جست‌وجوی استاد و پژوهشگر

Xiaowen Baiمشاهده پروفایل

استاد

• Stem Cell Biology
• Neurodegeneration
• Cardiotoxicity
• +۵ مورد دیگر

Xiaowen Bai, MD, PhD, is a Professor in the Department of Cell Biology, Neurobiology and Anatomy at the Medical College of Wisconsin. His research focuses on leveraging stem cells to model neurodegenerative diseases and cardiac disorders, with a particular emphasis on non-coding RNAs, mitochondria, and environmental stressors like anesthetics and alcohol. He holds a leadership role as Executive Director of Hematology at the World Federation of Chinese Medicine Societies (WFCMS). Education: MD, Shanxi Medical University (1993) PhD, Beijing University Stem Cell Center (2004) Postdoctoral Training, University of Texas MD Anderson Cancer Center (2007) Research Interests: Neurodegeneration mechanisms involving microRNAs, lncRNAs, and mitochondrial dysfunction Stem cell-based therapies for myocardial regeneration Diabetic cardiomyopathy pathogenesis and cardioprotection 3D organoid models for studying anesthetic and alcohol neurotoxicity Recent Work: Recent publications emphasize translational studies using human-induced pluripotent stem cell (iPSC)-derived models to investigate alcohol-induced brain injury, anesthetic neurotoxicity, and cardiac fibrosis. His lab also explores immunomodulatory pathways and redox signaling in inflammatory diseases. Labs/Teams: The Bai Lab develops novel in vitro models for disease modeling and drug testing, focusing on cerebral organoids and cardiac organoids to bridge basic research and clinical applications.

Rolf Brekkenمشاهده پروفایل

استاد

• Tumor Microenvironment
• Extracellular Matrix (ECM)
• Angiogenesis
• +۹ مورد دیگر

Dr. Rolf Brekken is a Professor at UT Southwestern Medical Center, where he serves as the Effie Marie Cain Research Scholar in Angiogenesis Research and Principal Investigator in the Hamon Center for Therapeutic Oncology Research. His laboratory investigates tumor-host interactions with a particular emphasis on extracellular matrix (ECM) and angiogenesis, focusing on pancreatic, breast, and lung tumors. Dr. Brekken received his Bachelor of Arts degree from Luther College in Decorah, Iowa and his PhD from UT Southwestern Graduate School of Biomedical Sciences. His graduate research focused on Vascular Endothelial Growth Factor (VEGF) as a target for therapy of solid tumors. He completed postdoctoral training in the Department of Vascular Biology at the Hope Heart Institute in Seattle, Washington. Dr. Brekken's research interests center on the tumor microenvironment, particularly pathways that drive epithelial plasticity and immune suppression. His laboratory studies how angiogenesis and ECM remodeling are key components of the metastatic cascade, with current projects focused on the function of matricellular proteins (e.g., SPARC and fibulin-5) as regulators of ECM remodeling and angiogenesis. Another major area of study is the development and evaluation of novel cancer therapies with a focus on anti-angiogenic strategies. Analysis of Dr. Brekken's recent publications (2017-2018) reveals a consistent focus on pancreatic cancer biology, particularly examining the tumor microenvironment, ECM remodeling, and angiogenesis. His work frequently investigates specific molecular targets including Axl receptor, discoidin domain receptors, and phosphatidylserine, with applications in both diagnostic approaches and therapeutic interventions. A notable trend is the exploration of combination therapies and mechanisms of resistance to anti-angiogenic treatments. Effie Marie Cain Research Scholar in Angiogenesis Research Dr. Brekken leads the Brekken Laboratory located in the Hamon Center for Therapeutic Oncology Research, where his team conducts robust modeling of pancreatic and breast cancer. His research has significant implications for understanding resistance mechanisms to anti-VEGF therapy and hypoxia-induced tumor progression. The laboratory's work spans from basic molecular mechanisms to preclinical therapeutic applications, with strong translational potential.

Prashant Bharadwajمشاهده پروفایل

پژوهشگر ارشد

• Alzheimer’s Disease
• Beta amyloid protein
• Autophagy
• +۳ مورد دیگر

Dr. Prashant Bharadwaj is a Research Fellow in Neuroscience at Edith Cowan University (ECU) and an Adjunct Research Fellow at Curtin University. He holds a PhD from ECU and specializes in Alzheimer’s disease, autophagy, and neuroprotective strategies. His research focuses on amyloid beta proteins, neurodegenerative mechanisms, and therapeutic interventions. He has been recognized with the NHMRC-ARC Dementia Research Development Fellowship (2016–2022) and a CSIRO travel award. Education: PhD (Edith Cowan University, 2011), Master of Biotechnology (Royal Melbourne Institute of Technology, 2007), Bachelor of Engineering (India, 2004). Research interests include Alzheimer’s pathogenesis, autophagy modulation, and biomarker discovery. Recent work explores polyphenol-rich Sorghum bicolor as a complementary therapy and investigates the role of amylin and beta amyloid interactions. Dr. Bharadwaj has authored over 30 peer-reviewed articles, with contributions to understanding autophagy’s role in amyloid clearance and disease progression. Grants include $640,220 for neuroprotective autophagy research and $250,000 for neurofilament biomarker analysis. He supervises three PhD students as Principal Investigator and collaborates on projects targeting childhood dementia and neuroinflammation.

• Sex Differences in Neurology
• Estrogen and Neuroprotection
• Cerebral Ischemia
• +۴ مورد دیگر

Dr. Ami P. Raval is a Research Assistant Professor in the Department of Neurology at the University of Miami Miller School of Medicine. Her research focuses on sex differences in stroke pathophysiology, particularly the interplay between estrogen, nicotine addiction, and hormonal contraceptives in exacerbating ischemic brain damage in females. She explores mechanisms of estrogen-mediated neuroprotection and investigates therapeutic interventions like whole body vibration therapy to mitigate post-stroke deficits in menopausal women. Dr. Raval's work also addresses the impact of electronic cigarettes and oral contraceptives on cerebral ischemia, emphasizing sex-specific vulnerabilities. Education & Training: PhD in Zoology (1995), Maharaja Sayajirao University of Baroda, India MS in Zoology (1991), Maharaja Sayajirao University of Baroda, India MS in Public Health (2012), University of Miami Neuroscience Fellowship (2003), University of Miami Research Interests: Dr. Raval investigates: Sex differences in stroke outcomes, particularly post-menopausal vulnerability due to estrogen decline Nicotine and oral contraceptive synergies in worsening ischemic injury in females Therapeutic strategies like estrogen receptor beta agonists and whole body vibration for cognitive recovery Awards & Honors: Best Poster Award (Basic Science), University of Miami Women's Health Research (Year not specified) American Heart Association Young Scientist Award (2013) ICMR Fellowship (New Delhi, India) Gujarat Government PhD Fellowship (India) Professional Contributions: Dr. Raval serves as a peer reviewer for journals like Stroke , Journal of Neuroinflammation , and grant review panels including the NIH and American Heart Association. She leads studies in the Peritz Scheinberg Cerebral Vascular Disease Research Laboratory, collaborating on translational research to advance stroke therapies.

• Immunology
• Infection Immunology
• Respiratory Viral Infections
• +۶ مورد دیگر

Prof. Dunja Bruder is a university professor of infection immunology at the Institute of Medical Microbiology and Hospital Hygiene at Otto von Guericke University (OVGU) and Head of the Immune Regulation Research Group at the Helmholtz Center for Infection Research (HZI). She obtained her PhD in immunology from TU Braunschweig and completed postdoctoral training at Harvard Medical School and Yale University School of Medicine. Her research focuses on immune regulation in respiratory viral infections, chronic inflammatory lung diseases, and mucosal immunity. She leads two research groups and holds leadership roles in the Research Focus on Respiratory Viral Infections (HZI), the CRC 854, and the Otto-von-Guericke Graduate Academy. Her work integrates innate and adaptive immunity, with particular emphasis on T-cell responses, microbiota interactions, and vaccine development. She also contributes to teaching in master's programs and human medicine. Education Bachelor/Master: Biotechnology, Technical University of Braunschweig PhD (1996–1999): Immunodominance and T-cell responses to bacterial toxins, HZI Habilitation (2009): Immunology, Hannover Medical School Research Interests Bruder’s work examines immune responses to respiratory pathogens like influenza and SARS-CoV-2, the role of epithelial-T-cell interactions in asthma, and the modulation of immune defenses by microbiota. She investigates vaccine strategies using defective interfering particles and antigen-targeting approaches. Her studies also address antitumor immune training via viral infections and metabolic control of T-cell function in asthma endotypes. Key Contributions She pioneered the analysis of alveolar macrophage dysfunction in chronic lung diseases and demonstrated the protective role of IL-33 in microbiota-mediated antibacterial defenses. Her team developed predictive models for influenza infection using hematological data and identified CD47 as a critical regulator of antiviral macrophage activity. Labs/Teams Leads the Immune Regulation Group at HZI and collaborates with the Institute of Medical Microbiology at OVGU. Active in the HZI’s Respiratory Viral Infections Research Focus and the CRC 854.

Manuel Salinasمشاهده پروفایل

دانشیار

• Biomechanics
• Thermal and fluid systems
• Engineering education
• +۳ مورد دیگر

Manuel Salinas, Ph.D., is an Associate Professor in the Department of Engineering at Nova Southeastern University's College of Computing and Engineering. He teaches core and biomedical engineering courses while mentoring students in honors theses and independent research. Postdoctorate, Harvard Medical School (2015) Ph.D. and B.S., Florida International University His research focuses on biomechanics and thermal/fluid systems, particularly how hemodynamic forces influence vascular inflammation, tissue engineering, and disease mechanisms. He develops bioreactor models for atherosclerosis and vasculitis studies, exploring oscillatory shear stress effects on stem cell differentiation and valvulogenesis. Recent work includes computational and experimental studies on vascular erosion, aortic valve tissue engineering, and mechanical stress in periodontitis-related osteoclast differentiation. Key collaborators include researchers from Harvard Medical School and Florida International University. Scientific Awards: Outstanding PhD Graduate Award (CEC, 2015) FIU Academy of Leaders Induction (2014) NIH Fellowship (2010) Alpha Eta Mu Beta Induction (2009) Salinas serves as a research mentor and NSU CEC representative for ASEE. He has led 28 courses in biomedical engineering, biomechanics, and physiological systems modeling. Current projects include Oticon-funded reverse engineering education initiatives.

Ping Zhouمشاهده پروفایل

دانشیار

• Neurovascular Biology
• Alzheimer's Disease
• Cerebral Amyloid Angiopathy
• +۵ مورد دیگر

Ping Zhou is an Associate Professor of Research in Neuroscience at the Brain and Mind Research Institute, Weill Cornell Medical College, where he has been conducting groundbreaking research since 2013. His work focuses on understanding the molecular mechanisms underlying neurovascular dysfunction in Alzheimer's disease and other neurodegenerative conditions. Dr. Zhou received his Ph.D. from the University of Illinois at Urbana-Champaign in 1991 and completed his undergraduate studies at Nanjing University in China in 1982. His educational background provided the foundation for his extensive research career in neuroscience and neurovascular biology. Dr. Zhou's research interests center on neurovascular coupling, cerebral amyloid angiopathy, and mitochondrial mechanisms of neuroprotection. His laboratory investigates how ApoE4 contributes to neurovascular dysfunction, the role of border-associated macrophages in white matter injury, and the neuroprotective functions of prohibitin through S-nitrosylation. His work bridges basic molecular mechanisms with translational applications for treating neurodegenerative diseases. Analysis of Dr. Zhou's recent publications reveals a strong focus on the intersection of neuroinflammation and neurovascular dysfunction in Alzheimer's disease. His work increasingly incorporates single-cell and epigenetic approaches while maintaining a strong emphasis on mitochondrial function and oxidative stress pathways. The research shows a clear progression from basic mechanisms of neurovascular coupling to more complex investigations of macrophage interactions and genetic risk factors like ApoE4. Dr. Zhou currently serves as Principal Investigator on the NINDS-funded project "Role of prohibitin nitrosylation in its neuroprotective functions" (2022-2027) and as Co-Investigator on two additional NINDS grants: "ApoE4, neurovascular injury and cognitive impairment" (2022-2027) and "Dietary sodium, neurovascular dysfunction and cerebrovascular risk" (2021-2026). These projects reflect his continued focus on molecular mechanisms of neuroprotection and vascular contributions to cognitive impairment. While the available information doesn't specify his laboratory structure, Dr. Zhou's extensive publication record spanning over two decades suggests leadership of a productive research team focused on neurovascular biology and neurodegeneration mechanisms.

Joseph M Castellanoمشاهده پروفایل

دانشیار

• Aging
• Alzheimer's Disease
• Blood-Brain Barrier
• +۱۲ مورد دیگر

Joseph M. Castellano is an Associate Professor in Neuroscience and Neurology at the Icahn School of Medicine at Mount Sinai. His research focuses on molecular mechanisms underlying aging and Alzheimer's disease (AD) pathogenesis, particularly the role of blood-CNS interactions in regulating synaptic and neuroimmune function. PhD from Washington University in St. Louis Postdoctoral training at Stanford University His work spans several key areas: Aging , Alzheimer's Disease , Microglia , Immunology , and Stem Cells . Notable contributions include demonstrating how APOE4 impedes amyloid-beta clearance and identifying youth-associated peripheral factors like TIMP2 that reverse brain aging. Recent publications highlight his exploration of neuroimmune regulation via blood-borne factors, proteomic changes linked to APOE genotype, and therapeutic strategies targeting aging-associated conditions. His lab employs advanced techniques like microdialysis and in vivo imaging to study these mechanisms. 2021 ISMMS ADRC Developmental Project Grant 2020 Black Family Stem Cell Institute Pilot Award 2018 Katz and Martin Friedman Brain Institute Research Scholar 2016 K99/R00 Pathway to Independence Award (NIA/NIH) Dr. Castellano's research bridges fundamental aging biology with translational neurodegeneration studies, supported by grants from NIH and private foundations. His lab continues to investigate how systemic factors influence brain health, aiming to develop novel AD interventions.

Sean Palecekمشاهده پروفایل

استاد

• Biomedical Engineering
• Stem Cell Research
• Tissue Engineering
• +۴ مورد دیگر

Sean Palecek is the Milton J. and A. Maude Shoemaker Professor in the Department of Chemical and Biological Engineering at the University of Wisconsin–Madison. His research focuses on engineering platforms to regulate human pluripotent stem cell (hPSC) differentiation for cardiovascular and neurovascular applications. He is based in 3637 Engineering Hall and leads the Palecek Lab, which develops innovative methods for stem cell fate specification and tissue engineering. BChE, Chemical Engineering, University of Delaware MS, Chemical Engineering, University of Illinois at Urbana-Champaign PhD, Chemical Engineering, Massachusetts Institute of Technology Postdoc., Molecular Genetics and Cell Biology, University of Chicago His research integrates stem cell biology with biomaterials and engineering principles to generate functional cardiomyocytes, brain endothelial cells, and mural cells. His lab leverages Notch3 signaling, proteomics, and organoid systems to model diseases and develop regenerative therapies. Current projects emphasize hypoimmunogenic cardiac organoids, vascular grafts, and blood-brain barrier models for neurotherapeutic screening. The 15 most recent articles highlight his work in human pluripotent stem cell (hPSC) differentiation to cardiomyocytes, endothelial cells, and brain mural cells. Key trends include organoid manufacturing , neurovascular unit modeling , metabolic profiling , and biomaterials for cell transport . His team explores Notch3 activation, SNRK signaling, and transcriptional analysis to optimize cell therapy and disease models. Scientific contributions include the Milton J. and A. Maude Shoemaker Professorship, a named chair recognizing his leadership in chemical and biological engineering. For more details, visit his lab’s website: Palecek Lab .

Eiron Cudabackمشاهده پروفایل

دانشیار

• Neurodegenerative Diseases
• Neuroinflammation
• Molecular Mechanisms of Disease
• +۶ مورد دیگر

Eiron Cudaback serves as an Associate Professor in DePaul University's College of Science and Health, teaching core courses including Integrated Human Anatomy and Physiology, Molecular Mechanisms of Human Disease, and Introduction to Pharmacology as a full-time faculty member. His academic credentials include: PhD in Health Sciences and Neuroscience Dr. Cudaback's research centers on neurodegenerative diseases with emphasis on APOE-driven neuroinflammation. His laboratory investigates how APOE ε4—the strongest genetic Alzheimer's risk factor—dysregulates microglial and astrocyte function, employing biochemical, in vitro , and in vivo approaches to uncover molecular pathways in Alzheimer's, Parkinson's, MS, and TBI. Key focus areas include chemokine signaling, neuroimmune crosstalk, and genetic modulation of disease progression. Analysis of his 2014-2021 publications reveals consistent exploration of APOE genotype effects on neuroinflammatory mechanisms, spanning from molecular studies of glial chemokine production to behavioral outcomes in transgenic models. His work demonstrates translational connections between genetic risk factors, innate immune responses, and neuropathology across multiple neurodegenerative conditions. No student advising details or research grants were specified in available materials. He leads an active research laboratory utilizing multidisciplinary techniques to dissect neuroinflammatory pathways in neurodegeneration, with collaborations examining microglial depletion effects, APOE-neprilysin interactions, and dopamine-neurodegeneration relationships in disease models.

• Diabetes Mellitus
• Beta-cell Secretion
• Clinical Trials
• +۵ مورد دیگر

Professor Steve Luzio holds a Personal Chair in Biomedical Sciences at Swansea University, based in the Faculty of Medicine, Health and Life Science. He leads a nationally accredited clinical laboratory focused on diabetes diagnostics and research, collaborating with pharmaceutical and medical device industries globally. His research spans diabetes pathophysiology, clinical trial methodologies, and diagnostic innovations, with particular emphasis on β-cell function, diabetes complications, and non-invasive glucose monitoring technologies. He has co-authored over 150 peer-reviewed articles and 200 conference abstracts. Recent work includes evaluations of diabetes prevention programs, non-invasive glucose monitoring devices, and immunotherapies for type 1 diabetes in adolescents. He also investigates health disparities in diabetes management within prison populations and retinopathy screening compliance. Luzio oversees the Swansea University Medical School’s diabetes laboratory, contributing to Welsh and international research networks. His work bridges clinical practice and innovation, with a focus on improving diagnostic accuracy and patient outcomes in diabetes care.

Adam J. Caseمشاهده پروفایل

دانشیار

• Psychological Trauma
• Post-Traumatic Stress Disorder (PTSD)
• Redox Biology
• +۴ مورد دیگر

Adam J. Case, PhD, is an Associate Professor in the Department of Psychiatry and Behavioral Sciences and affiliated with the Department of Medical Physiology at Texas A&M University School of Medicine. His research focuses on the bidirectional interactions between the brain and immune system, particularly how psychological trauma like PTSD influences autoimmune disorders and cardiovascular diseases, and vice versa. He holds a BS in Biochemistry (2004) and a PhD in Free Radical and Radiation Biology (2011) from the University of Iowa, followed by a postdoctoral fellowship at the University of Nebraska Medical Center (2012-2015). Dr. Case’s work integrates redox biology, immunology, and physiology to explore trauma-induced inflammation and its systemic consequences. He has received the 2020 Distinguished Graduate Student Mentor Award from the University of Nebraska Medical Center. His teaching interests include Redox Biology, Immunology, and Scientific Communication. His research highlights include investigations into IL-6’s role in trauma-cardiovascular links, beta-adrenergic signaling as an autoimmune therapeutic target, and hemoglobin’s redox-sensitive regulation of T-lymphocytes. He actively contributes to societies such as the Society for Biological Psychiatry and the American Physiological Society. Dr. Case’s lab explores neuroimmune mechanisms underlying PTSD and seeks translational therapies. His recent articles emphasize mitochondrial ROS regulation, T-lymphocyte neurotransmission dynamics, and trauma-induced inflammatory pathways.

• Alzheimer's Disease
• Huntington's Disease
• Parkinson's Disease
• +۵ مورد دیگر

Frank Longo is a Professor in the Department of Neurology and Neurological Sciences at Stanford University School of Medicine. He actively teaches across the neuroscience curriculum including Medical Scholars Research (NENS 370), Directed Reading courses (NEPR 299/NENS 299), Graduate Research (NENS 399/NEPR 399), and Early Clinical Experience (NENS 280), demonstrating his commitment to training the next generation of neuroscientists and clinicians. His research program centers on neurodegenerative diseases with particular emphasis on Alzheimer's, Huntington's, and Parkinson's diseases. Longo investigates the therapeutic potential of neurotrophin receptor modulation (especially p75NTR and TrkB) using small-molecule ligands like LM11A-31, exploring mechanisms spanning protein aggregation, synaptic dysfunction, neuroinflammation, and metabolic dysregulation. His work bridges molecular neuroscience with translational applications, utilizing advanced techniques including transcriptomics, neuroimaging, and biomarker analysis in cellular and animal models. Analysis of his 2024-2025 publications reveals consistent focus on targeting neurotrophin pathways to reverse disease phenotypes. Key themes include: 1) Development of small-molecule modulators for p75NTR/TrkB to rescue synaptic and cognitive deficits; 2) Biomarker discovery for early detection (e.g., plasma Aβ42/Aβ40 ratios); 3) Mechanistic studies on autophagy, white matter changes, and metabolic dysfunction; and 4) Leadership in collaborative initiatives like the TREAT-AD Center for target validation. His research demonstrates both breadth across neurodegenerative disorders and depth in neurotrophin signaling mechanisms. Through undergraduate and graduate research courses, Longo mentors students in neuroscience research despite no specific advisees being listed. His extensive publication record and involvement in multi-institutional consortia (including EU-US task forces) indicate significant research funding and national leadership in neurodegenerative disease research, though specific grant details aren't provided in the source material.

Ting Duمشاهده پروفایل

استادیار

• Neuroscience
• Neurology
• Brain Fluid Homeostasis
• +۷ مورد دیگر

Ting Du, M.D., Ph.D., is an Assistant Professor in the Department of Neurology at the University of Rochester Medical Center's School of Medicine and Dentistry. She leads the Du Lab, focusing on brain fluid homeostasis and its role in neurological diseases. Faculty: Assistant Professor, Department of Neurology Institution: University of Rochester Medical Center Lab: Du Lab Collaborations: Nedergaard Lab, Center for Translational Neuromedicine Research Interests: The Du Lab investigates cerebrospinal fluid (CSF) dynamics, glymphatic system function, and cervical lymphatic vessels in Alzheimer's disease, hydrocephalus, and traumatic brain injury. Key projects include: Restoring cervical lymphatic function to improve CSF drainage in aging Characterizing the subarachnoid lymphatic-like membrane (SLYM) in trauma Probing choroid plexus regulation in hydrocephalus Publication Trends: Recent work spans Neuroscience , Neurodegeneration , and Biophysics , with emphasis on: Glymphatic dysfunction in Alzheimer's Arterial pulsation-driven CSF flow Aquaporin-4's role in interstitial fluid Machine learning-based flow quantification Lab Affiliations: The Du Lab is part of the Neuroscience Ph.D. Program and Pathology - Cell Biology of Disease program, collaborating with experts in neuroimaging , fluid dynamics , and neurotherapeutics .

Karen D. Parfittمشاهده پروفایل

استاد

• Neuroscience
• Synaptic Physiology
• Synaptic Plasticity
• +۷ مورد دیگر

Karen D. Parfitt is Professor of Neuroscience and Chair of the Neuroscience Department at Pomona College , where she has been a faculty member since 1994. Her research is centered on synaptic transmission and plasticity in the hippocampus, with a focus on the molecular mechanisms of memory and their disruption in neurodegenerative diseases such as Alzheimer’s Disease. She employs both mouse and Drosophila models to investigate these processes. Her educational background includes a Ph.D. from the University of Colorado Health Sciences Center and a Bachelor of Science from Cornell University. She teaches core neuroscience courses such as Neurobiology with Lab, Neuropharmacology, and Senior Experimental Thesis in Neuroscience. Research Interests: Synaptic Physiology and Plasticity Molecular Mechanisms of Neurotransmitter Release Neurobiology of Alzheimer’s Disease Effects of Exercise on Synaptic Plasticity Neurobiology of Aging Her recent publications reveal a strong trend in exploring how secreted amyloid precursor protein-alpha modulates long-term potentiation (LTP), how exercise influences dopaminergic transmission and synaptic plasticity, and how inflammatory pathways contribute to tauopathy. These works span molecular, cellular, and behavioral neuroscience, reflecting a multidisciplinary approach. Scientific Awards and Honors: President, Faculty for Undergraduate Neuroscience (2000–2001) National Institute of Aging Academic Research Enhancement Award (1998) National Science Foundation Instrumentation Grant (1995) Faculty for Undergraduate Neuroscience Service Award (2011) Mellon Partnership Grant (2009) Multiple Pomona College Faculty Grants (1998, 2014, 2018) She has advised numerous undergraduate students, many of whom are co-authors on her publications, highlighting her commitment to mentored research. She has secured sustained funding from the NSF, NIH, NIA, and AFAR, supporting both instrumentation and research. Dr. Parfitt leads an active laboratory in Seaver Biology, focusing on electrophysiological and molecular analyses of synaptic function.