Marte Lie Høivik is a Professor at the University of Oslo's Institute of Clinical Medicine, Department of Gastroenterology and Hepatology. She serves as Senior Consultant and Head of the IBD Unit at Oslo University Hospital, leading the IBD research group and directing the IBSEN III study. Her research focuses on clinical and registry-based epidemiology in inflammatory bowel disease (IBD), with emphasis on clinical effectiveness, quality studies, prognostic research, and biomarker identification. Her work leverages large population-based cohorts like IBSEN to investigate disease course, treatment outcomes, and quality-of-life impacts. Recent publications reveal strong trends in risk prediction modeling, microbiome analysis, therapeutic drug monitoring, and real-world treatment effectiveness. Her team frequently examines understudied aspects like fatigue in remission, dietary impacts, and sex/gender differences in IBD outcomes using longitudinal cohort designs. Høivik actively supervises research within the IBSEN III cohort and collaborates extensively across Nordic countries on IBD epidemiology. Her team has published over 30 papers since 2021 in high-impact journals including Nature , Gastroenterology , and The Lancet Gastroenterology & Hepatology . She leads multiple research projects including the IBSEN III cohort study and NORDTREAT, with current funding supporting investigations into biomarkers, treatment strategies, and long-term disease outcomes in inflammatory bowel disease.
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Felix Sommerمشاهده پروفایل
پژوهشگر
- Microbiome
- Host-Microbiome Interactions
- Immunology
- +۶ مورد دیگر
Felix Sommer is a Researcher and Group Leader of the Functional Host-Microbiome Research group at the Institute of Clinical Molecular Biology (IKMB) of Kiel University and University Medical Center Schleswig-Holstein (UKSH). He heads the Systems Immunology Microbiome Platform and leads the Junior Research Group Functional Host-Microbiome Research and Systems Immunology, which was established in 2020. His work focuses on understanding the intricate interactions between the host, microbiome, and environmental factors in health and disease. Felix Sommer received his PhD in cell biology in 2010 from Kiel University and completed postdoctoral training in the lab of Fredrik Bäckhed in Gothenburg, Sweden, from 2010 to 2015 before joining IKMB. His research centers on host-microbiome interactions, particularly how the microbiome influences host metabolism (such as glycolysis) and immune responses, with implications for inflammatory bowel disease, cancer, and metabolic disorders. His work employs gnotobiotic in vivo conditional knockout models, functional in vitro organoid systems, and high-throughput 'omics' technologies including next-generation sequencing and metabolomics. Analysis of his recent publications reveals a strong focus on the metabolic aspects of host-microbiome interactions, particularly how microbial metabolites influence host immune responses and disease states. His work spans multiple disease models including inflammatory bowel disease, cancer, metabolic disorders, and aging, with a consistent theme of identifying specific molecular pathways that could be targeted therapeutically. Recent work has increasingly focused on hexokinase regulation, gut barrier function, and the therapeutic potential of microbiome modulation. Felix Sommer leads a research team that includes doctoral researchers Xue Gu, Hongshu Huang, Lea Järke, Xiangyu Meng, and Saskia Weber-Stiehl. His group is supported by funding from the German Research Foundation (DFG) for the project 'Role of DUOX2 in shaping the intestinal microbiota and effects on inflammation, cancer and metabolic disease' (SO1141/10-1), the Research Group 'miTarget – The Microbiome as a Target in Inflammatory Bowel Diseases' (FOR5042), and the Excellence Cluster 'Precision Medicine in Chronic Inflammation' (EXS2167). The Functional Host-Microbiome Research group operates within the Institute of Clinical Molecular Biology at Kiel University, utilizing specialized facilities including gnotobiotic models and advanced 'omics' technologies. The group aims to develop novel strategies for treating metabolic and inflammatory disorders by targeting the intestinal microbiome through nutritional or probiotic interventions, with a particular focus on understanding the molecular mechanisms underlying host-microbiome crosstalk.
Katherine T. Mills is an Associate Professor in the Epidemiology Department at Tulane University's School of Public Health and Tropical Medicine. Her research focuses on cardiovascular and renal disease epidemiology, implementation science, and health disparities, with particular emphasis on blood pressure control interventions and chronic kidney disease. Dr. Mills received her PhD in Epidemiology from Tulane University, completed a postdoctoral fellowship in cardiovascular disease epidemiology at Johns Hopkins University, earned an MSPH in Epidemiology from the University of North Carolina at Chapel Hill, and completed her undergraduate studies in Chemistry at Colorado College. Her research program spans multiple domains of public health and clinical epidemiology. Dr. Mills has established herself as a leader in studies examining the relationship between cardiovascular disease, kidney function, and health disparities. She has conducted extensive research on implementation science approaches to improve blood pressure control, particularly in underserved populations. Her work also investigates health outcomes in chronic kidney disease patients, including progression to end-stage renal disease, cardiovascular events, and mortality. Through her research, Dr. Mills has contributed significantly to understanding how social determinants of health impact cardiovascular outcomes and how community-based interventions can reduce health disparities. She has been particularly active in engaging Black churches as venues for cardiovascular health interventions. Analysis of Dr. Mills' recent publications reveals a strong focus on cardiovascular epidemiology, kidney disease outcomes, and implementation science for hypertension control. Her work frequently employs advanced statistical methods and large cohort studies, particularly the Chronic Renal Insufficiency Cohort (CRIC) study. She has published extensively on topics including blood pressure management, kidney disease progression, cardiovascular risk factors, and health disparities in minority populations. Her research often examines the intersection of social determinants of health with clinical outcomes. Dr. Mills has received numerous scientific awards including: 2020: Outstanding Achievement in Receiving Your First R01 Award 2018: Young Investigator Travel Award, NIH, NIGMS Seventh Biennial National IdeA Symposium of Biomedical Research Excellence (NISBRE) 2017: Sandra A. Daugherty Award for Excellence in Cardiovascular Disease for Hypertension Epidemiology, American Heart Association Council on Epidemiology and Prevention 2016: Award for Excellence in Research and Presentation by a Postdoctoral Fellow, Health Sciences Research Days, Tulane University 2015-2016: NIH/NHLBI T32 Cardiovascular Disease Epidemiology Training Program, Johns Hopkins University 2014: Dorothy R. LeBlanc Memorial Scholarship Award, Tulane University 2010-2012: Dean's Research Council Scholarship, Tulane University Dr. Mills has secured significant NIH funding for her research on blood pressure control interventions and health disparities. She has mentored numerous students and collaborated extensively with interdisciplinary teams across Tulane and other institutions. Her work on church-based interventions for cardiovascular health has been particularly influential in addressing racial health disparities. She has developed and tested multicomponent implementation strategies that engage community health workers and faith-based organizations to improve blood pressure control in low-income communities. Dr. Mills is actively engaged with the Chronic Renal Insufficiency Cohort (CRIC) study and has led multiple analyses examining cardiovascular outcomes in patients with chronic kidney disease. She collaborates with interdisciplinary teams including nephrologists, cardiologists, biostatisticians, and community health workers to address complex questions in cardiovascular and kidney disease epidemiology. Her current research focuses on identifying which populations benefit most from intensive blood pressure interventions and how social determinants of health influence treatment effectiveness.
Amanda W. Lundمشاهده پروفایل
دانشیار
- Lymphatic Vessels
- Cutaneous Immunity
- Melanoma
- +۶ مورد دیگر
Amanda W. Lund is an Associate Professor at the NYU Grossman School of Medicine , holding dual appointments in the Ronald O. Perelman Department of Dermatology and Department of Pathology. She also serves as Director of the PhD Curriculum at the Vilcek Institute. Education : PhD from Rensselaer Polytechnic Institute Postdoctoral Training : Laboratory of Lymphatic and Cancer Bioengineering at Ecole Polytechnique Federale de Lausanne Dr. Lund's research focuses on the critical role of lymphatic vasculature in cutaneous and tumor immunity. Her work explores how lymphatic vessels regulate adaptive immune responses, influence tumor immune escape, and impact metastasis dynamics. Key methodologies include 3D imaging , single-cell transcriptomics , and cell-specific proteomics to study these interactions in vivo. Her recent publications highlight lymphatic vessel contributions to T-cell trafficking in melanoma, immune surveillance mechanisms, and strategies to enhance cancer immunotherapy by targeting lymphatic-immune interfaces. Research trends show a strong emphasis on translational approaches bridging lymphatic biology with clinical oncology challenges. Contact: Amanda.Lund@nyulangone.org | Lab: Lund Lab
Sally Mortlockمشاهده پروفایل
عضو هیئت علمی
- Genetics
- Genomics
- Epigenetics
- +۴ مورد دیگر
Dr. Sally Mortlock is an Affiliate Senior Research Fellow at the Institute for Molecular Bioscience and a Senior Research Fellow at the School of Public Health, University of Queensland. With a PhD from the University of Sydney, her work focuses on the genetic and molecular mechanisms underlying endometriosis and related reproductive disorders. Positions: Institute for Molecular Bioscience, School of Public Health Key Research Areas: Genetics, Genomics, Epigenetics of endometriosis Her research integrates GWAS, epigenetic profiling, and multi-omics approaches to uncover genetic risk factors, gene-environment interactions, and molecular pathways in endometriosis. Recent work examines: Genetic regulation of endocannabinoid system in reproductive conditions Endometrial DNA methylation and disease risk scores Comorbidities with gastrointestinal/psychiatric disorders Scientific contributions include: Developing molecular staging models for endometrial biopsies Identifying pleiotropic genetic factors in endometriosis-depression links Characterizing endometriosis lesion subtypes through gene expression Dr. Mortlock actively supervises PhD students and has secured grants from: National Endometriosis Clinical and Scientific Trials Network (NECST) National Institutes of Health (NIH) National Health and Medical Research Council (NHMRC)
Michele K. Nishiguchi serves as Professor in the Department of Molecular and Cell Biology within the School of Natural Sciences at the University of California, Merced. Her research program centers on marine symbiosis evolution, specifically investigating the mutualistic relationship between sepiolid squids and Vibrio bacteria as a model system for understanding host-symbiont specificity, population dynamics, and environmental adaptation. Current affiliations include leadership of the Nishiguchi Lab and active participation in collaborative research initiatives addressing climate change impacts on symbiotic systems. Her primary research focuses on the molecular mechanisms governing squid-Vibrio symbiosis, examining how environmental factors and genetic determinants influence symbiont recognition, competitive exclusion, and cospeciation. Key areas include Vibrio population genetics, biofilm dynamics in host colonization, T6SS-mediated bacterial competition, and thermal stress responses in symbiotic partnerships. Recent work leverages CRISPR-based tools, transcriptomics, and genomic approaches to dissect symbiosis regulation across marine ecological niches. Analysis of her 15 most recent publications reveals a strong emphasis on environmental drivers of symbiosis (47%), molecular mechanisms of host-symbiont interaction (33%), and methodological innovations in symbiosis research (20%). Dominant themes include bacterial competition within host environments, climate change impacts on symbiotic stability, and evolutionary adaptations in symbiont transmission. Her work consistently bridges molecular microbiology with ecological and evolutionary frameworks. Scientific recognition includes: NSF CAREER Award for research on individual variation in dispersal through social landscapes Nishiguchi actively mentors undergraduate and graduate researchers, with 15 documented students in her 2022-2023 lab cohort including UROC and NSF REU participants. Current funding includes the SICBEDU collaborative grant (2024) focused on equity in integrative biology education and the BII: INSITE climate adaptation initiative (2022). Her grant portfolio demonstrates dual commitment to symbiosis research and broadening participation in STEM. The Nishiguchi Lab operates as a multidisciplinary research hub investigating marine symbiosis through molecular, ecological, and evolutionary approaches. Current projects examine biofilm-predator interactions, Vibrio genomics across host species, and climate stress impacts on symbiotic fitness, utilizing the squid-Vibrio model system's experimental tractability for both basic and applied research in microbiome science.
- Genetics
- Epigenetics
- Germline Development
- +۷ مورد دیگر
Valerie Reinke is the Harvey and Kate Cushing Professor of Genetics and Chair of the Department of Genetics at Yale School of Medicine. A leading expert in Caenorhabditis elegans germline gene regulation, her work explores epigenetic mechanisms, piRNA biogenesis, and tissue-specific transcriptional networks using cutting-edge genomic and molecular approaches. B.S. in Genetics (University of Illinois, 1990) Ph.D. in Biomedical Sciences (University of Texas Health Sciences Center, 1996) Postdoctoral work on C. elegans genomics at Stanford University (1996-2000) Her research addresses fundamental questions about genome organization, chromatin dynamics, and trans-generational epigenetic inheritance. Key projects include: Mechanisms of temporally regulated piRNA clusters Chromatin-based regulation of germline-to-embryo transitions Evolutionary conservation of histone modification patterns Her lab's work has broad implications for human health, spanning cancer biology, neurodegenerative diseases, and male infertility. Collaborations include researchers from the Center for RNA Science and Medicine, Yale Stem Cell Center, and international institutions.
Serkan Kırمشاهده پروفایل
استادیار
Serkan Kır is an Assistant Professor in the Department of Molecular Biology and Genetics at Koç University, Turkey. His research focuses on the molecular mechanisms underlying cancer cachexia, muscle wasting, and metabolic dysregulation in tumor-host interactions. Education: PhD, UT Southwestern Medical Center (2011) Bachelor’s, Bilkent University (2006) His work investigates: Role of interleukin-6 family cytokines in muscle degradation EDA2R-NIK signaling in skeletal muscle pathophysiology Tumor-derived ligands regulating MEK activation Adipose tissue thermogenesis and fibroblast growth factors PTHrP and EGFR interactions in cachexia models Recent publications highlight advancements in targeting metabolic reprogramming and signaling pathways for cachexia therapies. Key article trends include cancer metabolism, muscle physiology, and cytokine-driven wasting mechanisms.
Professor Alexander van Oudenaarden is a distinguished academic at Utrecht University, where he serves as Professor in the Faculty of Science, specifically within the Developmental Biology department. His research program focuses on cutting-edge approaches in developmental biology, stem cell research, and organoid technology. Dr. van Oudenaarden's research interests span multiple areas of developmental and cellular biology. He is particularly known for his work on stem cell systems, organoid development, and spatial transcriptomics. His laboratory has made significant contributions to understanding embryonic development through stem cell models, cancer therapy applications using organoids, and sex-specific gene expression patterns. His approach often combines quantitative methods with biological systems to uncover fundamental principles of development and disease. His publication record demonstrates significant impact, with multiple high-profile papers in journals such as Nature, Cell, and Nature Genetics. His 2018 paper on blastocyst-like structures generated from stem cells has been particularly influential, garnering over 386 citations and receiving attention from 35 news outlets. The research has implications for understanding early embryonic development and potential regenerative medicine applications. LifeTime and improving European healthcare through cell-based interceptive medicine (2020) - Highly cited review with over 121 citations Blastocyst-like structures generated solely from stem cells (2018) - Landmark paper with 386 citations Oral mucosal organoids as a potential platform for personalized cancer therapy (2019) - Significant contribution to cancer research with 294 citations Professor van Oudenaarden leads a research group focused on developmental dynamics and biocomplexity. His team employs advanced techniques including single-cell analysis, spatial transcriptomics, and organoid culture systems to address fundamental questions in developmental biology and their applications to human health. The group has established collaborations across Europe and maintains strong ties with clinical researchers to translate basic findings into potential therapeutic approaches.
Marina Sirota is a Professor of Pediatrics at the University of California, San Francisco (UCSF) School of Medicine and the Acting Director at the Bakar Computational Health Sciences Institute. She leads the UCSF March of Dimes Prematurity Research Center and co-directs ENACT, a center focused on precision medicine for endometriosis. Dr. Sirota is also the founding director of the AI4ALL program at UCSF, which introduces high school girls to AI applications in biomedicine. Education: B.S. in Biomedical Computation, Stanford University (2006) M.S. in Biomedical Informatics, Stanford University (2006) Ph.D. in Biomedical Informatics, Stanford University (2010) Dr. Sirota's research focuses on developing computational integrative methods for disease diagnostics and therapeutics, with special emphasis on women's health. Her work spans biomedical informatics, integrative genomics, clinical data analysis, immune informatics, and data science. She applies advanced computational approaches to understand complex biological systems and translate findings into clinical applications, particularly in the areas of preterm birth, endometriosis, and Alzheimer's disease. Her recent publications demonstrate a strong focus on precision medicine applications across multiple disease areas including women's health, neurodegenerative disorders, and immunology. Dr. Sirota frequently employs cutting-edge techniques like single-cell genomics, transcriptomics, and AI-driven approaches to analyze complex biological data. Her research shows particular strength in sex-specific analyses and the application of computational methods to understand disease heterogeneity. Scientific Awards: AMIA Young Investigator Award (2017) Dr. Sirota has secured significant research funding from multiple sources including NIA, NLM, NIAMS, Pfizer, March of Dimes, and the Burroughs Wellcome Fund. Her current projects include studies on endometriosis, preterm birth, Alzheimer's disease, and immune-related disorders. She mentors numerous students and researchers in computational health sciences and has co-authored over 170 scientific publications. Her laboratory actively collaborates with clinical and basic science researchers across UCSF and other institutions. Dr. Sirota directs the Bakar Computational Health Sciences Institute at UCSF and leads multiple research centers focused on women's health, including the March of Dimes Prematurity Research Center and ENACT (Endometriosis: Novel Clinical Approaches Through Technology). Her team combines expertise in bioinformatics, machine learning, clinical medicine, and basic science to advance precision medicine approaches for complex diseases.
Tomokazu S Sumidaمشاهده پروفایل
استادیار
Dr. Tomokazu S Sumida is an Assistant Professor of Neurology at Yale University School of Medicine, where he leads the Sumida Lab. His research focuses on understanding the molecular mechanisms that drive T cell dysfunction, particularly regulatory T cells (Tregs), in human diseases. His work utilizes cutting-edge technologies including single-cell multi-omics, ATAC-seq, and CRISPR gene editing/regulation to investigate immune tolerance and dysfunction in autoimmune diseases. Dr. Sumida received his MD from Chiba University School of Medicine in Japan in 2004, completed residency and cardiology fellowship in Japan, and practiced as a cardiologist before obtaining his PhD in 2012 studying the interface between the immune system and cardiovascular disease. He joined Yale in 2015 as a postdoctoral fellow in Dr. David Hafler's lab and was appointed Assistant Professor in 2020. His research interests span Autoimmune Diseases, Cardiovascular System, Immune System, Nervous System, Genetics, Genomics, Epigenetics, and Immunology. The Sumida Lab specifically investigates the factors and mechanisms controlling immune tolerance with emphasis on regulatory T cells, studying how dysregulation of Tregs contributes to immunopathogenesis in conditions like multiple sclerosis. Their work explores the dynamic balance of pro- and anti-inflammatory signals necessary to decipher complex immune responses to genetic and environmental factors. Analysis of Dr. Sumida's recent publications reveals a strong focus on regulatory T cell biology, particularly in multiple sclerosis and other autoimmune conditions. His work frequently employs single-cell technologies to understand immune dysregulation, with significant contributions to understanding how genetic variants affect immune cell function. His research bridges immunology, genetics, and neurology, with particular emphasis on how immune dysfunction contributes to neurological diseases. Harry Weaver Scholar Awards (2023) Race to Erase MS Young Investigator Award (2020) MSD Life Science Foundation Research Fellowship (2016) LEGEND Study Abroad Grant (2015) Uehara Memorial Foundation Research Fellowship (2015) Dr. Sumida has established significant collaborations across Yale, particularly with Dr. David Hafler's lab, where he was a postdoctoral fellow. His research program has received support from multiple sources, including the National Multiple Sclerosis Society. As Principal Investigator of the Sumida Lab, he leads investigations into the molecular mechanisms of immune tolerance and dysfunction, with the goal of developing therapeutic approaches for autoimmune diseases. His work bridges basic immunology with clinical applications, particularly in neuroimmunological disorders. The Sumida Lab focuses on basic human immunobiology and gene regulatory programs causing autoinflammatory disease, especially multiple sclerosis. They study the interaction of genetics and epigenetics to understand pathogenic gene regulatory circuits in human T cells, with particular focus on co-inhibitory receptors and the functional roles of Foxp3+ regulatory T cells in maintaining immune homeostasis.
Michael J. Gandal is the William & Noreen Heznecker Associate Professor in Psychiatry at the University of Pennsylvania. He holds secondary affiliations with the Lifespan Brain Institute (LIBI), Penn Epigenetics Institute, Center for Mitochondrial and Epigenomic Medicine (CHOP), and Institute for Biomedical Informatics. His research combines computational biology and functional genomics to investigate neurodevelopmental and psychiatric disorders , including Autism Spectrum Disorder (ASD) , ADHD , bipolar disorder , and schizophrenia . Education: BS in Biomedical Computation, Stanford University (2006) PhD in Bioengineering, University of Pennsylvania (2011) MD in Clinical Neuroscience, University of Pennsylvania (2013) Research Themes: His work focuses on genetic mechanisms , gene network biology , and single-cell genomics in the human brain. Key methodologies include GWAS , transcriptomics , and methylomics . Scientific Contributions: He has led major publications in Science , Nature Genetics , and Cell , particularly in single-cell analysis across 388 human brains and cross-ancestry gene regulation atlases. His team has uncovered cell-type shifts in neuropsychiatric conditions and novel isoform-level associations with disorders.
Domagoj Ševerdijaمشاهده پروفایل
استادیار
Domagoj Ševerdija is an Assistant Professor at the School of Applied Mathematics and Informatics at Josip Juraj Strossmayer University of Osijek, where he leads the Computer Science and Machine Learning Research Group. He holds a PhD in Electrical Engineering (2013) and a dual BS in Mathematics and Computer Science (2007) from the University of Osijek. His research spans computational linguistics, natural language processing, machine learning, and combinatorial optimization, with applications in bioinformatics, robotics, and energy systems. Recent work focuses on neural language models, domain adaptation techniques, and efficient algorithm design. Publications show strong interdisciplinary trends: computational linguistics (Croatian morphology, sentence embeddings), bioinformatics (cell typing, RNA splicing), and algorithm optimization (terrain guarding, matrix operations). Recent papers emphasize knowledge distillation, domain adaptation, and compressed representations. Awards: Best paper award in AIS - Artificial Intelligence Systems track (MIPRO 2023) Research funding includes: Computer-Assisted Corpus Linguistics (UNIOS, 2019-2020) Croatian Identity Network Framework (Adris Foundation, 2020-2021) Croatian Language in Global Cloud (Adris Foundation, 2019-2020) Leads the Computer Science and Machine Learning Research Group, coordinating projects in NLP, computational geometry, and AI applications.
Gayle Overendمشاهده پروفایل
پژوهشگر
- Molecular Biology
- Genetics
- Physiology
- +۳ مورد دیگر
Dr Gayle Overend is a Researcher at the University of Glasgow , affiliated with the Institute of Molecular, Cellular and Systems Biology and the Department of Molecular Biosciences . Her work spans molecular biology, genetics, and physiology, with a focus on Drosophila melanogaster and human disease models. Her research interests include: Epigenetic mechanisms in myotonic dystrophy (DM1) Ion transport and renal physiology in insects Gene expression regulation in disease progression Stress and immune response cross-talk in Drosophila Recent publications highlight her expertise in comparative physiology, Drosophila genetics, and epigenetic regulation of neuromuscular disorders. She has contributed to databases like FlyAtlas 2 and explored molecular pathways in malaria vectors. Advising : No student information available. Labs/teams : Collaborates with Julian A.T. Dow and Darren G. Monckton's research groups.
Irina Voineaguمشاهده پروفایل
دانشیار
Professor Irina Voineagu is an Associate Professor at the University of New South Wales (UNSW) within the School of Biotechnology and Biomolecular Sciences. Her research focuses on molecular genetic mechanisms underlying neurodevelopmental disorders using functional genomic approaches in human brain tissue and neuronal models. She leads the Functional Neurogenomics Group and maintains an active research program with consistent high-impact publications. Her research interests center on molecular genetics of brain disorders , particularly autism spectrum disorders and intellectual disability. Through transcriptome analysis, she investigates convergent molecular pathways, circular RNA biology, and genomic instability mechanisms. Her work bridges basic molecular biology with clinical neuroscience, identifying shared pathological mechanisms across neurodevelopmental conditions using post-mortem brain tissue and iPSC-derived neuronal models. Analysis of her 15 most recent publications reveals strong emphasis on transcriptomic regulation in neurodevelopment , with particular focus on circular RNAs, single-cell resolution mapping, and cross-species evolutionary comparisons. Her work consistently identifies convergent molecular pathways across diverse neurodevelopmental conditions, demonstrating how different genetic lesions lead to common pathological endpoints. Australian Academy of Science Gani Medal (2018) UNSW Scientia Fellowship (2017) Lorne Genome Women in Science Award (2017) Biological Psychiatry Australia Aubrey Lewis Award (2015) ARC Development Early Career Award (2013) Simons Foundation top 10 autism papers (2015) NARSAD Young Investigator Award (2012) Autism Speaks top 10 papers (2011) Professor Voineagu's research program is supported by major competitive grants including ARC and NHMRC funding. Her laboratory employs cutting-edge genomic technologies to dissect gene regulatory networks in the developing brain. The Functional Neurogenomics Group maintains collaborations with international consortia studying autism genetics and brain development. The Voineagu Lab operates within UNSW's neuroscience research ecosystem, utilizing advanced genomic facilities and collaborating with clinical researchers to translate molecular findings into potential therapeutic targets for neurodevelopmental disorders. Her team combines computational biology with experimental validation to characterize disease mechanisms at unprecedented resolution.