Un J. Kang, MD is the Founders Professor of Neurology at NYU Grossman School of Medicine and Professor in the Department of Neuroscience and Physiology. He serves as Director of Translational Research at the Marlene and Paolo Fresco Institute for Parkinson's and Movement Disorders and Co-Director of the Parekh Center for Interdisciplinary Neurology. His clinical practice focuses on Parkinson's disease and related movement disorders including tremor, dystonia, and Huntington's disease. Dr. Kang earned his MD from Johns Hopkins University in 1982, completed residency in Neurology at NY Presbyterian - Columbia University Medical Center in 1986, and specialized in Movement Disorders through fellowship training at the same institution in 1987. His research investigates the pathogenesis of Parkinson's disease and mechanisms of therapy, with particular focus on basal ganglia circuit plasticity, neural adaptation to dopamine loss, and development of biomarkers for neurodegenerative disorders. He pioneered experimental gene therapy approaches for Parkinson's disease and now directs the Kang Lab where his team studies how brain circuitry is perturbed by dopamine loss and pharmacological treatment in mouse models. Dr. Kang's research portfolio includes multiple NIH-funded projects examining striatal cholinergic interneuron dysfunction, freezing of gait mechanisms, and biomarker development for Parkinson's disease. His laboratory has published extensively on Parkinson's disease mechanisms with 178 publications to date, including significant recent work on alpha-synuclein detection methods and striatal pathway alterations in Parkinsonian motor deficits. Before joining NYU Langone in 2019, Dr. Kang created and led a multidisciplinary research team at the University of Chicago for two decades and previously served as chief of the movement disorders division at Columbia University. He leads the BioFIND consortium as principal investigator, banking biospecimens for Parkinson's disease biomarker discovery.
Anirban Paul is an Associate Professor in the Department of Neuroscience and Experimental Therapeutics at Pennsylvania State University, affiliated with the Penn State Neuroscience Institute. His research focuses on cellular and molecular mechanisms of GABAergic inhibitory circuits, with particular emphasis on interneuron biology and its implications in neurological disorders. Dr. Paul's research spans multiple neuroscience domains, with primary focus on GABAergic inhibitory circuits and interneuron biology. His work investigates how specific neuron subtypes, particularly Chandelier cells and cortical interneurons, contribute to brain function and dysfunction. He has made significant contributions to understanding the role of these cells in schizophrenia, Alzheimer's disease, and other neurological conditions. His research integrates molecular, cellular, and systems-level approaches to uncover fundamental mechanisms of neural circuit assembly, plasticity, and function. Key areas include RNA regulation in neuronal development, transcriptomic subtypes of inhibitory neurons, and cell-type specific vulnerabilities in neurodegenerative diseases. His research portfolio demonstrates consistent productivity with publications spanning from 2003 to 2025, showing an evolving focus from basic molecular neuroscience to translational research in neurological disorders. Recent work emphasizes single-cell analysis techniques and the role of specific interneuron populations in disease mechanisms, particularly in schizophrenia and Alzheimer's disease. His publications appear in high-impact neuroscience journals including Neuron, BMC Biology, and Frontiers in Cellular Neuroscience. Dr. Paul has received the NARSAD Young Investigator Award (2018), recognizing his promising research in neuroscience. His scientific contributions have been supported by multiple competitive grants from prestigious organizations including the National Institute on Aging (NIA) and the Brain and Behavior Research Foundation. He serves as Principal Investigator on multiple active research projects, including two major grants from the National Institute on Aging focused on cell-type specific risk and resilience in Alzheimer's disease and aging (2021-2024 and 2024-2026), as well as previous projects from the Brain and Behavior Research Foundation investigating Chandelier cells in schizophrenia. His research program demonstrates sustained funding and scientific leadership in the field of interneuron biology and its clinical implications.
Marc V Fuccillo is an Associate Professor of Neuroscience at the Perelman School of Medicine, University of Pennsylvania, where he leads a research laboratory focused on understanding the neural circuit mechanisms underlying behavioral control. His work bridges molecular, synaptic, and behavioral approaches to investigate how striatal circuits regulate mouse behavior from simple motor patterns to complex goal-directed actions. Fuccillo holds dual appointments in the Neuroscience and Cell and Molecular Biology Graduate Groups at Penn and maintains an active laboratory investigating the synaptic and circuit basis of neuropsychiatric disorders. Education: B.A. in Molecular and Cellular Biology and Music Performance (Violin) from Brown University (1998) Ph.D. in Developmental Genetics from New York University School of Medicine (2007) M.D. from New York University School of Medicine (2008) Fuccillo's research centers on the synaptic and circuit mechanisms of behavioral control, with particular emphasis on striatal circuits. His laboratory employs a range of technologies including mouse genetics, in vitro electrophysiology, in vivo imaging, and quantitative behavioral analysis to explore how neural circuits of the striatum regulate behavior and how disruptions in these circuits contribute to neuropsychiatric disorders. His work has particularly focused on autism-associated abnormalities in behavioral control, examining how synaptic adhesion molecules like neuroligins and neurexins shape circuit function and behavior, with significant findings regarding D1 dopamine receptor positive medium spiny neurons in the nucleus accumbens. Analysis of Fuccillo's recent publications reveals a strong focus on striatal circuit function across multiple dimensions. His work spans molecular neuroscience (examining synaptic adhesion molecules), cellular physiology (studying specific neuron types in striatal circuits), systems neuroscience (mapping circuit connectivity), and behavioral neuroscience (quantifying motor learning and decision-making). A unifying theme is how disruptions in specific molecular pathways lead to circuit-level abnormalities that manifest as behavioral phenotypes relevant to neuropsychiatric disorders, with particular attention to autism, OCD, and schizophrenia models. Scientific Recognition: Publications in high-impact journals including Nature Neuroscience, Current Biology, Cell Reports, and Neuron Research supported by multiple NIH grants including NIMH F32, NIMH K01, and HHMI Gilliam Fellowship awards for lab members Fuccillo actively mentors a diverse group of trainees including postdoctoral fellows, graduate students, and undergraduates. His laboratory has produced numerous successful alumni who have gone on to faculty positions, medical residencies, and graduate programs at prestigious institutions. His mentoring approach emphasizes technical skill development across multiple neuroscience disciplines while fostering independent scientific thinking. Current research in his lab is supported by NIH funding focused on understanding the molecular architecture of striatal circuits and their role in behavioral control, with three major research directions exploring molecular logic of striatal circuits, circuit mechanisms of behavioral control, and striatal dysfunction in neuropsychiatric disease models. The Fuccillo Laboratory operates within the Department of Neuroscience at the University of Pennsylvania, with access to state-of-the-art facilities for molecular, electrophysiological, imaging, and behavioral neuroscience research. The lab maintains active collaborations with other neuroscience research groups at Penn and beyond, creating a rich intellectual environment for studying the neural basis of behavior. Current research directions include investigating whether there is a molecular logic to striatal circuit composition, how striatal circuits shape behavioral control, and what mouse models of autism, schizophrenia, and OCD can reveal about striatal circuit dysfunction in disease pathophysiology.
John D. Murray is the Gregg L. Engles Associate Professor of Psychological and Brain Sciences at Dartmouth College and an Adjunct Associate Professor of Psychiatry at Yale School of Medicine. He holds a PhD in Physics from Yale University (2013) and a BS in Physics and Mathematics from Yale (2006). His research focuses on computational neuroscience and computational psychiatry, with secondary appointments in Physics and Neuroscience at Yale until 2023. His work integrates computational modeling, neuroimaging, and systems neuroscience to study decision-making processes, cortical organization, and psychiatric disorders. Collaborators include prominent researchers like Dr. John Krystal and Dr. Anticevic. Research interests include hierarchical brain organization, neuroimaging analysis techniques, and pharmacological effects on neural circuits. His lab (Murray Lab) develops computational tools like PsychRNN for cognitive task modeling. Notable contributions include linking transcriptomic data to neuroimaging patterns and modeling LSD’s effects on brain topography. He has been featured in YaleNews and Nature Communications for innovations in mapping mental illness variability and neural circuit dynamics. Grants and collaborations span translational neuroscience, addiction, and PTSD research through partnerships with Yale’s Center for Biomedical Data Science and VA National Center for PTSD. His interdisciplinary approach bridges physics, computer science, and clinical psychiatry to advance understanding of brain function and dysfunction.
Kevin M. Franks is an Associate Professor of Neurobiology at Duke University, where he investigates how the olfactory system forms neural representations of sensory environments. His work focuses on functional neural circuits in the olfactory bulb and piriform cortex, using techniques like in vivo recordings, optogenetics, and behavioral assays. His research explores Neural circuit dynamics and plasticity Odor coding mechanisms Role of recurrent circuitry Integration of sensory modalities Recent publications highlight his contributions to understanding cortical odor representations, developmental neural connectivity, and cross-modal interactions. Awards include the 2024 Don Tucker Finalist recognition. He teaches advanced neuroscience courses at Duke, including Neurobiology research and concepts in neuronal systems.
Dr. Sabine Krabbe is a Group Leader at the German Center for Neurodegenerative Diseases (DZNE) in Bonn, Germany, where she leads research on neural circuit mechanisms underlying adaptive learning and state-dependent decision-making. Her work integrates neuroscience, molecular biology, and behavioral approaches to understand how internal states influence behavior and how these processes are disrupted in neurological disorders. Dr. Krabbe's research focuses on the interactions between midbrain circuits of the substantia nigra and ventral tegmental area with their output structures such as the striatum and amygdala. She investigates how these networks integrate internal states with environmental cues to produce appropriate behavioral responses. Her laboratory employs state-of-the-art techniques including deep-brain calcium imaging at single-cell resolution in mice, opto- and pharmacogenetic manipulations, anatomical tracings, and molecular approaches to characterize neural circuit elements in detail. Her recent publications reveal significant insights into amygdala interneuron plasticity during fear learning, brain-wide representational drift in memory consolidation, and the molecular mechanisms underlying Parkinson's disease progression. Her work demonstrates how activity patterns within specific neural circuits change in early stages of neurodegenerative diseases and how this dysfunction contributes to cognitive deficits and emotional disturbances. Dr. Krabbe is actively involved in the neuroscience community, organizing the BonnBrain Conference 2026 and sharing research through social media platforms. She has established herself as an emerging leader in the field of systems neuroscience with a particular focus on the neural basis of emotional states and decision-making processes.
Michael J. Frank is the Edgar L. Marston Professor of Psychology and Professor of Brain Science at Brown University's School of Cognitive, Linguistic, and Psychological Sciences. He holds academic affiliations with the Carney Institute for Brain Science and specializes in cognitive neuroscience, computational neuroscience, and decision-making processes. Frank earned his Ph.D. in Neuroscience & Psychology from the University of Colorado at Boulder in 2004, and joined Brown University in 2011 after serving as a Professor at the University of Arizona. His research integrates computational modeling and experimental methods to explore neural mechanisms underlying reinforcement learning, decision-making, and cognitive control, with a focus on prefrontal cortex-basal ganglia interactions and dopamine modulation. Frank's honors include the Troland Research Award (2021), Kavli Fellowship (2016), and the Cognitive Neuroscience Society Young Investigator Award (2011). He is an editor for eLife, Behavioral Neuroscience, and the Journal of Neuroscience. His lab, based at http://ski.clps.brown.edu, investigates topics such as neural circuit models of cognitive control, neuropsychological testing, and translational applications of computational models in psychiatry. Frank's research emphasizes interdisciplinary approaches, combining behavioral experiments, neuroimaging (fMRI, EEG), and pharmacological studies to dissect brain-behavior relationships. Key findings include insights into dopamine's role in motivation, decision-making deficits in schizophrenia, and computational phenotyping of mental disorders. His work bridges basic science and clinical applications, aiming to inform therapeutic strategies for neurological and psychiatric conditions.
Daniel M. Wolpert is a Professor of Neuroscience at Columbia University and Principal Investigator at the Zuckerman Institute. His research focuses on computational models of movement, integrating sensory cues and cognitive elements to understand motor control, memory, and rehabilitation strategies for cerebellar disorders. Key Research Areas: Sensorimotor integration, probabilistic inference, reinforcement learning, predictive modeling of movement, and aging effects on motor learning. Selected Awards: Royal Society Fellow (2012), Minerva Golden Brain Award (2010), Fulbright Scholarship (1992-1995). Recent publications highlight his work on contextual learning, motor memory formation, and the computational basis of sensorimotor uncertainty. His lab develops robotic interfaces to study human motor behavior and collaborates on clinical applications for movement disorders. Current opportunities include postdoctoral fellowships in sensorimotor control and decision-making.
Istvan Mody is a Professor at the University of California, Los Angeles (UCLA) with appointments in the Department of Neurology and Department of Physiology . His research focuses on synaptic signaling in health and disease, including mechanisms of GABAergic transmission, calcium homeostasis, and their roles in neurological disorders such as epilepsy, Alzheimer's disease, Huntington's disease, stress, alcoholism, and postpartum depression. He utilizes advanced techniques like patch-clamp electrophysiology, neuroanatomical and immunohistochemical methods, and molecular biology in animal models and human brain tissue . Research Interests: Dr. Mody investigates the physiology, pharmacology, and pathology of synaptic transmission and extrasynaptic receptor activation , with a particular emphasis on GABA(A) receptors and their subunit-specific modulation. His work explores how disruptions in excitation-inhibition balance contribute to neurological diseases, including mechanisms of tonic inhibition , calcium signaling , and neurosteroid interactions . He also studies the effects of chronic stress and hormonal fluctuations on neural excitability and behavior. Publications Trends: Recent studies highlight his work on gamma oscillations in Alzheimer's models, microglial dynamics , and rehabilitation strategies for stroke. His lab develops optical tools like dqGEVI for neuronal activity monitoring and investigates human brain organoids to model network dysfunction in epilepsy and intellectual disability. Laboratory Location: 635 Charles Young Dr S, Los Angeles, CA 90095, United States.
Chandan J Vaidya is a Professor in the Department of Psychology at Georgetown University, directing the Developmental Cognitive Neuroscience Laboratory (DCNL). His research focuses on cognitive neuroscience mechanisms underlying adaptive behaviors, particularly implicit learning, executive control, and their dysfunction in ADHD, ASD, and other developmental disorders. Using multidisciplinary methods including fMRI, behavioral testing, and genetic analysis, he investigates how dopamine systems, brain connectivity, and environmental factors influence cognitive processes. Primary appointment: Professor, College of Arts and Sciences - Department of Psychology Education: Ph.D. from Syracuse University Research interests include neurodevelopmental disorders, neuroimaging of cognitive control, and translational neuroscience. Recent work examines striatal connectivity changes in ADHD due to stimulant use, executive dysfunction subtypes in autism, and brain correlates of reward processing in obesity. Key findings highlight hyperconnectivity in ASD, dopamine genotype influences on executive function, and age-related changes in default mode networks. Ongoing studies explore transdiagnostic models of psychopathology and precision medicine approaches in neurodevelopmental disorders. Lab activities focus on translational research bridging basic neuroscience with clinical applications. Collaborations involve pediatric neurology, psychiatry, and computational modeling.
Kamal Sen is an Associate Professor in the Department of Biomedical Engineering at Boston University, serving as Director of the Natural Sounds and Neural Coding Laboratory and Director of Admissions and Recruitment for Master’s Programs. He holds a PhD and MA in Physics from Brandeis University and a BA in Physics from Bates College. His research focuses on understanding how neurons encode natural sounds, particularly in the auditory cortex. Key areas include neural coding efficiency, hierarchical auditory processing, and the role of learning in shaping receptive fields. He developed the BOSSA algorithm to address sound segregation challenges in noisy environments, with applications for hearing aid technology. Sen’s work integrates electrophysiological techniques with theoretical approaches from signal processing, information theory, and systems theory. His lab explores neural discrimination of behaviorally relevant sounds and models cortical processing dynamics using computational frameworks. Recent studies investigate parvalbumin neuron contributions to temporal coding and cortical noise reduction in complex auditory scenes. His publications span neural circuit modeling, fNIRS applications in BCI, and biomimetic algorithms for auditory scene analysis. Research highlights include exploring schizophrenia-related gene effects on neural circuits and developing 3D neurosphere models for Parkinson’s disease.
Professor Eva Kosek holds dual academic positions as Professor of Clinical Pain Research at Karolinska Institutet (since 2015) and Uppsala University (since 2020). She is affiliated with the Department of Clinical Neuroscience and leads the Mechanisms of Pain and Treatment Research Group . Her roles include senior consultant at Uppsala University Hospital's Pain Center. Education: MD from Uppsala University (1986), PhD from Karolinska Institutet (1996). Specializations: Rehabilitation Medicine (1998), Pain Relief (2001). Academic promotions: Associate Professor at Karolinska (2004), Full Professor (2015). Research Focus: Chronic pain mechanisms, neuroimmune interactions, fibromyalgia, and autoimmune pathways. Key projects include the RAFT trial (Rituximab for fibromyalgia autoantibody therapy) and the BACPAP consortium for low back pain phenotyping. She chairs IASP's Terminology Task Force, introducing the 'nociplastic pain' concept. Key Contributions: Discovered anti-satellite glial cell IgG antibodies in fibromyalgia patients, linked to symptom severity. Pioneered neuroimaging studies on pain modulation circuits. Authored over 300 publications, with recent work on cerebrospinal fluid biomarkers and lipid metabolite profiles. Awards: 2024 Roland Melzack Lecture Award from IASP. Recognized for redefining pain taxonomy and translational research. Grants: Active funding includes Swedish Research Council grants on fibromyalgia autoimmunity, environmental exposures, and neuroinflammation. Total grants exceed SEK 100M over her career. Labs/Teams: Leads a multidisciplinary team at Karolinska's Pain Research Center, collaborating internationally on translational pain studies. Active in biobanking initiatives like the Swedish Chronic Pain Biobank.
Ruben Portugues is a Professor of Brain Circuit Function and Dysfunction at the Institute of Neuroscience, Technical University of Munich (TUM). He is a full member of the Graduate School of Systemic Neurosciences (GSN), an associate and advisory board member of the Munich Center for Neurosciences (MCN), and leads a research group focused on understanding the neural basis of behavior. His lab uses larval zebrafish as a model organism to investigate sensorimotor control, decision-making, and motor learning through whole-brain imaging and circuit analysis. His research interests lie at the intersection of systems neuroscience and behavior. He investigates how brain circuits process sensory information, integrate it with motor output, and enable adaptive and flexible behavior. Key areas include the function of the cerebellum, heading direction networks, sensorimotor transformations, and the neural mechanisms of decision-making. His lab employs cutting-edge techniques including custom-built microscopes, behavioral assays, and computational analysis. The recent publications and preprints from his lab demonstrate a strong trend in decoding distributed neural circuits underlying navigation and decision-making in zebrafish. There is a clear focus on identifying specific brain regions (e.g., interpeduncular nucleus, cerebellum) and cell types involved in processing visual, motor, and spatial information. The work increasingly emphasizes whole-brain functional imaging and the emergence of cognitive-like representations such as allocentric heading direction. FENS-Kavli Network of Excellence (FKNE) PhD Thesis Prize (awarded to student Luigi Petrucco) Ruben Portugues actively mentors PhD students, including current advisees Luigi Petrucco, Ot Prat, and Shuhong Huang, and has successfully graduated Dr. Elena Dragomir and Dr. Vilim Štih. His lab engages in extensive collaborations, hosts visiting researchers, participates in teaching (e.g., CSHL Imaging Course, Cajal Course), and secures resources for advanced research. The lab is known for building its own microscopes and software, fostering technical innovation. The Portugues Lab operates as a dynamic, interdisciplinary team that combines experimental neuroscience with computational and engineering approaches. They regularly hold retreats, participate in scientific events, and contribute to community initiatives like the Munich Brain Day. The lab is preparing to relocate to the Department of Neurobiology and Behavior at Cornell University, marking a new phase in its research trajectory.
Alicia Che is an Assistant Professor of Psychiatry at Yale University School of Medicine and serves as Director of Graduate Admissions for the Interdepartmental Neuroscience Program. She joined the Yale Department of Psychiatry in 2021 after completing her postdoctoral fellowship with Dr. Natalia De Marco García at Weill Cornell Medical College and Dr. Gord Fishell at NYU. Her research is conducted through the Che Lab at Yale, where she investigates how early life experiences impact brain circuit assembly and mature function in models of psychiatric illness. Yale School of Medicine, Department of Psychiatry Interdepartmental Neuroscience Program Center for Brain & Mind Health Division of Molecular Psychiatry Wu Tsai Institute Yale Center for the Science of Cannabis and Cannabinoids Dr. Che earned her Ph.D. in Physiology and Neurobiology from the University of Connecticut in 2014, where she worked in the laboratory of Dr. Joseph LoTurco. She received her B.S. with triple majors in Biology, Physics, and Physical Chemistry from Pacific Lutheran University in Washington state in 2009. Her research focuses on understanding developmental trajectories following early life experiences to develop diagnostics and early interventions for psychiatric illnesses. Dr. Che's research examines how sensory inputs, social bonding, stress, and substance exposure impact brain development. She employs a multi-dimensional approach to assess transcriptional, circuit, neuronal activity, and behavioral changes across the entire developmental timeline. Her lab currently focuses on four specific areas: the role of oxytocin in social behavior development, circuit dysfunction in PTSD, early-life cannabinoid exposure effects, and the impact of early life stress on development and adulthood. She utilizes advanced techniques including mouse genetics, slice electrophysiology, and longitudinal in vivo 2-photon imaging on behaving animals. Her most recent publications demonstrate significant contributions to understanding neural circuit development, PTSD mechanisms, and the effects of early life experiences on brain function. Her work spans from molecular neuroscience to behavioral outcomes, with publications in top journals including Nature, Neuron, and Nature Communications. Her research has revealed important insights into how translaminar neuronal activity strengthens cortical columns, how oxytocin facilitates social touch development, and how PTSD affects brain transcriptomics. NARSAD Young Investigator Award (2020) K99/R00 Pathway to Independence Award from NINDS (2019) Dr. Che's work has significant implications for understanding and treating neurodevelopmental disorders, PTSD, and the consequences of early life experiences on mental health. She collaborates extensively with researchers across Yale and beyond, with frequent co-authorship with colleagues including Lin Lin, Alex Kwan, and Christopher Pittenger. Her research program bridges basic neuroscience with clinical applications, aiming to translate findings into potential interventions for psychiatric conditions.
Dr. Jason Yi is an Assistant Professor of Neuroscience at Washington University School of Medicine (WashU Medicine). His research focuses on understanding the molecular pathways that shape nervous system development and function, with particular emphasis on autism spectrum disorders (ASD). He leads the Yi Lab, which investigates the role of the ubiquitin ligase UBE3A in the brain and its implications for neurodevelopmental disorders. Dr. Yi received his BS in Biochemistry and Molecular Biology from Dickinson College in 2001 and his PhD in Pharmacology from Duke University in 2009. His laboratory is broadly interested in the molecular pathways that shape nervous system development and function, with the ultimate goal of understanding how dysfunction in these pathways contributes to disease. The current focus is on autism spectrum disorders (ASD), using genetic information from human patients to guide in vitro and in vivo experiments employing biochemical, genetic manipulation, cell biological, and microscopy techniques. Dr. Yi's research has significant clinical implications, particularly in understanding how UBE3A dysfunction relates to both Angelman syndrome (caused by lack of UBE3A activity) and autism (caused by excessive UBE3A activity). His lab discovered that a single phosphorylation event in UBE3A turns off its ubiquitin ligase activity, and that mutations in this site are linked to autism. This work bridges disease genetics with a mechanistic understanding of ASD neurobiology and aims to define developmental timepoints for ASD onset. Dr. Yi's research has been recognized with numerous prestigious awards: Ruth K. Broad Biomedical Research Foundation Predoctoral Fellowship (2006) F32 Kirschstein National Research Service Award (2011) Christina Castellana Postdoctoral Fellowship (2011-2014) The University of North Carolina Postdoctoral Award for Research Excellence (2015) Bridge to Independence Award, The Simons Foundation (2017) NARSAD Young Investigator Award, Brain and Behavior Research Foundation (2018) Whitehall Foundation Research Grant (2018) Alfred P. Sloan Foundation Research Fellowship (2019) Dr. Yi's research program is supported by significant grant funding from organizations including The Simons Foundation, Brain and Behavior Research Foundation, and the Whitehall Foundation. His work bridges basic molecular neuroscience with clinical implications for neurodevelopmental disorders, particularly autism spectrum disorders. Through his research, Dr. Yi is contributing to a deeper understanding of the molecular mechanisms underlying ASD, which may ultimately lead to new therapeutic approaches and interventions. The Yi Lab maintains a collaborative research environment focused on cutting-edge neuroscience techniques. The lab combines molecular, cellular, and genetic approaches to study UBE3A function and its role in neurodevelopment. Their work utilizes patient-derived genetic information to guide experimental approaches, ensuring clinical relevance to autism spectrum disorders. Dr. Yi is also actively involved in mentoring graduate students and postdoctoral fellows, contributing to the training of the next generation of neuroscientists.
