جست‌وجوی استاد و پژوهشگر

Lydia Finleyمشاهده پروفایل

پژوهشگر

• Cancer Biology
• Cell Biology
• Metabolomics
• +۴ مورد دیگر

Lydia Finley , PhD, is a Researcher at the Cell Biology Program within Memorial Sloan Kettering Cancer Center (MSKCC) and holds the Geoffrey Beene Junior Faculty Chair . She is also affiliated with the Gerstner Sloan Kettering Graduate School of Biomedical Sciences and leads a laboratory focused on the interplay between cellular metabolism and cell fate decisions. PhD, Harvard University BS, Yale University Her research investigates how metabolic pathways regulate stem cell differentiation and cancer cell survival , with particular emphasis on metabolite availability, chromatin landscape modifications, and gene expression programs. Key areas include non-canonical TCA cycle activity, amino acid metabolism in pluripotent cells, and metabolic vulnerabilities in cancer stem cells. Dr. Finley’s recent work highlights the role of α-ketoglutarate , pyruvate dependencies , and non-canonical metabolic circuits in cell identity maintenance. These studies span disciplines such as Cancer Biology , Stem Cell Metabolism , and Epigenetic Regulation . Scientific Awards : Tsuneko and Reiji Okazaki Award (2024) New York Stem Cell Foundation – Robertson Investigator (2023) Louise and Allston Boyer Young Investigator Award for Basic Research (2022) National Cancer Institute R37 MERIT Award (2020) Pershing Square Sohn Prize for Cancer Research (2020) Searle Scholar (2018) Dale F. Frey Breakthrough Award (2017) Dr. Finley advises Graduate Students and Postdoctoral Fellows , including current members Abigail Xie, Monika Komza, and Angela Montero. Her lab explores metabolic flux , nutrient sensing , and metabolite-epigenetic crosstalk , with implications for both developmental biology and cancer therapeutics . Lab Affiliations include MSKCC’s Cell Biology Program and collaborations with the Gerstner Sloan Kettering Graduate School.

Bjorn F Kafsackمشاهده پروفایل

دانشیار

• Malaria Parasite Biology
• Plasmodium falciparum
• Transcriptional Regulation
• +۷ مورد دیگر

Bjorn F Kafsack is an Associate Professor of Microbiology and Immunology at Weill Cornell Medical College , Cornell University, where he has held this position since 2022. His research is centered on malaria parasite biology , particularly the developmental regulation and transcriptional plasticity of Plasmodium falciparum , with a focus on immune evasion mechanisms and sexual differentiation . Education: Ph.D. , The Johns Hopkins University (2008) M.H.S. , The Johns Hopkins University (2006) M.S. , The Johns Hopkins University Bloomberg School of Public Health, Molecular Microbiology & Immunology, Bioinformatics B.S. , University of Washington (2001) Research Focus: Kafsack’s laboratory investigates how malaria parasites adapt to host immune pressures through epigenetic regulation , transcriptional reprogramming , and nutrient sensing . His work integrates single-cell genomics , chemical biology , and functional genomics to uncover new drug targets and understand parasite transmission biology . Publication Trends: His recent articles (2022–2025) emphasize transcriptional plasticity , antigenic variation , and proteasome targeting in P. falciparum . These studies reveal mechanisms of immune evasion , sexual commitment , and stress adaptation , often leveraging CRISPR-based tools and high-resolution transcriptomics . Funding & Leadership: Principal Investigator , NIH/NIAID project on Babesia inhibitor targets (2022–2025) Principal Investigator , ongoing NIH-funded malaria research (2019–2025) Co-Investigator , multi-site parasite biology consortium (2018–2024) Laboratory & Teams: Kafsack leads a multidisciplinary team at Weill Cornell, collaborating with chemical biologists , parasitologists , and computational biologists to advance antimalarial strategies and train next-generation scientists in infectious disease research .

Dazhong Xuمشاهده پروفایل

دانشیار

• Cancer Biology
• Molecular Biology
• Environmental Toxicology
• +۵ مورد دیگر

Dazhong Xu, Ph.D. , is an Associate Professor at the School of Medicine, New York Medical College (NYMC) , where he joined in 2014 as Assistant Professor of Pathology and Head of the Flow Cytometry Core Laboratory. He served as Vice Chair of Research in the Department of Pathology from 2017 to 2021 and was promoted to Associate Professor of Pathology, Microbiology, and Immunology in 2020. Prior to NYMC, he worked at New York University School of Medicine as a Research Assistant Professor and at Tufts University School of Medicine as an Instructor of Medicine. Education: Ph.D. in Biomedical Sciences, Wright State University M.S. in Zoology, University of Florida B.S. in Biology, Nanjing University Postdoctoral Training: Massachusetts General Hospital/Harvard Medical School, Tufts Medical Center Dr. Xu's research focuses on the molecular and cellular biology of cancer , particularly lung carcinogenesis caused by environmental and occupational carcinogens. His lab aims to identify novel molecular mechanisms driving cancer initiation and progression using advanced tools like mouse models and genetic engineering , with applications in DNA damage response and chromatin modifications . His recent publications highlight studies on hexavalent chromium toxicity (2024), DNA polymerase regulation (2025), and adapter protein dynamics (2021-2023). These works span environmental toxicology , genomic instability , and immunomodulation . Honors & Awards: 2005 Jay N. Cohn New Investigator Award in Basic Science 2005 & 2004 Travel Awards, The Heart Failure Society of America 2002 National Research Service Award Individual Postdoctoral Fellowship Dr. Xu teaches Selected Topics in Cancer Biology and Introduction to Pathology at NYMC. His collaborations include researchers from Tufts, Harvard, and the Heart Failure Society of America, with grants emphasizing environmental carcinogenesis and cellular stress responses .

Irfan A Asanganiمشاهده پروفایل

دانشیار

• Cancer Epigenetics
• Chromatin Regulation
• Transcriptional Addiction
• +۶ مورد دیگر

Irfan A Asangani is an Associate Professor of Cancer Biology at the Perelman School of Medicine, University of Pennsylvania. He is affiliated with the Abramson Family Cancer Research Institute and the Penn Epigenetics Institute, focusing on molecular epigenetic events in cancer progression and therapy resistance. Education: B.S. and M.S. in Biochemistry (First Class) from Madras University, India; Ph.D. in Cancer Biology (Summa cum laude) from Klinikum Mannheim/DKFZ/Heidelberg University, Germany. Research Interests include cancer epigenetics, chromatin regulation, non-coding RNA, androgen receptor signaling, and mechanisms of resistance to targeted therapies. His lab employs multidisciplinary approaches to translate epigenetic insights into clinical tools for diagnostics, prognostics, and combination therapies. Recent Publications highlight roles of chromatin regulators like NSD2 in prostate cancer, LCMT1 loss in therapy resistance, and SOX4 in cellular reprogramming. Themes span epigenetic reprogramming, metabolic adaptations, and combinatorial strategies to suppress resistant tumor cells. Lab Affiliation : The Asangani Lab at Penn investigates transcriptional addiction in cancer cells, collaborating with Abramson Cancer Center and the Penn Epigenetics Institute.

Nadia Dahmaneمشاهده پروفایل

دانشیار

• Developmental Biology
• Neuroscience
• Glioblastoma
• +۶ مورد دیگر

Nadia Dahmane is an Associate Professor of Developmental Biology in Neuroscience at the Brain and Mind Research Institute , Weill Cornell Medical College (2017–present). Her work spans developmental biology, neuro-oncology, and translational medicine, focusing on glioblastoma, pediatric brain tumors, and Sonic Hedgehog signaling. Research Themes : Tumor microenvironment, chromatin organization, radiogenomics, immunotherapy, and signal transduction in gliomas. Grants : Funded by the Pablove Foundation (2025–2026), National Cancer Institute (2023–2025), and National Institute of Neurological Disorders & Stroke (2020–2025). Publications highlight applications of machine learning in glioblastoma subtyping, CRISPR screening in pediatric tumors, and focused ultrasound for drug delivery. Her 2025 articles address CAR T cell immunotherapy resistance, single-dose radiotherapy, and chromatin landscapes in pediatric cancers. Scientific Awards : Not explicitly mentioned. Students : No advisees listed. Email : nad2639@med.cornell.edu .

Golnaz Vahediمشاهده پروفایل

استاد

• Genomic Immunology
• Chromatin Organization
• T Cell Development
• +۶ مورد دیگر

Professor Golnaz Vahedi of the University of Pennsylvania School of Medicine Department of Genetics is a leading figure in genomic immunology with key affiliations to the Institute for Diabetes, Obesity and Metabolism, Abramson Cancer Center, Institute for Immunology & Immune Health, and co-directorship of the Penn Epigenetics Institute. Her educational background spans Electrical Engineering degrees from Sharif University (B.Sc.), University of Alberta (M.Sc.), and Texas A&M University (Ph.D.), followed by NIH postdoctoral work under Dr. John O'Shea. 314 BRB II/III, 421 Curie Boulevard, Philadelphia, PA 19104 Primary email: vahedi@pennmedicine.upenn.edu Her research integrates computational biology with immune cell epigenetics to decode: 3D chromatin organization in T cells Transcription factor mechanisms (e.g., TCF-1's nucleosome-unwrapping properties) Genetic variation's impact on T1D pathology Lentiviral integration epigenomic effects Recent publications demonstrate her lab's expertise in chromatin tracing and multi-enhancer interactions . Notable awards include the Chan Zuckerberg Initiative award and multiple journal cover features. Her work bridges genomics , computational immunology , and autoimmune disease research through innovative techniques like PRISM, EpiVIA, and Stripenn. Key collaborations: Human Pancreas Analysis Program (HPAP), Abramson Cancer Center

Suming Huangمشاهده پروفایل

استاد

• Epigenetics
• Gene Regulation
• Leukemia Research
• +۵ مورد دیگر

Suming Huang serves as Professor and Chair of the Four Diamonds Epigenetics and Gene Regulation Research Program in Pediatric Oncology at Penn State University's College of Medicine. Holding dual appointments in the Department of Pediatrics Division of Hematology and Oncology and the Department of Molecular and Precision Medicine, Huang directs research within the Penn State Cancer Institute's Mechanisms of Carcinogenesis program. With an h-index of 36 and over 4,000 citations, Huang maintains an active research profile through the National Cancer Institute. Huang's research centers on epigenetic mechanisms in leukemia development, specializing in transcriptional regulation, non-coding RNA functions, and chromatin organization. Key focus areas include TAL1/SCL regulation in T-cell leukemia, lncRNA-mediated genome organization in AML, and the role of histone modifiers like HDAC1 in tumor suppression. The research integrates hematopoietic stem cell biology with leukemogenesis pathways, particularly examining how non-coding RNAs reprogram chromatin architecture in NUP98-rearranged leukemias and other hematological malignancies. Current publication trends reveal consistent output in high-impact oncology journals, with 2025 publications examining CK2α deletion effects on stem cells, IKAROS/HDAC1 heterochromatin regulation, guanine biosynthesis blockade in MLL leukemias, and HoxBlinc lncRNA functions. These works demonstrate Huang's shift toward therapeutic targeting of epigenetic vulnerabilities while maintaining core investigations into transcriptional dysregulation in pediatric cancers. Huang leads eight major research projects including the active National Cancer Institute grant Role of lncRNA mediated R-loops in CTCF boundary function and AML genome organization (2021-2025). Past projects span leukemogenesis mechanisms, long non-coding RNA functions in hematopoiesis, and TAL1 regulation in T-cell leukemia, securing sustained NCI funding since 2008. The research program maintains strong collaborations across hematology-oncology and molecular medicine departments. The Four Diamonds Epigenetics and Gene Regulation Research Program in Pediatric Oncology serves as Huang's primary research hub, focusing on translating epigenetic discoveries into therapeutic strategies for childhood leukemias. Current work integrates multi-omics approaches to dissect lncRNA-mediated chromatin reorganization in leukemic stem cells.

John M Sedivyمشاهده پروفایل

استاد

• Cellular Senescence
• Aging
• Cancer Biology
• +۵ مورد دیگر

John M. Sedivy is the Hermon C. Bumpus Professor of Biology and Director of Brown University's Center for the Biology of Aging. A faculty member since 1996, he holds appointments in the Department of Molecular Biology, Cell Biology and Biochemistry. His career spans groundbreaking work in cell cycle regulation , oncogene biology , and cellular aging . Education: PhD in Molecular Biology (Harvard, 1985), BSc (University of Toronto, 1978) Sedivy's research focuses on cellular senescence , epigenetic regulation , and transposable element dynamics in aging. He pioneered single-cell senescence assays and epigenetic aging studies , notably demonstrating age-associated heterochromatin expansion and transposable element activation in somatic tissues. His 15+ recent publications highlight themes in telomere biology , chromatin changes during aging , Myc signaling , and bioinformatics approaches to gene network analysis. Key contributions include first in vivo quantification of cellular senescence in primates (2006) and epigenetic clocks for aging (2011). Scientific Awards: NIH Merit Award (2009) Glenn Award for Aging Research (2011) Hermon C. Bumpus Chair (2006) Ellison Senior Research Scholar (2007) As an administrator, he chaired Brown's Department of Molecular Biology, Cell Biology and Biochemistry and founded its Genomics and Proteomics Center. He served on NIH CMAD study section (2003-2012) and co-edited Aging Cell (2006-2012). His lab's work has been continuously funded by NIH since 1989, including a 10-year MERIT award (R37 AG016694).

Hongjie Liمشاهده پروفایل

استادیار

• single-cell RNA sequencing
• neural development
• aging biology
• +۷ مورد دیگر

Hongjie Li is an Assistant Professor at the Huffington Center on Aging within Baylor College of Medicine , affiliated with the Department of Molecular and Human Genetics and Dan L Duncan Comprehensive Cancer Center . He leads the Hongjie Li Lab and serves as Graduate Faculty in multiple programs. Education : PhD from University of Rochester & Buck Institute for Research on Aging (2016), Postdoctoral Training at Stanford University (2020) Research Focus : Develops multi-omics technologies (transcriptomics, epigenomics, proteomics) to study neural development, aging, and age-triggered diseases. His work examines: Glia-neuron interactions in brain aging Systemic inflammatory signals Gut-brain axis in aging Tumor initiation mechanisms in aging intestines Integrating fly genetics with cross-species models Notable Contributions : Pioneered single-cell RNA sequencing in Drosophila neurons (2017 Cell, 2020 Current Biology), co-developed single-nucleus RNA-seq for the Fly Cell Atlas project (2021 eLife). His recent work (2023-2025) explores: Whole-organism aging features Tauopathy neuroprotection mechanisms Food odor-feeding suppression circuits Computational frameworks for cell communication Histone modulation of lifespan Scientific Awards & Funding : NIH R00 Pathway to Independence Award CPRIT (Cancer Prevention and Research Institute of Texas) Ted Nash Long Life Foundation Welch Foundation Award Longevity Research Grant

Michael D Taylorمشاهده پروفایل

استاد

• Pediatric Brain Tumors
• Molecular Genetics
• Epigenomics
• +۴ مورد دیگر

Dr. Michael D. Taylor is a Professor at Baylor College of Medicine and holds the Cyvia and Melvyn Wolff Chair of Pediatric Neuro-Oncology at Texas Children’s Hospital , where he serves as Director of the Pediatric Brain Tumor Research Program. He is also a Staff Neurosurgeon at Texas Children’s Hospital and a CPRIT Scholar in Cancer Research. Education: MD, University of Western Ontario (1994) PhD, University of Toronto (2002) Residency, University of Toronto (2003) Postdoctoral Fellowship in Cancer Genomics, St. Jude Children’s Research Hospital (2004-2005) Research Focus: Dr. Taylor’s work centers on the molecular genetics of medulloblastoma and ependymoma , two common malignant pediatric brain tumors. His lab has redefined medulloblastoma as at least four distinct diseases through genomic and epigenomic studies, with recent discoveries linking cerebellar tumors to disruptions in early brain development ( Nature 2019, 2022 ). His team has published over 400 peer-reviewed articles, including breakthroughs in Nature , Cell , and Lancet Oncology , cited 70,000+ times. Scientific Awards: Canada’s Top 40 under 40 (2008) Royal College Gold Medal in Surgery (2010) Zulch Prize in Neuroscience (2016) Royal Society of Canada Fellowship (2019) Team & Collaborations: His interdisciplinary Taylor Lab includes cell biologists, bioinformaticians, and clinicians working with preclinical models, clinical samples, and single-cell multiome sequencing. Key collaborators span institutions like St. Jude, Hospital for Sick Children, and University of Toronto.

Wolfgang Enardمشاهده پروفایل

استاد

• Primate Genomics
• Evolutionary Biology
• Computational Biology
• +۵ مورد دیگر

Prof. Dr. Wolfgang Enard is a faculty member at Ludwig Maximilian University of Munich, leading research in Primate Genomics , Evolutionary Biology , and Computational Biology . His work bridges evolutionary genomics with experimental molecular mechanisms to understand human-specific traits, particularly focusing on speech evolution and brain size development. Studying FOXP2 transcription factor in human speech evolution using mouse models Investigating genetic basis of brain size evolution through cross-species genomic comparisons Generating and analyzing induced pluripotent stem cells (iPSCs) from primates for evolutionary studies His laboratory employs RNA-Seq , ChIP-Seq , and proteomics to explore regulatory networks, with recent publications emphasizing cross-species comparisons, epigenetic evolution, and stem cell engineering. While no explicit awards are listed, his work has been cited in numerous high-impact publications across genomics, neurobiology, and disease modeling. Key trends in his research include comparative epigenomics of neural development, iPSC-based evolutionary studies , and multiomic approaches to disease mechanisms . His lab maintains active collaborations and generates specialized stem cell lines for cross-primate investigations.

Guoping Fanمشاهده پروفایل

استاد

• DNA Methylation
• Chromatin Remodeling
• Neural Development
• +۴ مورد دیگر

Guoping Fan is a Professor in the Department of Human Genetics at the David Geffen School of Medicine, University of California, Los Angeles (UCLA). His research focuses on the interplay between DNA methylation, chromatin remodeling, and neural development, with significant contributions to understanding epigenetic mechanisms in stem cell differentiation, reprogramming, and disease modeling. Key areas of interest include Epigenetic regulation of gene expression Neuronal function and development Stem cell biology (pluripotent and mesenchymal stem cells) Retinal pigment epithelium differentiation Single-cell multi-omics profiling Recent publications highlight his work on DNA/RNA methylation crosstalk (2025), hydrogel-mediated cell differentiation (2024), and single-cell epigenetic analysis of retinal organoids (2023). His studies span neurogenetics, cancer epigenetics, and bioengineering applications. His laboratory at UCLA explores molecular mechanisms linking epigenetics to developmental disorders and regenerative medicine, utilizing advanced techniques like single-cell RNA sequencing and chromatin accessibility profiling. No explicit awards or advisees are mentioned in the provided text.

Liming Peiمشاهده پروفایل

دانشیار

• Cardiac Endocrinology
• Single-Cell Multiomics
• Mitochondrial Function and Metabolism
• +۳ مورد دیگر

Liming Pei, Ph.D., serves as Associate Professor of Pathology and Laboratory Medicine at the University of Pennsylvania's Perelman School of Medicine and Children's Hospital of Philadelphia (CHOP), where he directs a research program focused on inter-organ communication in metabolic homeostasis. His laboratory investigates how the heart functions as an endocrine organ, employs single-cell multiomics to decode cardiac development and disease, and elucidates cell-type-specific metabolic regulation through nuclear receptors like ERRγ. Dr. Pei earned his B.S. from the University of Science and Technology of China (2000) and Ph.D. from UCLA (2006), followed by postdoctoral training at the Salk Institute. He joined CHOP/UPenn in 2013 as Assistant Professor before promotion to Associate Professor. His research spans three synergistic domains: (1) Cardiac endocrinology, where his team discovered GDF15 as a heart-derived hormone regulating body growth and liver metabolism, explaining failure-to-thrive in pediatric heart disease; (2) Single-cell multiomics, pioneering snRNA-Seq in cardiac research to build human heart atlases through NIH HuBMAP and study Fontan-associated liver disease; (3) Mitochondrial metabolism, identifying ERRγ as a master regulator of cell-type-specific energy pathways essential for cardiac, neuronal, and renal function. His work employs proximity labeling, single-nucleus sequencing, and transgenic models to uncover fundamental mechanisms with therapeutic implications. Recent publications reveal his lab's dominance in cardiac multiomics, with 2024-2025 studies on Fontan physiology, mitochondrial heteroplasmy, and ERRγ-targeted heart failure therapies appearing in Science Translational Medicine and Nature Cell Biology. His work demonstrates consistent innovation in linking molecular mechanisms to clinical metabolic and cardiac disorders. Funded by multiple NIH grants and Department of Defense awards, Dr. Pei actively mentors postdoctoral fellows and graduate students through CHOP/UPenn training programs. His lab offers rotation projects in cardiac hormone discovery, single-cell atlas development, and mitochondrial disease modeling, emphasizing computational and experimental integration. The Pei Lab operates within CHOP's Center for Spatial and Functional Genomics, collaborating with UPenn's Institute for Diabetes Obesity and Metabolism and the Cardiovascular Institute. Current initiatives include developing ERRγ-based therapies for kidney disease and expanding the understanding of heart-liver metabolic crosstalk through clinical-translational partnerships.

Fei Chenمشاهده پروفایل

استاد

• Cancer Epigenetics
• Environmental Carcinogenesis
• Cancer Stem Cells
• +۴ مورد دیگر

Fei Chen, PhD is a Professor in the Department of Pathology at the Renaissance School of Medicine, Stony Brook University. Dr. Chen leads the Chen Research Lab within the Oncogenic Drivers and Mechanisms of Carcinogenesis Research Program at the Stony Brook Cancer Center, investigating molecular mechanisms of environmental carcinogenesis. Dr. Chen's educational background includes: Medical degree from Nantong Medical College PhD in Immunology from Peking University Health Sciences Center Postdoctoral training in Pathology at Pennsylvania State University Dr. Chen's research program focuses on epigenetic and genetic regulations of genes in human cancers related to environmental exposures to mineral dust, carcinogenic metals, and chemical carcinogens. The lab employs biochemical, gene editing, and multi-omics approaches to elucidate mechanisms by which gene expression and function are altered in cancer. A principal research question addresses how metabolic reprogramming from mitochondrial TCA cycle to glycolysis affects DNA and histone methylation, chromatin configuration, and cancer stem cell self-renewal. Analysis of recent publications shows a strong focus on arsenic-induced carcinogenesis, with particular emphasis on the mdig gene, metabolic reprogramming, and cancer stem cell formation. The research spans toxicology, cancer biology, epigenetics, and molecular signaling, with applications to lung cancer, prostate cancer, and other malignancies. Recent work also explores connections between endoplasmic reticulum stress, mitochondrial function, and autophagy in carcinogenesis. Dr. Chen has mentored numerous graduate students and postdoctoral fellows, with several former trainees now holding independent research positions. The lab has been supported by multiple NIH grants focused on environmental carcinogenesis and cancer mechanisms. The Chen Research Lab operates at the Stony Brook Cancer Center (MART, Level 8, Room 830) and comprises a multidisciplinary team of molecular biologists, biochemists, and computational scientists. The lab maintains strong collaborations with clinical researchers at Stony Brook Cancer Center to translate basic findings into potential therapeutic approaches for environmentally-linked cancers.

Gilbert Rahmeمشاهده پروفایل

استادیار

• Cancer epigenetics
• Brain tumor epigenetics
• Gliomas
• +۷ مورد دیگر

Gilbert Rahme is an Assistant Professor in the Department of Pharmacological Sciences at Stony Brook University's Renaissance School of Medicine and the Cancer Center. He leads the Rahme Lab, which focuses on cancer epigenetics with particular emphasis on brain tumors called gliomas. His research is generously supported by the NIH/NCI, the Renaissance School of Medicine, and the Stony Brook Cancer Center. Dr. Rahme's research centers on how epigenetic reprogramming drives tumorigenesis and tumor progression, particularly in gliomas. His lab leverages mouse models, human tumor tissues, and computational analyses to discover epigenetic lesions, computationally infer their driver potential, and functionally demonstrate their role in tumorigenesis using in vitro and in vivo approaches. A major focus is understanding how IDH mutant gliomas exhibit epigenetic reprogramming due to CpG hypermethylation driven by 2HG production from mutant IDH. The Rahme Lab's publications reveal a consistent focus on epigenetic mechanisms in brain cancer, particularly examining DNA methylation, CTCF binding sites, and topologically associated domains (TADs) that define the 3-dimensional shape of the genome. Their research shows how epigenetic lesions can transform oligodendrocyte progenitor cells (OPCs) and drive gliomagenesis. Recent work published in Cell demonstrates how specific epigenetic lesions underlying hypermethylation can transform OPCs, providing critical insights into glioma development. NIH/NCI funding for brain tumor epigenetics research Support from Renaissance School of Medicine and Stony Brook Cancer Center Dr. Rahme actively mentors graduate students and postdocs, with his student Jina Yim recently winning the best poster presentation at the Gloria and Mark Snyder Symposium. The Rahme Lab currently has openings for technicians, graduate students, and postdocs, with particular interest in candidates applying to Molecular and Cellular Pharmacology, Molecular and Cellular Biology, Genetics, and Physiology and Biophysics programs. The Rahme Lab maintains an active presence at major cancer research conferences, including the American Association for Cancer Research special conference on brain cancer, where Dr. Rahme has presented the lab's work on modeling epigenetic drivers of gliomas.