معرفی
Dr. Lana Ly serves as a Lecturer in the School of Biotechnology and Biomolecular Sciences (BABS) at UNSW Sydney, where she holds a Bachelor of Advanced Science (Honours) in Molecular and Cell Biology and a PhD in Biochemistry and Molecular Genetics, both completed at UNSW Sydney.
Her educational credentials include:
- Bachelor of Advanced Science (Honours) in Molecular and Cell Biology, UNSW Sydney
- PhD in Biochemistry and Molecular Genetics, UNSW Sydney (completed 2021)
Lana's research centers on transcription factor mechanisms in hematopoietic disorders, specifically investigating ZBTB7A/LRF and ZNF410 to reactivate fetal hemoglobin as therapy for sickle cell anemia and beta-thalassemia. Her methodological expertise spans protein-DNA binding analysis, structural biology, and epigenetic regulation, with emphasis on therapeutic applications for hemoglobinopathies through NuRD complex modulation and globin gene reactivation.
Publication trends reveal consistent focus on transcriptional regulation of globin genes, structural characterization of zinc finger proteins, and chromatin remodeling mechanisms, demonstrating translational potential for blood disorder treatments through promoter engineering and fetal hemoglobin reactivation strategies.
Her scientific recognition includes:
- 2019: UNSW Arc Postgraduate Council Outstanding Research Student Award
- 2018: Second-place prize winner in the UNSW Science Postgraduate Research competition
Lana has delivered undergraduate instruction since 2017, advancing to a full-time education-focused Lecturer in 2022. She currently co-convenes and lectures BABS3121 Molecular Biology of Nucleic Acids, BIOC2101 Principles of Biochemistry (Advanced), and BIOC2201 Principles of Molecular Biology (Advanced), with teaching excellence acknowledged through multiple BABS Titans of Teaching Awards from 2021-2023 for course leadership and student engagement.
Her doctoral research occurred in the Crossley Lab under A/Prof. Kate Quinlan and Prof. Merlin Crossley, specializing in DNA binding mechanisms of transcription factors relevant to red blood cell disorders.
