معرفی
Christopher Quinn is an Associate Professor in the Department of Biological Sciences at the University of Wisconsin-Milwaukee (UWM), within the College of Letters & Science. His research focuses on neural circuit formation, particularly axon guidance mechanisms and the molecular basis of neurological disorders like Autism and Down Syndrome using the model organism C. elegans. He holds a PhD from Yale University (2001) and a BA from Rutgers University (1996), with postdoctoral training at Rutgers University (2009).
His work investigates the intracellular machinery guiding axon growth cones, including proteins like MIG-10/lamellipodin and their roles in asymmetry establishment. Recent studies explore autism-linked genes (e.g., RBM27) and mitochondrial dysfunction in neurodevelopment. Quinn’s lab employs genetic, biochemical, and cell culture approaches to uncover mechanisms regulating axon targeting and regeneration.
Publications highlight contributions to understanding axon guidance via UNC-6/netrin and SLT-1/slit cues, microRNA regulation of synaptogenesis, and the role of Rac1 in growth cone asymmetry. His findings advance therapies for neural injury and neurodevelopmental disorders. Advising graduate students on projects involving C. elegans genetics and selective autophagy mechanisms.
