Inflammatory myocardial dysfunction Professors and Supervisors | ApplySharif

جست‌وجوی استاد و پژوهشگر

استاد راهنما، استاد دانشگاه یا پژوهشگر مناسب برای مسیر پژوهشی‌تان را پیدا کنید و با او ارتباط بگیرید.

نمایش ۱-۱۵ از ۳۹۳ استاد راهنما

اطلاعات این پروفایل‌ها از صفحات عمومی دانشگاه‌ها جمع‌آوری شده و ممکن است به‌روز نباشد.

Xuan Liمشاهده پروفایل

پژوهشگر ارشد

University of CambridgeUnited Kingdom

Dr. Xuan Li is a British Heart Foundation (BHF) Senior Basic Science Research Fellow and Principal Investigator in Cardiovascular Health at the Victor Phillip Dahdaleh Heart & Lung Research Institute, University of Cambridge. Her research focuses on microtubule dysfunction in inflammatory cardiovascular diseases, particularly investigating how microtubule regulation impacts inflammasome activation and cardiomyocyte contractility in heart failure and atherosclerosis. Her academic journey includes medical training in China, a CRUK-funded PhD in biochemistry at the University of Birmingham, postdoctoral research in microtubule cell biology as an MRC Career Development Fellow at the MRC-LMB Cambridge, and specialized cardiovascular medicine training at the University of Cambridge. She established her independent research group at Cambridge, pioneering multidisciplinary approaches to study microtubule function in cardiovascular cells. Dr. Li's research program centers on the role of microtubule-regulating kinases (particularly MARK4) in cardiovascular pathologies. Her group employs advanced methodologies including cardiovascular disease models (atherosclerosis, myocardial infarction, cardiomyopathy), primary cell culture of cardiomyocytes and immune cells, single-cell physiology using IonOptix systems, and cutting-edge microscopy techniques. Key discoveries include the identification of MARK4 as a critical regulator of inflammasome activation in atherosclerosis and microtubule detyrosination in ischemic heart failure. Her publication record demonstrates consistent focus on microtubule-cytoskeleton interactions in cardiovascular inflammation, with recent high-impact papers in Nature, Nature Communications, and Circulation Research revealing novel therapeutic targets for heart disease treatment through microtubule modulation. Scientific recognition includes: British Heart Foundation Senior Basic Science Research Fellowship MRC Career Development Fellowship Cancer Research UK PhD Studentship Dr. Li's group currently pursues four major research thrusts: elucidating microtubule dysfunction mechanisms in cardiovascular disease development, dissecting inflammation-microtubule interplay in pathogenesis, establishing human cardiomyocyte culture systems for microtubule studies, and translating basic findings into diagnostic and therapeutic applications for cardiovascular diseases. Her work bridges fundamental cell biology with clinical cardiology to develop innovative treatments targeting the microtubule cytoskeleton.

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Barbara L. Hempsteadمشاهده پروفایل

استاد

Cornell UniversityUnited States

Dr. Barbara L. Hempstead is a Professor of Neuroscience and Medicine at Weill Cornell Medical College, where she has held positions since 2001 and 2002 respectively. Her research focuses on neurotrophin signaling mechanisms, particularly the roles of BDNF and its receptors in neuroinflammation, synaptic plasticity, and neurodegenerative diseases. She has made significant contributions to understanding proBDNF/proNGF signaling pathways in neuronal apoptosis and vascular biology. Education: M.D., Ph.D., Washington University School of Medicine (1982) B.A., Tufts University (1976) Dr. Hempstead's work bridges molecular neuroscience and cardiovascular biology, with a particular interest in receptor stoichiometry (p75NTR, TrkB), neurotrophin-induced synaptic remodeling, and therapeutic applications of neurotrophin modulators in Huntington's disease and post-seizure neuronal injury. Her lab investigates how genetic variants like BDNF Val66Met influence anxiety-related behaviors, social memory, and neurodegenerative disease progression through altered neurotrophin trafficking and signaling. Her recent publications highlight neuroinflammatory mechanisms (2023), immune-neurotrophin interactions (2022), and molecular pathways involving BDNF prodomain structure (2020) and SorCS2-mediated receptor trafficking (2017-2020). While no scientific awards are explicitly mentioned in the scraped text, her funded research (National Institute on Aging, NIMH) demonstrates sustained recognition of her work in neurotrophin biology. Dr. Hempstead's lab develops in vitro and in vivo models to study neurotrophin-receptor dynamics, including 3D culture systems for angiogenesis research and transgenic mouse models for Huntington's disease. Her interdisciplinary approach combines molecular neurobiology with vascular physiology to uncover novel therapeutic targets for neurological and cardiovascular conditions.

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Xiaochen Heمشاهده پروفایل

مدرس

University of Mississippi Medical CenterUnited States

Xiaochen He serves as an Instructor in the Department of Physiology & Biophysics at the University of Mississippi Medical Center's School of Medicine, where he focuses on cardiovascular research and teaching within this foundational medical science department. His research program centers on the intersection of cardiac pathophysiology and immunometabolism, with core interests including: Mechanisms of immune-mediated cardiac inflammation in heart failure Role of T cell subsets (Th17, γδ T, CD8+) in pressure overload models Molecular regulation by IL-12 family cytokines and metabolic enzymes (TIGAR, SIRT3) Endothelial dysfunction in cardiac hypertrophy and failure progression Therapeutic interventions targeting inflammatory pathways Analysis of Dr. He's recent publications (2022-2025) reveals a concentrated research trajectory investigating how specific immune pathways drive heart failure progression. His work consistently employs genetic mouse models to demonstrate that IL-12β inhibition, TIGAR deficiency, and selenium supplementation attenuate cardiac inflammation and dysfunction, while CD8+ T cell metabolic reprogramming exacerbates disease. Key discoveries include GPR174's role in Th17 differentiation and NK1.1 signaling's contribution to cardiopulmonary inflammation, establishing critical immune-metabolic axes in heart failure pathogenesis. No scientific awards were documented in the available profile information. Current departmental records indicate no graduate students are formally listed under Dr. He's mentorship, and no research grants are specified in the public profile. Details regarding laboratory infrastructure, research teams, or collaborative networks were not provided in the available institutional documentation.

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Craig Morrellمشاهده پروفایل

استاد

University of RochesterUnited States

Craig Morrell is a Professor at the University of Rochester School of Medicine and Dentistry , with joint appointments in the Departments of Medicine , Microbiology and Immunology , and Pathology and Laboratory Medicine . He serves as Associate Director of the Aab Cardiovascular Research Institute . Education: Sc.B. in Biology, Brown University (1995) D.V.M. , Tufts University School of Veterinary Medicine (2000) Ph.D. in Pathobiology, Johns Hopkins University (2005) His research examines the interface between platelets, vascular biology, and immune cells , focusing on platelet roles in thrombosis, inflammation, and immune regulation. Using advanced mouse models , his lab investigates platelet-derived mediators like β2M and PF4 in diseases such as malaria, stroke, and myocardial infarction. Article trends highlight platelets' dual roles in thrombosis and immune modulation , with recurring themes in vascular inflammation , ERK5 signaling , and age-related immune dysfunction . Scientific Awards: Kenneth M. Brinkous Young Investigators Prize (2008) Deans Gift research award (2006) Multiple travel scholarships and fellowships (2003-2005) Pathology Award-Excellence in Pathology (2000) Mentoring and Teaching : He has mentored 8 doctoral students , 7 undergraduates , and 6 postdoctoral fellows , with a focus on diversity. He serves on thesis committees and the URMC MSTP (MD/PhD) admissions committee . Labs and Collaborations : Leads the Morrell Lab at the Aab Cardiovascular Research Institute, collaborating with institutions like Johns Hopkins and Medical College of Wisconsin.

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Shengyu Muمشاهده پروفایل

دانشیار

University of Arkansas for Medical SciencesUnited States

Hypertension
Heart Failure Mechanisms
Immune-Mediated Pathways in Cardiovascular Disease
+۴ مورد دیگر

Shengyu Mu is an Associate Professor in the Department of Pharmacology and Toxicology at the University of Arkansas for Medical Sciences (UAMS) College of Medicine. His research focuses on the pathogenesis of hypertension and its progression to heart failure, with a particular emphasis on immune mechanisms in renal salt retention. Mu holds an M.D. from TianJin Medical University (2004) and a Ph.D. from the University of Tokyo (2011). His research portfolio includes NIH-funded projects investigating T cell homing to the kidney and immune memory in hypertension. Current grants span NIH-NHLBI (2R01 HL146713), American Heart Association (AHA23TPA1076467), and USDA-NIFA collaborations. Completed projects include studies on renal lymph-angiogenesis and macrophage metabolism in diabetes/tuberculosis comorbidity. Research areas integrate physiology, molecular genetics, and epigenetics to bridge basic science and clinical applications. The Mu Lab includes Assistant Professor Yunmeng Liu, Ph.D. candidates Christoph Mora and Kathrine Deck, and research team members recognized for awards like the AHA Predoctoral Fellowship and APS Research Recognition Awards. Key themes include CD8+ T cell activation, IFNγ signaling, and macrophage transition in hypertension pathogenesis.

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Gary K. Owensمشاهده پروفایل

استاد

University of VirginiaUnited States

Dr. Gary K. Owens is a Professor in the Department of Molecular Physiology and Biological Physics at the University of Virginia , with a secondary appointment in the Department of Medicine, Division of Cardiology. He serves as Director of the Robert M. Berne Cardiovascular Research Center and leads groundbreaking research on vascular smooth muscle cell (SMC) dynamics in atherosclerosis. His work challenges conventional paradigms about SMC roles in plaque stability and thromboembolic events. Primary Affiliation: University of Virginia Key Titles: Robert M. Beirne Professor of Cardiovascular Research, Professor, Director of CVRC Research Themes include: Cardiovascular Biology Atherosclerosis Pathogenesis Smooth Muscle Cell Plasticity Epigenetic Regulation Inflammatory Signaling Tumor Metastasis Mechanisms Publication Trends (2015–2024) reveal expertise in Nature Medicine and Circulation studies, focusing on SMC gene regulation (Klf4/Oct4), plaque stability, and cross-talk between vascular cells and tumors. His work demonstrates that SMCs account for >80% of advanced lesion cells and that Klf4 inhibition reduces metastasis by >70%. Lab Leadership includes mentoring PhD students Anita Salamon and Victoria Milosek , alongside senior scientists and postdocs. Collaborations span with Dr. Gwen Randolph (Washington University) and Dr. Rosey Kaplan (NIH) on IL1β signaling and metastasis.

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Andrew Braunمشاهده پروفایل

استاد

University of CalgaryCanada

Andrew Braun is a Professor at the University of Calgary, affiliated with the Libin, Hotchkiss Brain Institute, and Snyder Institute. His research focuses on vascular physiology, particularly the role of potassium channels in regulating blood flow and pressure, with applications in diabetes and neurodegenerative diseases. He utilizes advanced techniques like patch clamping and arterial pressure myography. His work explores KCa channels , nitric oxide signaling , and calcium dynamics in vascular cells, aiming to develop novel therapies for endothelial dysfunction. He investigates how these channels affect myogenic tone , vasodilation , and coronary circulation in diseased states such as Type 2 Diabetes and Alzheimer's. The 15 most recent publications highlight his focus on pharmacological targeting of KCa channels to treat vascular complications in diabetes, aging, and atherosclerosis. Key subfields include platelet aggregation , genetic mutations in IP3 receptors , glycation effects on vascular function , and sex-dependent cerebrovascular defects . Contact information: Email abraun@ucalgary.ca. Administrative Assistant: Theresea Connolly (tconnoll@ucalgary.ca, 403-220-3018).

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John Eberthمشاهده پروفایل

دانشیار

Drexel UniversityUnited States

Dr. John F. Eberth is an Associate Professor at Drexel University's School of Biomedical Engineering, Science and Health Systems. As a cardiovascular engineer with expertise in mechanical controls, continuum biomechanics, and hydrogel-based extracellular matrix mimetics, he leads the Applied Biomechanics and Mechanobiology Lab (ABML) to investigate vascular behavior under mechanical stimuli. PhD in Biomedical Engineering from Texas A&M University (2008) MS in Mechanical Engineering from Clemson University (2004) BS in Mechanical Engineering from Clarkson University (2001) His research focuses on vascular pathology and mechanobiology, including: Aortopathy and aneurysm mechanics Endothelial dysfunction and arterial stiffening Hydrogel-based vascular grafts Calcification chelation therapy Coronary artery disease Perfusion tissue culture Recent publications demonstrate expertise in vascular imaging techniques, mechanical modeling, and therapeutic interventions. Key themes include drug-coated balloon development, collagen fiber mechanics, and bioreactor systems for vascular conditioning.

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Anton Baysaمشاهده پروفایل

مدرس ارشد

University of OsloNorway

Anton Baysa is a Senior Lecturer at the University of Oslo's Department of Pathology within the Faculty of Medicine. His research focuses on cardiovascular pathophysiology, particularly mechanisms of inflammation, mitochondrial dysfunction, and cellular repair following myocardial infarction and ischemia-reperfusion injury. He has published extensively on topics including Toll-like receptor signaling, mitochondrial DNA-induced inflammation, and the role of adaptor proteins in cardiac healing. His work integrates molecular biology, immunology, and clinical cardiology to understand and mitigate cardiac damage. Collaborative projects include studies on extracellular vesicles in surgery-related complications and growth factor pathways in myocardial repair. Key Research Themes: Cardiac inflammation mechanisms, mitochondrial damage responses, post-infarction healing, oxidative stress mitigation Recent Projects: Investigating p66ShcA protein signaling, nucleolin inhibition effects, and BMP-mediated cardiac remodeling

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Daniela Carnevaleمشاهده پروفایل

استاد

Sapienza University of RomeItaly

Daniela Carnevale is a Full Professor at Sapienza University of Rome, where she serves in the Department of Medical-Surgical Sciences and Biotechnologies within the Faculty of Medicine. She directs the Laboratory of Neuro and Cardiovascular Immunology at the Department of Molecular Medicine, based at IRCCS Neuromed in Pozzilli. Her academic career spans over a decade with a permanent position as Assistant Professor since 2012, progressing to her current role as Full Professor. Education: PhD in Neuroscience (2010) - Faculty of Medicine and Surgery, Catholic University of the Sacred Heart in Rome Professional qualification in Biology (2006) - Sapienza University of Rome Degree in Biological Sciences (2005) - Sapienza University of Rome, final mark 110/110 cum laude Daniela Carnevale's research primarily focuses on the intersection of cardiovascular medicine, neuroscience, and immunology. She investigates neuroimmune mechanisms in hypertension, exploring how the nervous and immune systems interact to influence blood pressure regulation and organ damage. Her work encompasses biotechnologies and translational approaches to understand arterial hypertension, chronic heart failure, Alzheimer's related dementias, and adaptive immune responses. She has pioneered research on brain-spleen communication pathways in hypertension, demonstrating how neural signals prime immune responses that contribute to cardiovascular pathology. Analysis of her recent publications reveals a strong emphasis on neuroimmune cardiovascular interfaces, with particular focus on hypertension-induced cognitive impairment, cerebrovascular remodeling, and organ damage mechanisms. Her work increasingly integrates advanced imaging techniques, genetic analyses, and immune profiling to uncover novel therapeutic targets for cardiovascular and neurodegenerative conditions. Scientific Awards: Fellow of the American Heart Association (FAHA) (2016) Council on Hypertension Mid Career Award for Research Excellence (2019) International Patent in medical technology (2010) Multiple research awards from the Italian Society of Arterial Hypertension Research Award from the Foundation "Roberto Cornelli" (2009) Professor Carnevale has secured significant research funding as Principal Investigator, including grants from Sapienza University, Istituto Pasteur-Fondazione Cenci Bolognetti, and the Italian Ministry of Health. She has supervised numerous PhD students, post-doctoral fellows, and undergraduate students across various biomedical programs. Her laboratory collaborates internationally with leading researchers from Harvard University, Vanderbilt University, University of Glasgow, and University of Munich. At IRCCS Neuromed, Professor Carnevale directs the Laboratory of Neuro and Cardiovascular Immunology, where her team investigates the cellular and molecular mechanisms underlying neuroimmune interactions in cardiovascular diseases. Her research facility includes advanced preclinical imaging capabilities, including a 7Tesla MRI for small animal research, which she secured through competitive grant funding.

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Xiaowen Baiمشاهده پروفایل

استاد

Medical College of WisconsinUnited States

Xiaowen Bai, MD, PhD, is a Professor in the Department of Cell Biology, Neurobiology and Anatomy at the Medical College of Wisconsin. His research focuses on leveraging stem cells to model neurodegenerative diseases and cardiac disorders, with a particular emphasis on non-coding RNAs, mitochondria, and environmental stressors like anesthetics and alcohol. He holds a leadership role as Executive Director of Hematology at the World Federation of Chinese Medicine Societies (WFCMS). Education: MD, Shanxi Medical University (1993) PhD, Beijing University Stem Cell Center (2004) Postdoctoral Training, University of Texas MD Anderson Cancer Center (2007) Research Interests: Neurodegeneration mechanisms involving microRNAs, lncRNAs, and mitochondrial dysfunction Stem cell-based therapies for myocardial regeneration Diabetic cardiomyopathy pathogenesis and cardioprotection 3D organoid models for studying anesthetic and alcohol neurotoxicity Recent Work: Recent publications emphasize translational studies using human-induced pluripotent stem cell (iPSC)-derived models to investigate alcohol-induced brain injury, anesthetic neurotoxicity, and cardiac fibrosis. His lab also explores immunomodulatory pathways and redox signaling in inflammatory diseases. Labs/Teams: The Bai Lab develops novel in vitro models for disease modeling and drug testing, focusing on cerebral organoids and cardiac organoids to bridge basic research and clinical applications.

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Fadi Gabriel Akarمشاهده پروفایل

استاد

Yale UniversityUnited States

Dr. Fadi Gabriel Akar is a Professor of Medicine (Cardiovascular Medicine) and Biomedical Engineering at Yale University School of Medicine. He holds affiliations with the Cardiovascular Medicine Department, the Yale Cardiovascular Research Center, and the Molecular Medicine Program. His primary appointment is as a Professor in the Department of Medicine, specializing in cardiovascular research and translational medicine. Dr. Akar’s research focuses on unraveling mechanisms promoting arrhythmias in structural heart diseases and developing gene-based therapies to prevent malignant arrhythmias. His lab investigates mitochondrial dysfunction, metabolic signaling, and mechano-electrical feedback in arrhythmogenesis, with a translational pipeline from target identification to pre-clinical testing in large animal models. Key areas include arrhythmias in heart failure, myocardial infarction, obesity, diabetes, atrial fibrillation, and pulmonary hypertension. Notable achievements include his role as Chairman of the AHA Cardiac Electrophysiology Fellowships Committee (2022). His work bridges basic science and clinical applications, emphasizing mechanism-based therapies over traditional antiarrhythmic approaches. Collaborations include studies on AMPK deletion models, mitochondrial network remodeling, and resolvin D1’s role in post-MI atrial remodeling. Publications highlight contributions to understanding arrhythmia mechanisms in metabolic diseases, mitochondrial dysfunction, and translational therapies. His research is supported by NIH grants and industry partnerships, focusing on advancing diagnostics and treatments for electrical cardiac disorders.

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Peixin Yangمشاهده پروفایل

استاد

University of Maryland, BaltimoreUnited States

Dr. Peixin Yang is the Christopher R. Harman, MD Endowed Professor of Obstetrics, Gynecology, and Reproductive Sciences at the University of Maryland School of Medicine. He serves as Professor with tenure in the Department of Obstetrics, Gynecology and Reproductive Sciences and holds a secondary appointment in the Department of Biochemistry & Molecular Biology. Dr. Yang is the founding director of the Center for Birth Defect Research at the University of Maryland School of Medicine and leads multiple NIH-funded research projects totaling millions of dollars. Dr. Yang's educational background includes: B.S. in Animal Science from Zhejiang Agricultural University (1986-1990) M.S. in Animal Reproductive Sciences from Nanjing Agricultural University (1990-1993) Ph.D. in Biophysics from Tokyo University of Agriculture & Technology and Zhejiang University (1994-1999) Postdoctoral Research Associate at University of Nebraska Medical Center (1999-2002) BIRCWH scholar (NIH K12) at University of Maryland Baltimore (2008-2009) Dr. Yang has built an extensive research program focused on diabetic embryopathy, particularly examining how maternal diabetes induces neural tube defects (NTDs), congenital heart defects (CHDs), and kidney defects. His laboratory was the first to establish a mouse model of diabetic embryopathy and reveal the causal role of JNK1/2 in neural tube defects. He has made significant contributions to understanding the molecular mechanisms of cellular stress, endoplasmic reticulum stress, and autophagy in neural tube defect formation. Dr. Yang also investigates the effects of maternal obesity on placental function and has established the Maryland Maternal Health Research Center of Excellence. His recent work has expanded to include studies on SARS-CoV-2 infection in pregnancy and connections between insulin resistance signaling and Alzheimer's disease. Analysis of Dr. Yang's recent publications reveals a strong focus on the molecular mechanisms of diabetic embryopathy, with particular emphasis on epigenetic regulation, cellular stress signaling pathways, and placental function. His work consistently bridges basic science with clinical applications, developing potential therapeutic approaches for preventing birth defects. A significant portion of his recent research examines the intersection of maternal metabolic conditions (diabetes and obesity) with fetal development, while also expanding into novel areas like viral infections in pregnancy and connections to neurodegenerative diseases. Dr. Yang's notable scientific achievements include: The F. Clarke Fraser New Investigator Award from the Teratology Society (2013) BIRCWH scholar (NIH K12) (2008-2009) The Lalor foundation postdoctoral Fellowship (2002-2003) Dr. Yang currently directs a multi-million dollar NIH-funded research group with multiple active R01 grants. His current projects investigate the intersection of mTOR/p70S6K1 signaling and HIPPO-Yap tissue organizer in neurulation, heightened hypoxia and DNA methylation in heart defects of diabetic embryopathy, hyperglycemia-induced cardiac progenitor dysfunction, and epitranscriptomic alterations in diabetic embryopathy. He has developed a robust research program in maternal diabetes-induced heart defects, which was previously an understudied area. Dr. Yang is also leading efforts to establish the Maryland Maternal Health Research Center of Excellence, focusing on the adverse effects of obesity, placental accreta spectrum, and opioid use disorder. As the founding director of the Center for Birth Defect Research at the University of Maryland School of Medicine, Dr. Yang leads a multidisciplinary team of translational and clinical scientists. His laboratory has made original contributions to understanding the molecular mechanisms underlying maternal diabetes-induced structural birth defects. The team employs genetically modified mouse models, whole-embryo culture systems, and human placental studies to investigate the effects of metabolic conditions on fetal development. Dr. Yang's group has been instrumental in developing natural compounds as potential preventatives for diabetic embryopathy, including trehalose, epigallocatechin-3-gallate, and curcumin.

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Marie Dam Lauridsenمشاهده پروفایل

مدرس

Aalborg UniversityDenmark

Chronic Obstructive Pulmonary Disease (COPD)
Cardiogenic Shock
Cohort Analysis
+۵ مورد دیگر

Marie Dam Lauridsen is a Clinical Instructor at North Denmark Regional Hospital, affiliated with Aalborg University's Faculty of Medicine, Department of Clinical Medicine. Her research focuses on chronic obstructive pulmonary disease (COPD), cardiogenic shock, and patient outcomes in respiratory medicine. She leads the 2025 project 'SEXA-PRO' investigating gender differences in COPD prognosis. Her work integrates telemedicine innovations and cohort analysis to improve diagnostic accuracy and treatment efficacy. Education : Not explicitly stated in provided text. Key research interests include COPD severity stratification, biomarker identification, and healthcare system optimization through telemedicine. Recent studies examine diuretic use impacts, patient mortality patterns, and treatment intensity as prognostic tools. Her 2024 publications highlight telehealth solutions for COPD and epidemiological cohort studies on blood eosinophil levels. Collaborative projects emphasize clinical trial design, healthcare equity, and multidisciplinary care approaches. She has supervised research projects and contributed to national cohort analyses, with findings published in Respiratory Medicine and European Respiratory Journal . Awards : None explicitly listed in text. Advising and grants involvement includes supervision in the 2025 project and contributions to grant-funded COPD research initiatives. Active in lab teams focusing on respiratory disease mechanisms and clinical decision-making tools. Labs/teams: Engaged in Aalborg University's clinical medicine research groups, particularly those addressing COPD heterogeneity and telehealth integration.

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Travis W. Heinمشاهده پروفایل

استاد

Texas A&M UniversityUnited States

Travis W. Hein, PhD, is a Professor in the Department of Medical Physiology at Texas A&M University's School of Medicine. His research focuses on microvascular dysfunction in diabetes, particularly in retinal and coronary microvessels, and its implications for vision loss and heart failure. He also investigates spaceflight-associated neuro-ocular syndrome (SANS) mechanisms in astronauts. Hein holds significant teaching awards, including the 2023 R. Kelly Hester Distinguished Teaching Award and membership in the Academy of Distinguished Medical Educators. Education: BA in Biology from St. Olaf College (1992), PhD in Medical Physiology from Texas A&M Health Science Center (1997). Research interests emphasize molecular mechanisms of vasomotor regulation, diabetes-induced vascular damage, and space health challenges. His work identifies therapeutic targets for microvascular diseases and explores ocular blood flow dynamics in space environments. Key publications highlight breakthroughs in retinal degeneration therapies (e.g., stanniocalcin-1), diabetic microvascular complications, and cardiovascular dysfunction mechanisms. His articles span 20+ years, demonstrating sustained contributions to vascular biology and translational medicine. Scientific awards: 2023 R. Kelly Hester Distinguished Teaching Award 2021 Elected to Academy of Distinguished Medical Educators 2017 Texas A&M Distinguished Teaching Award Grants and mentoring: Extensive NIH-funded research programs and training of graduate students/postdocs in cardiovascular and ocular physiology. Collaborates on space health initiatives through NASA partnerships. Labs/Teams: Directs Texas A&M's Microvascular Physiology Lab, focusing on translational research in diabetes and space health. Active in multidisciplinary teams addressing ocular vascular dysfunction and cardiac microcirculation.

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