معرفی
William Caudle is an Associate Research Professor at Emory University's Rollins School of Public Health, holding a primary appointment in the Gangarosa Department of Environmental Health. He also contributes to the Neuroscience Program in GDBBS, the Center for Neurodegenerative Disease, and the Neuroscience and Behavioral Biology Program. His research integrates toxicology and neuroscience to investigate environmental chemical impacts on the nervous system across life stages. Caudle holds a BS from Colorado State University, a PhD from Emory University, and completed postdoctoral training at the University of Washington.
Key awards include the 2018 Rollins Professor of the Year and 2017 Excellence in Teaching Award. His work has been highlighted in NIEHS Paper of the Month/Year recognitions for studies linking environmental exposures to neurological disorders. Caudle teaches courses like Human Toxicology and Neurotoxicology, emphasizing molecular mechanisms linking environmental chemicals to neurodegenerative and neuropsychiatric conditions.
Research focuses on pesticide-induced neurotoxicity, developmental exposures, and neurodegenerative pathways. He investigates how environmental chemicals disrupt dopamine systems, contributing to ADHD, Parkinson’s, and other conditions. Collaborations span public health, toxicology, and neuroscience to address translational questions in environmental neurology.
Recent work examines trans fat exposure in street foods, prenatal pesticide effects on fetal brain transcriptomes, and air pollution’s role in dementia and metabolic disorders. His lab employs rodent models, human population studies (NHANES), and systems toxicology approaches to bridge cellular mechanisms and population health outcomes.
Affiliations include the Caudle Lab (studying neurotoxicological impacts of environmental chemicals) and interdisciplinary programs at Emory. His research emphasizes systems-level approaches to understand chemical interactions with neural pathways, aiming to reduce environmental contributors to neurological disease.
