معرفی
Konrad Zinsmaier is a Professor and Department Head at the University of Arizona, where he leads research on synaptic function and neurodegenerative mechanisms. His work combines molecular biology and genetics in Drosophila models to study synaptic plasticity, mitochondrial dynamics, and neuroprotective pathways.
His research focuses on mechanisms maintaining synaptic integrity, including roles of cysteine-string protein (CSP) in preventing neurodegeneration and DCAF12’s involvement in ubiquitination pathways. Key projects include modeling neuronal ceroid lipofuscinosis (CLN4) and investigating mitochondrial transport via Miro GTPases.
Publications highlight contributions to understanding ALS, Parkinson’s disease mechanisms, and synaptic vesicle recycling. His studies bridge basic neuroscience with disease modeling, emphasizing translational potential for neuroprotective therapies.
Teaching responsibilities include courses on neurogenetics, cellular neurobiology, and neuroscience methodology. Research also extends to cryopreservation techniques for primary neurons and olfactory receptor function in Drosophila.
Labs: Automated Analysis of Mitochondrial x-y-t Tracks focuses on real-time mitochondrial dynamics and signaling.
