
معرفی
Chi Zhou is an Assistant Professor at the University of Arizona, holding dual appointments in the School of Animal and Comparative Biomedical Sciences and the Department of Obstetrics and Gynecology. His research centers on placental and fetal endothelial biology, with a strong emphasis on sex-specific responses in preeclampsia and related pregnancy complications.
His research interests include:
- Pregnancy complications, particularly preeclampsia
- Placental biology and vascular development
- Fetal endothelial and vascular programming
- Sexual dimorphisms in fetal responses
- Role of microRNAs and AhR signaling in endothelial dysfunction
- Hypoxia and its impact on fetal development
His recent publications reveal a consistent focus on molecular mechanisms underlying preeclampsia, particularly using transcriptomic and functional assays in fetal endothelial cells. A recurring theme is the fetal sex-specific dysregulation of gene expression and signaling pathways, especially involving microRNA-29 and aryl hydrocarbon receptor (AhR) ligands like ITE. His work bridges reproductive physiology and cardiovascular development, with implications for long-term offspring health.
Scientific awards and recognitions include:
- ACE Award, Society for Reproductive Investigation (2022)
- American Heart Association Career Development Award (2019–2022)
- Outstanding Paper by an Associate Member Award, Perinatal Research Society (2019)
- SRI NIH Travel Grant (2019)
- AHA Postdoctoral Fellowship (2017–2018)
- Young Investigator Award, Perinatal Research Society (2016)
Dr. Zhou actively mentors students through honors theses and independent study courses. He has served as a reviewer for numerous journals including Biology of Reproduction, Journal of Clinical Medicine, and FASEB Journal. He has also contributed to professional societies such as the Society for Reproductive Investigation and the American Heart Association. His lab, accessible at https://sites.arizona.edu/chizhou-lab/, focuses on molecular mechanisms of endothelial dysfunction in pregnancy and long-term cardiovascular implications.
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