
معرفی
Neil Hogg is a Professor in the Department of Biophysics at the Medical College of Wisconsin (MCW), serving as Associate Dean of Academic Affairs in the Graduate School of Biomedical Sciences. He also holds leadership roles as Vice Chair for Education in Biophysics and Director of the Redox Biology Program. His academic journey includes a BSc (honors) in Biochemistry from the University of Leeds, followed by an MSc and PhD in Molecular Biochemistry and Biological Chemistry from the University of Essex, UK. Postdoctoral training in Free Radical Biology at MCW preceded his faculty appointment in 1996.
Education:
- BSc (Honors), Biochemistry, University of Leeds, 1987
- MSc, Molecular Biochemistry, University of Essex, 1989
- PhD, Biological Chemistry, University of Essex, 1993
- Postdoctoral Fellow, Free Radical Biology, MCW, 1994–1996
Research Interests: Dr. Hogg’s work focuses on nitric oxide (NO) signaling mechanisms, particularly its role in cellular signaling and disease pathogenesis. Key areas include:
- NO interactions with proteins and their impact on gene expression
- Pathophysiology of sickle cell disease (SCD), including hemolysis-induced vascular dysfunction
- Nitrite reduction pathways and their role in hypoxia-driven vasodilation
Publications: Recent work highlights NO’s role in β-cell regulation, SCD vascular injury, and nitrite-dependent cytoprotection. Articles emphasize translational applications in therapies for anemia and vascular diseases.
Awards: No explicit awards listed, but his leadership roles and extensive publications reflect scholarly recognition.
Advising & Grants: Supervised seven graduate students and multiple postdoctoral fellows. Leads the Redox Biology Program and directs the National Biomedical EPR Center, fostering collaborative research in redox biology and biophysics.
Labs/Teams: Principal Investigator in the Biophysics Department, with affiliations to the Redox Biology Program and National Biomedical EPR Center, advancing interdisciplinary research in nitric oxide biology and vascular diseases.
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