J. Nathan Davis

J. Nathan Davis is a Professor of Medical Sciences at the Frank H Netter School of Medicine, Quinnipiac University, where he has served as faculty since 2013 and was one of the founding faculty members of the medical school. His primary teaching responsibilities focus on the basic science curriculum for the first two years of medical education, with specialized expertise in Medical Biochemistry and Molecular Biology.

His educational background includes:

• BS from the University of Arkansas at Little Rock
• PhD from the University of Texas - Austin

Dr. Davis' research centers on molecular mechanisms of cancer, particularly alterations in cellular signaling pathways and transcriptional regulation. His work extensively investigates the RUNX family of transcription factors in carcinomas, sphingolipid metabolism in cancer cells, and molecular pathways in prostate, ovarian, pancreatic, and brain cancers. Key methodologies include biochemical analysis of transcription factors, kinase signaling, and tumor suppressor mechanisms.

Analysis of his publication record reveals a consistent trajectory from fundamental biochemical studies of lipid kinases and transcription factors (1990s-2000s) toward applied cancer biology (2010s), with recent expansion into medical education as evidenced by his 2023 pediatrics clerkship study. The predominant research themes span oncology, molecular signaling, and transcriptional regulation across diverse cancer models.

No specific scientific awards are documented in the available information.

While student advising details are not provided, Dr. Davis has maintained a significant research collaboration with Dr. Shari Meyers focusing on RUNX transcription factors in carcinomas. Grant funding specifics are not mentioned in the source material.

His laboratory research has centered on collaborative investigations of transcription factor networks in cancer progression, particularly examining how RUNX proteins interact with signaling pathways to drive malignant phenotypes in epithelial cancers.