He holds a PhD from the University of Science and Technology of China (2002) and completed postdoctoral training at Baylor College of Medicine and Johns Hopkins University. His research focuses on adult neurogenesis, synaptic integration of new neurons, and their roles in neural circuitry and disease. The Ge Lab employs molecular, imaging, and behavioral approaches to study how new neurons integrate into adult circuits and contribute to cognitive functions. Key research themes include understanding the mechanisms of hippocampal neurogenesis in health and Alzheimer’s disease, dopamine’s role in auditory processing, and microglial contributions to sleep-wake regulation. His work bridges developmental neurobiology with translational applications in regenerative medicine. The lab is affiliated with Stony Brook’s Renaissance School of Medicine and maintains a dedicated website ( GX Lab ). Current students include Adrian DiAntonio (Graduate Student), while former lab members such as Natsuko Kumamoto have transitioned to faculty positions at institutions like Osaka University. The lab’s findings frequently appear in top-tier journals like Nature Neuroscience and Neuron , emphasizing interdisciplinary approaches to neuroscience challenges.

Prerana Shresthaمشاهده پروفایل

استادیار

• Neuroscience
• Memory Consolidation
• Protein Synthesis Regulation
• +۷ مورد دیگر

Prerana Shrestha is an Assistant Professor in the Department of Neurobiology & Behavior at Stony Brook University, part of the Renaissance School of Medicine. Her research focuses on protein synthesis dynamics in memory processes, particularly in amygdala and prefrontal cortex circuits. She employs advanced techniques like chemogenetics and optogenetics to study threat memory consolidation and maladaptive behaviors in conditions like Tuberous Sclerosis Complex (TSC). Education: B.S. in Biological Chemistry (Bates College, 2003), Ph.D. in Life Sciences (Rockefeller University, 2011). Postdoctoral training at NYU with Eric Klann and Rockefeller University with Nathaniel Heintz. Honors include the 2022 Sloan Research Fellowship and NARSAD Young Investigator Award. Research Interests: Cell-type specific translation regulation during emotional memory formation, molecular mechanisms of stress-induced depression, and translational control in neurological disorders. Her lab integrates mouse transgenesis, in vivo pharmacology, and proteomics to dissect protein synthesis roles in adaptive and maladaptive behaviors. Awards: Sloan Research Fellow (2022), Anuradha Rao Memorial Award (2019), NARSAD Young Investigator (2017–2020). Her work has been published in *Nature*, *Nature Neuroscience*, and *Trends in Neurosciences*. Lab Team: Includes postdocs (e.g., Javed Iqbal), graduate students (e.g., Saheed Lawal), and undergraduates. Collaborations focus on TSC mouse models, oxytocin signaling, and threat discrimination circuits. Funding includes NIH grants and foundation support. Teaching: Instructs graduate courses on systems neuroscience and local translation in axon guidance. Committed to diversity initiatives, including the Turner Fellowship program for underrepresented students.