Joel D. Trinity, PhD, is an Associate Professor in the Department of Internal Medicine at the University of Utah and holds adjunct appointments in the Department of Nutrition and Integrative Physiology and the Department of Physical Therapy & Athletic Training. He is affiliated with the Geriatrics Division and the Utah Vascular Research Laboratory (UVRL), where he has been a member since 2009. Education: PhD – The University of Texas at Austin MA – The University of Texas at Austin BA – Occidental College BA – University of California, Santa Cruz Postdoctoral Fellowship – University of Utah Advanced Fellowship – George E. Whalen VA Medical Center Research Focus: Dr. Trinity’s laboratory employs integrative in-vivo and in-vitro methodologies to investigate how aging and chronic diseases—particularly hypertension, heart failure, COPD, and peripheral artery disease—impair vascular function and blood-flow regulation. His work emphasizes the triad of neural control, oxidative stress, and endothelial signaling (especially nitric oxide) in determining vascular health across the lifespan. Publications Snapshot: From 2021-2025, Dr. Trinity has published extensively on human cardiovascular responses to exercise, vascular aging, and the vascular consequences of COVID-19. A recurring theme is the translational evaluation of passive leg movement as a non-invasive probe of endothelial function, with studies spanning young adults to older patients with multiple comorbidities. Funding & Collaborations: While specific grant numbers are not provided, the breadth of multi-center collaborations (e.g., RECOVER Consortium for long COVID) and continuous publication output indicate active federal and institutional funding. His laboratory hosts interdisciplinary trainees bridging geriatrics, exercise physiology, and vascular biology. Laboratory & Teams: Dr. Trinity conducts research within the Utah Vascular Research Laboratory (UVRL), leveraging state-of-the-art Doppler ultrasound, plethysmography, and metabolic assays to dissect vascular control mechanisms in human participants and murine models.







