معرفی
Lluis Fajas Coll is a full professor at the University of Lausanne (UNIL), Faculty of Biology and Medicine, where he has served as director of the Department of Physiology since August 1, 2012. In 2016, he joined the UNIL Integrative Genomics Center, further expanding his research integration across disciplines.
His research focuses on the intersection of cellular energy metabolism with the cell cycle, particularly in the context of metabolic diseases and cancer. He and his team investigate how disruptions in lipid metabolism and energy homeostasis contribute to conditions such as obesity, diabetes, and cancer. A key finding from his work is the identification of the E2F1 protein as a critical regulator of pancreatic beta-cell function and insulin secretion, linking cell cycle regulation directly to metabolic control.
Lluis Fajas Coll's work is characterized by an integrative, fundamental research approach with strong clinical relevance. His studies utilize in vitro models and animal systems to explore how cell cycle regulators influence metabolic reprogramming in pathophysiological states.
His scientific leadership includes founding the Metabolism and Cancer Laboratory in Montpellier and leading research groups at the Cancer Research Institute and the Institute of Molecular Genetics before joining UNIL.
- Bachelor of Science in Biology, University of Barcelona (1991)
- Master’s in Biotechnology, Polytechnic University of Catalonia
- PhD in Biology, University of Barcelona (1995)
Throughout his career, Fajas has held key positions at prestigious institutions, including the Institut Pasteur in Lille, the Institute of Molecular Genetics in Montpellier, and Inserm, where he advanced from research fellow to research director by 2005. He has not received any explicitly mentioned scientific awards in the provided text. There is no information available about his advisees or grants. His lab is part of the Department of Physiology and the UNIL Integrative Genomics Center, focusing on molecular mechanisms linking metabolism and cell proliferation.
