Teresa Anglada Pons

Teresa Anglada Pons is a Researcher in the Department of Cellular Biology, Physiology and Immunology at the Universitat Autònoma de Barcelona's Faculty of Biosciences. She specializes in molecular mechanisms of genome instability, particularly focusing on chromosome bridge resolution, DNA repair pathways, and the application of CRISPR/Cas9 technology to study mitotic processes.

Her research interests center on understanding how chromatin bridges form and resolve during cell division, and how defects in these processes lead to genome instability. She investigates the roles of key proteins like BRCA1 and 53BP1 in DNA repair mechanisms, with particular attention to age-related changes in these pathways. Her work bridges fundamental cell biology with potential clinical implications for cancer research and radiation biology.

Dr. Anglada Pons has been actively involved in multiple significant research projects since 2012, including studies on Serbian children exposed to radiation, mammography risk assessment, and chromatin bridge resolution mechanisms funded by organizations including Fundació La Marató de TV3, Consejo de Seguridad Nuclear, European Commission, and Institute of Health Carlos III.

Her scholarly contributions include multiple peer-reviewed publications in high-impact journals such as International Journal of Molecular Sciences, FASEB Journal, Seminars in Cell and Developmental Biology, Frontiers in Cell and Developmental Biology, and Aging. Her work has garnered substantial attention in the scientific community with citations across multiple platforms.

She completed her doctoral training in 2018 with a thesis examining the contribution of BRCA1 and 53BP1 to DNA break repair, supervised by Dr. Anna Genescà and Dr. Martín Flix. Her ongoing research continues to explore the intricate relationship between DNA repair mechanisms, chromosome segregation, and genome stability.