Susan Currie

Susan Currie is an Associate Professor/Reader at the Strathclyde Institute of Pharmacy & Biomedical Sciences, University of Strathclyde, where she leads research in cardiovascular signalling and pharmacology. She holds leadership roles including Director of Equality, Diversity and Inclusion and has previously directed international MPharm collaborations and served as Deputy Director of the MPharm programme. She is an elected Fellow of the Physiological Society (2017) and the Royal Society of Biology (2024), and chairs committees in national cardiovascular research societies.

Her research focuses on the role of CaMKII in cardiac disease, cardiotoxicity of drugs and medical devices (especially cobalt from implants), heart failure with preserved ejection fraction (HFpEF), and cardiovascular ageing. She utilizes in vivo models, human cell cultures, and advanced imaging to investigate molecular mechanisms of cardiac dysfunction.

Recent publications highlight trends in sex-specific cardiac responses, mitochondrial dysfunction, fibrosis, and excitation-contraction coupling defects, particularly in female models of disease. Her work bridges molecular pharmacology with translational safety screening and clinical implications.

• Fellow of the Royal Society of Biology (2024)
• Teaching Excellence Award (2023)
• Elected to Committee of British Society for Cardiovascular Research (2020)
• Fellow of the Physiological Society (2017)
• Ambassador for British Society of Cardiovascular Research (2017)

Susan Currie actively mentors PhD and undergraduate researchers, as evidenced by her co-authored publications with students. She leads externally and internally funded projects, including those on cobalt cardiotoxicity and mitochondrial damage in chemotherapy. She contributes to academic governance through editorial roles and peer review for research councils.

She leads the Cellular Basis of Disease research group (2021–2023) and collaborates on interdisciplinary projects such as CULTURE-GAPS in STEM and killifish models for multimorbidity. Her laboratory investigates both contractile and non-contractile cardiac cells, focusing on inflammation, fibrosis, and metabolic stress.