Henrik Hasseldam

Henrik Hasseldam is an Associate Professor at the Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen. His research is conducted at the Biotech Research & Innovation Centre (BRIC), specifically within the Brakebusch Group, focusing on neuroimmunology and stroke-related mechanisms.

Education:

• Ph.D. in Health Sciences, University of Copenhagen (2012)
• M.Sc. in Human Biology, University of Copenhagen (2003)

Hasseldam's research interests center on Multiple Sclerosis with specific focus on neurodegeneration versus inflammation, cerebrospinal fluid as prognostic markers, and the relationship between Multiple Sclerosis and the microbiota. His current research investigates mitochondrial dysfunction in relation to autoimmune diseases, particularly using experimental autoimmune encephalomyelitis models. His work demonstrates a strong interdisciplinary approach bridging immunology, neurology, and molecular biology.

Analysis of his recent publications reveals a consistent research trajectory focused on neuroinflammatory processes in stroke and multiple sclerosis. His work spans biomarker discovery, immune cell dynamics, and therapeutic interventions, with particular emphasis on translational applications from animal models to human patients. The research shows increasing sophistication in methodologies, incorporating advanced imaging and molecular techniques.

Teaching and Professional Activities:

• Member of 'Teaching Corps 2013' developing innovative teaching initiatives
• Manager of the OAK brain conference (Odense, Aalborg, Copenhagen)
• Course leader in Basic Pathology
• Member of the study board for Human Biology (2008-2010)
• Completed university pedagogy training for assistant professors

Hasseldam maintains an active laboratory presence within the Brakebusch Group at BRIC, collaborating extensively with clinical researchers at the University of Copenhagen. His research program integrates basic science with clinical applications, particularly in understanding immune responses following stroke and in multiple sclerosis pathogenesis. Current work appears to be expanding into the role of mitochondrial dysfunction in autoimmune conditions.