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Jessica E. TreismanView profile

Professor

• Developmental genetics
• Cell signaling
• Synapse formation
• +5 more

Jessica E. Treisman is a Professor in the Department of Cell Biology and Department of Ophthalmology at NYU Grossman School of Medicine. Her research focuses on developmental genetics and molecular neuroscience, particularly in the context of visual system development and synapse formation in Drosophila . Research Interests: Cell fate determination, tissue morphogenesis, neural circuit assembly, and corneal lens development Contact: Jessica.Treisman@nyulangone.org | 212-263-1031 Lab: Treisman Lab, Skirball Institute, New York, NY Her work explores how intrinsic transcription factors and extrinsic signaling pathways interact to regulate cell differentiation and tissue organization in the Drosophila visual system, with implications for understanding human corneal development and neural connectivity disorders. Recent publications highlight her contributions to understanding: Molecular mechanisms of corneal lens curvature formation Regulation of synaptic targeting specificity Role of Sidekick in epithelial junction dynamics Transcriptional synergy between Glass and EGFR signaling The Treisman Lab employs interdisciplinary approaches in Drosophila genetics to uncover fundamental principles of cell signaling and neural circuit development, with potential applications in human vision research and developmental disorders.

Yukiko GotohView profile

Professor

• Neural Development
• Neural Stem/Progenitor Cells
• Epigenetics
• +7 more

Yukiko Gotoh is a Professor at the Department of Pharmaceutical Sciences, Graduate School of Pharmaceutical Sciences, The University of Tokyo. She serves as the Deputy Director and Principal Investigator at the International Research Center for Neurointelligence (IRCN). Her research focuses on understanding the mechanisms that regulate neural stem/progenitor cell fate during embryonic brain development and in the adult brain. Dr. Gotoh's research interests include: Genetic and epigenetic regulation of neural stem/progenitor cell fate Neuronal maturation processes Genesis and maintenance of adult neural stem cells Relevance of neural stem/progenitor cell dysregulation in neurodevelopmental disorders such as autism spectrum disorders Investigation of mechanisms regulating neural stem-progenitor cell fate during neocortical development Genetic and epigenetic regulation of neuronal activation Analysis of Dr. Gotoh's recent publications reveals a strong focus on neural stem cell biology, epigenetic regulation, and neurodevelopmental disorders. Her work demonstrates how chromatin modifiers like Polycomb group proteins and HMGA proteins regulate neural stem cell fate decisions during brain development. A significant portion of her research explores the embryonic origins of adult neural stem cells and how dysregulation of these processes contributes to conditions like autism spectrum disorders and schizophrenia. Her laboratory also investigates the basic mechanisms of cellular responses to viral infection in the brain and their relevance to neurodevelopmental disorders. Dr. Gotoh has made significant contributions to understanding: The role of Polycomb group proteins in neural development How chromatin modifiers regulate neurogenic potential Cell cycle regulation in neural stem cells The PDK1-Akt pathway in neuronal migration Layer-specific heterogeneity of astrocytes Mechanisms underlying schizophrenia-related abnormalities Dr. Gotoh's laboratory conducts research on multiple fronts related to neural development and stem cell biology. Her team investigates: Mechanisms regulating neural stem-progenitor cell fate during neocortical development Genetic and epigenetic regulation of neuronal activation The embryonic origin of adult neural stem cells Dysregulation of neural stem-progenitor cell and neuronal fate in neurodevelopmental disorders Innate immune responses in the brain

Trond Vidar HansenView profile

Professor

• Organic Chemistry
• Biochemistry
• Pharmacology
• +3 more

Trond Vidar Hansen is a Professor at the Department of Pharmacy, University of Oslo , and leads the LIPCHEM research group . He collaborates with institutions including the University of Bergen and Vestlandets Innovasjonsselskap through the VITADEL project, which recently received NOK 5,000,000 in verification support from the Research Council of Norway. His research focuses on the synthesis and biological evaluation of specialized pro-resolving lipid mediators derived from omega-3 fatty acids, with applications in inflammation resolution, neuroinflammation, and drug development. University : University of Oslo Department : Department of Pharmacy Research Group : LIPCHEM Collaborations : University of Bergen, Vestlandets Innovasjonsselskap Research Interests : H Hansen's work centers on the organic synthesis of bioactive lipid derivatives, particularly pro-resolving mediators from omega-3 polyunsaturated fatty acids. His team investigates their roles in inflammatory disease models , neuroinflammation , and PPAR receptor activation , aiming to develop therapeutic agents for conditions like chronic pain, diabetes, and neurodegenerative disorders. The research integrates stereoselective chemistry , biochemical profiling , and pharmacological evaluation to validate these mediators' clinical potential. Recent Awards : 2025: NOK 2,000,000 verification support from Research Council of Norway 2025: Co-leader of NOK 5,000,000 VITADEL project Publications : His articles (2015–2024) reveal a focus on stereoselective synthesis of resolvins, protectins, and maresins, with applications in anti-inflammatory and neuroprotective therapies . Key subfields include omega-3 metabolite profiling , PPAR agonist design , and biosynthetic pathway elucidation , often utilizing human cell models and mouse disease models . Collaborative projects emphasize commercialization of academic research and translational medicine .

Craig R. ForestView profile

Professor

• Neuroengineering
• BioMEMS Technologies
• High-Throughput Genomics
• +4 more

Dr. Craig R. Forest is a Professor at the Georgia Institute of Technology's Woodruff School of Mechanical Engineering, specializing in bioMEMS, neuroengineering, and high-throughput instrumentation. He leads the Precision Biosystems Laboratory, focusing on developing robotic tools for neuroscience and genomics. His research bridges mechanical engineering with biological systems, creating innovations like the PatcherBot for automated electrophysiology. Forest earned his Ph.D. (2007) and M.S. (2003) from MIT and B.S. (2001) from Georgia Tech. He has been recognized with awards including the 2013 Georgia Tech Class of 1940 W. Roane Beard Outstanding Teacher Award and Engineer of the Year (2013). His work emphasizes interdisciplinary collaboration, particularly through initiatives like CREATE-X and the Invention Studio, fostering student entrepreneurship and maker culture. Key contributions include ultra-high-throughput genomics tools, microfluidic systems, and acoustic reporter genes for medical imaging. Forest’s lab explores emerging fields like intracellular robotics in neuroscience and molecular communication networks, with applications in drug discovery and personalized medicine. Scientific awards highlight his impact in education and engineering innovation. His grants and collaborations span academic and industrial partnerships, advancing both theoretical and applied research in bioengineering and nanotechnology.

Anna G OrrView profile

Assistant Professor

• Neuroscience
• Neurodegenerative Diseases
• Astrocyte Biology
• +5 more

Anna G Orr serves as Nan and Stephen Swid Assistant Professor of Frontotemporal Dementia Research and Assistant Professor of Neuroscience at Weill Cornell Medical College's Brain and Mind Research Institute since 2016, leading pioneering research on astrocyte biology in dementia pathogenesis. Her educational background includes: Ph.D. from Emory University (2008) B.S. from Allegheny College (2002) Dr. Orr's research program centers on astrocytic-neuronal interactions , mitochondrial signaling , and neuroimmune mechanisms through three interconnected pathways: neuroimmune , oxidative , and G protein-coupled signaling . Her lab investigates how these mechanisms influence neuroinflammation, protein aggregation, synaptic function, and behavioral outcomes in dementia, with parallel therapeutic discovery efforts targeting astrocytic pathways for novel dementia treatments. Analysis of her 15 most recent publications (2025-2010) reveals escalating focus on astrocyte-specific dementia mechanisms , particularly mitochondrial ROS signaling, sex-dimorphic memory effects, TDP-43 pathology interactions with antiviral pathways, and lipid dysregulation in neurodegeneration. Her work consistently bridges molecular discoveries with therapeutic applications, demonstrating increasing NIH funding support for translational approaches. Key scientific recognitions include: NIH K99/R00 Pathway to Independence Award (2017) Leon Levy Fellowship in Neuroscience (2021) Nan and Stephen Swid Endowed Professorship (2021) Outstanding Neuroscience Teaching Award (2021) Dr. Orr actively mentors eight trainees across career stages, including Ph.D. candidates Evelyn Hardin and Constance Zhou, while securing major NIH grants as Principal Investigator for projects like Uncovering Dementia-Related Lipid Alterations in Astrocytes (NIA 2024-2026) and Mitochondrial Complex III Free Radicals in Dementia Pathology (NIA 2020-2026), alongside collaborative awards from the Alzheimer's Association. Her Orr Lab maintains a dual focus on mechanistic astrocyte biology and therapeutic translation, with current projects examining astrocytic TDP-43 dysregulation, mitochondrial complex III signaling, and sex-specific memory mechanisms using advanced in vivo techniques and disease models.

Quentin S. FischerView profile

Academic

• Neuroscience
• Synaptic Plasticity
• Neural Rehabilitation
• +7 more

Quentin S. Fischer, Ph.D., is a Research Assistant Professor at the Fralin Biomedical Research Institute at Virginia Tech Carilion (VTC), where he conducts neuroscience research within the Friedlander Lab. His work focuses on synaptic plasticity mechanisms, particularly long-term potentiation (LTP) and depression (LTD), in the context of mild traumatic brain injury (mTBI) and neural rehabilitation through stimulation. Education: Ph.D. in Psychology (Neuroscience program), University of California, Riverside Master of Arts in Psychology (Neuroscience program), University of California, Riverside Bachelor of Arts in Psychology (Behavioral Neuroscience program), University of Colorado, Boulder Dr. Fischer's research explores how patterns of synaptic stimulation influence calcium signaling and plasticity in both normal and injured brains, aiming to optimize noninvasive neurostimulation therapies such as magnetic or optical stimulation. His prior work extensively examined visual cortex plasticity, ocular dominance, and molecular signaling pathways involving PKA and calcineurin. The publication trends reflect a deep engagement with synaptic and cortical plasticity, particularly in visual systems, spanning molecular, cellular, and systems-level neuroscience. His early work focused on developmental and experience-dependent plasticity in visual cortex, while recent research aims to translate these findings into therapeutic strategies for brain injury. Professional Experience: Instructor, Baylor College of Medicine (Neuroscience; Psychiatry & Behavioral Sciences) Postdoctoral Associate, Yale University Medical School, Dept. Ophthalmology & Visual Science Postdoctoral Research Associate, Ohio University, Neurobiology Program Graduate Student, University of California, Riverside Professional Research Associate II, University of Colorado Research Assistant, University of Colorado Dr. Fischer is actively involved in pre-clinical research and is part of the Friedlander Lab team, contributing to the development of evidence-based neural stimulation protocols for neurorehabilitation. No formal scientific awards or student mentorship details are listed in the provided text.

• Neurochemistry
• Neuroscience
• Pain Research
• +3 more

Andrew J. Todd is a Professor and Honorary Fellow in the School of Psychology & Neuroscience at the University of Glasgow. His research focuses on neurochemistry and synaptic connections in the mammalian spinal cord, particularly the organization of neuronal circuits underlying pain and itch perception. He employs techniques like immunocytochemistry, confocal microscopy, and electron microscopy. Collaborations include researchers from institutions such as UCL, Saga University, and the University of Pittsburgh. His work is funded by the Wellcome Trust and BBSRC. Roles: Professor, Honorary Fellow Affiliations: School of Psychology & Neuroscience, University of Glasgow Research Interests Dr. Todd investigates spinal dorsal horn circuits, including projection neurons, interneurons, and synaptic plasticity. Key topics include: Neurochemical characterization of spinal neurons Role of neuropeptides like substance P and gastrin-releasing peptide Mechanisms of neuropathic pain and spinal circuit adaptations Functional roles of specific neuron populations in laminae I-III Articles Overview Recent work includes studies on spinal projection neuron markers (e.g., Tacr1, Gpr83), synaptic circuits involving GRP-expressing neurons, and interneuron subtypes' roles in pain/itch. Notable findings include the absence of neuronal loss in neuropathic pain models and the identification of novel spinal circuits. Grants & Funding Funded by the Wellcome Trust and BBSRC . Collaborations span international institutions, emphasizing spinal neurobiology and sensory processing.

Miler T. LeeView profile

Associate Professor

• Gene Regulation
• Developmental Biology
• Computational Genomics
• +9 more

Miler T. Lee is an Associate Professor at the University of Pittsburgh , focusing on gene regulation during early embryonic development through high-throughput experimental and computational genomics. He earned his Ph.D. in Genomics and Computational Biology in 2009 from the University of Pennsylvania under Dr. Junhyong Kim, followed by postdoctoral work with Dr. Antonio Giraldez at Yale University. Joining the university in 2016, his research spans maternal-to-zygotic transition (MZT), RNA stability, pluripotency networks, and evolutionary developmental biology, utilizing model organisms like zebrafish, Xenopus, and Hydractinia symbiolongicarpus. Key Research Themes: Maternally inherited RNA dynamics during embryogenesis Mechanisms of RNA degradation and transcriptome remodeling Evolution of pluripotency networks in hybrid species Role of zinc signaling in fertilization barriers Computational tools for RNA regulation and sensing Scientific Awards: Pan-American Society for Evolutionary Developmental Biology Junior Faculty Award (2024) Outstanding New Investigator – International Xenopus Board (2023) Basil O'Connor Scholar – March of Dimes (2017-2019) Recent publications highlight his work on enhancer classification, RNA degradation mechanisms, and cross-species MZT comparisons. His lab develops innovative methods like RESA for regulatory sequence analysis and studies evolutionary divergence in RNA localization patterns. While the articles span computational and experimental approaches, they consistently address RNA's role in cellular identity, developmental timing, and evolutionary adaptation. Applications include understanding pluripotency, designing RNA biosensors, and elucidating fertilization barriers. Prospective Ph.D. students are encouraged to contact him for opportunities in gene regulation, development, evo-devo, and computational genomics.

Barbara L. HempsteadView profile

Professor

• Neuroscience
• Neurotrophin Signaling
• Neurodegenerative Diseases
• +5 more

Dr. Barbara L. Hempstead is a Professor of Neuroscience and Medicine at Weill Cornell Medical College, where she has held positions since 2001 and 2002 respectively. Her research focuses on neurotrophin signaling mechanisms, particularly the roles of BDNF and its receptors in neuroinflammation, synaptic plasticity, and neurodegenerative diseases. She has made significant contributions to understanding proBDNF/proNGF signaling pathways in neuronal apoptosis and vascular biology. Education: M.D., Ph.D., Washington University School of Medicine (1982) B.A., Tufts University (1976) Dr. Hempstead's work bridges molecular neuroscience and cardiovascular biology, with a particular interest in receptor stoichiometry (p75NTR, TrkB), neurotrophin-induced synaptic remodeling, and therapeutic applications of neurotrophin modulators in Huntington's disease and post-seizure neuronal injury. Her lab investigates how genetic variants like BDNF Val66Met influence anxiety-related behaviors, social memory, and neurodegenerative disease progression through altered neurotrophin trafficking and signaling. Her recent publications highlight neuroinflammatory mechanisms (2023), immune-neurotrophin interactions (2022), and molecular pathways involving BDNF prodomain structure (2020) and SorCS2-mediated receptor trafficking (2017-2020). While no scientific awards are explicitly mentioned in the scraped text, her funded research (National Institute on Aging, NIMH) demonstrates sustained recognition of her work in neurotrophin biology. Dr. Hempstead's lab develops in vitro and in vivo models to study neurotrophin-receptor dynamics, including 3D culture systems for angiogenesis research and transgenic mouse models for Huntington's disease. Her interdisciplinary approach combines molecular neurobiology with vascular physiology to uncover novel therapeutic targets for neurological and cardiovascular conditions.

• Neuroscience
• Neurobiology
• Retina
• +4 more

Karl Farrow is a Senior Lecturer at the Faculty of Sciences , KU Leuven , affiliated with the Department of Biology and the Animal Physiology and Neurobiology unit. He is also a member of the VIB-KU Leuven Center for Neuro Electronics Research Flanders (NERF) and the KU Leuven Brain Institute . Dr. Farrow's research focuses on neurobiology and visual processing , particularly the role of retinal ganglion cells and neural circuits in the superior colliculus . His work explores direction selectivity, motion perception, and adaptive mechanisms in visual systems under varying ambient conditions. His recent projects include dissecting the neural basis of threat responses in the superior colliculus, studying evolutionary influences on innate behavior circuits, and developing tools like a single-objective light sheet microscope for 3D tissue analysis. Collaborative efforts involve synaptic transmission studies in amyloid-beta precursor protein interactions.

Kuo-Fen LeeView profile

Professor

• Molecular Neurobiology
• Nerve Regrowth
• Spinal Cord Injury
• +5 more

Kuo-Fen Lee, PhD is a Professor at the Salk Institute for Biological Studies, holding the prestigious Helen McLoraine Chair of Molecular Neurobiology. He leads the Clayton Foundation Laboratories for Peptide Biology, where his research focuses on nerve regeneration, spinal cord injury, and molecular mechanisms underlying neural development and neurodegenerative diseases. His work bridges basic neuroscience with potential therapeutic applications for conditions like ALS, paralysis, and Alzheimer's disease. Dr. Lee received his educational training from multiple prestigious institutions: a degree in Plant Pathology from National Taiwan University; an MS in Cancer Enzymology and Cell Differentiation from National Yang-Ming Medical College, Taiwan; a PhD in Endocrinology from Baylor College of Medicine, Houston; and completed his postdoctoral training at the Whitehead Institute for Biomedical Research. His primary research interests center on understanding why humans cannot regenerate damaged nerves while many other animals can. Dr. Lee has made significant discoveries regarding the p45 protein, which promotes nerve regrowth in mice but is absent in humans (who instead have p75, which inhibits nerve growth). His laboratory also studies neuregulin signaling, neuromuscular synapse formation, and the role of various proteins like nestin in neural development and maintenance. His work often employs mouse models to investigate spinal cord injury, pain pathways, and neurodegenerative conditions. Analysis of Dr. Lee's recent publications reveals a consistent focus on molecular neurobiology with particular emphasis on neural signaling pathways, synaptic maintenance, and nerve regeneration mechanisms. His research spans from basic molecular mechanisms to potential therapeutic applications, with increasing attention to pain pathways, Alzheimer's disease models, and the intersection of neuroscience with immunology and metabolism in recent years. As holder of the Helen McLoraine Chair of Molecular Neurobiology, Dr. Lee has received significant institutional recognition for his contributions to neuroscience. While specific awards aren't detailed in the provided text, his sustained funding and leadership position indicate substantial peer recognition in his field. Dr. Lee's research program involves extensive collaboration with other neuroscience laboratories, as evidenced by his numerous co-authored publications across various neuroscience subdisciplines. His work has been consistently funded, allowing for the maintenance of an active research laboratory focused on nerve regeneration and molecular neurobiology. The Clayton Foundation Laboratories for Peptide Biology serves as the primary research environment for Dr. Lee's team, where they investigate molecular mechanisms of nerve development, regeneration, and degeneration using advanced genetic, molecular, and cellular approaches. The laboratory maintains active research programs in multiple areas of neural signaling and development.

Karl Peter GieseView profile

Professor

• Neurobiology of Mental Health
• Alzheimer's Disease
• Synaptic Plasticity
• +6 more

Professor Karl Peter Giese holds the position of Professor of Neurobiology of Mental Health and Co-Head of the Basic & Clinical Neuroscience Department at King's College London's Institute of Psychiatry, Psychology & Neuroscience (IoPPN). His research focuses on memory mechanisms in health and disease, particularly Alzheimer's pathology, synaptic dysfunction, and aging effects. He leads projects funded by Alzheimer's Research UK and other institutions, investigating molecular and cellular bases of memory storage. His work bridges experimental models (e.g., mice) with translational insights for clinical applications. He has over 140 publications, including high-impact studies on CYFIP proteins in dementia and CaMKII in synaptic plasticity. Collaborations include researchers at King's College London and international partners. His lab explores mechanisms linking amyloid-beta, tau, and synaptic proteins to cognitive decline, with recent work applying computational methods to model aging brains. Education: PhD from ETH Zurich (1992), MSc Chemistry from Ruhr-University Bochum (1989). Current grants include Alzheimer's Research UK Network Centres and studies on MNK inhibition for Alzheimer's therapies. Projects span protein synthesis dysregulation, thalamic amyloid pathology, and intellectual disability genetics. His research has been featured in Nature Neuroscience , Brain , and Neuron . He advises on translational neuroscience initiatives and mentors early-career researchers.

Tim DenisonView profile

Professor

• Neural interface technologies
• Algorithm technologies for neurological disease treatment
• Physiologic closed-loop systems
• +7 more

Professor Tim Denison FREng holds a joint appointment in the Department of Engineering Science and Nuffield Department of Clinical Neurosciences at the University of Oxford, where he serves as the Royal Academy of Engineering Chair in Emerging Technologies and an MRC Investigator. His research focuses on the fundamentals of physiologic closed-loop systems and developing next-generation neural interface technologies for treating chronic neurological diseases. Professor Denison received his A.B. in Physics from The University of Chicago, followed by M.S. and Ph.D. degrees in Electrical Engineering from MIT. He later completed an MBA at The University of Chicago, where he was named a Wallman Scholar. His research spans neural engineering, closed-loop neuromodulation systems, and computational neuroscience, with particular emphasis on deep brain stimulation, neural oscillations, and adaptive neurostimulation techniques. His work integrates engineering principles with clinical neuroscience to develop innovative treatments for neurological disorders. Professor Denison's approach combines computational modeling with experimental validation to optimize brain stimulation parameters for individual patients. Professor Denison has received numerous prestigious awards, including membership in the Bakken Society (2012, Medtronic's highest technical honor), the Wallin leadership award (2014), election to the College of Fellows for the American Institute of Medical and Biological Engineering (2015), and recognition as a Fellow of the Royal Academy of Engineering (FREng). As a former Technical Fellow at Medtronic PLC and Vice President of Research & Core Technology for the Restorative Therapies Group, Professor Denison brings significant industry experience to his academic work. His research group focuses on developing advanced neurostimulation technologies that incorporate chronobiology principles and adaptive algorithms to improve treatment outcomes for neurological conditions.

• Auditory Neuroscience
• Neuromodulation
• Neuronal Excitability
• +4 more

Dr. Conny Kopp-Scheinpflug is an Associate Professor (PD) at the Faculty of Biology, Ludwig Maximilian University of Munich, where she leads a research group focused on auditory neuroscience. Her laboratory investigates the function and mechanisms of activity-dependent neuromodulation in the mammalian auditory system, with particular interest in how ambient sensory stimulation activates neuromodulators and how these influence neural processing of relevant information. Dr. Kopp-Scheinpflug's research spans auditory neuroscience, neuromodulation, neuronal excitability, and synaptic transmission. She employs electrophysiological (single cell in vivo and patch clamp in brain slices), anatomical, and optogenetic techniques to study how hyper- or hypo stimulation lead to acquisition or loss of function in the auditory system. Her work has significant implications for understanding and potentially treating functional disorders of neuronal excitability. Current research examines potassium channels, nitric oxide signaling, and neuromodulators like urocortin 3 in auditory processing. Analysis of Dr. Kopp-Scheinpflug's recent publications (2016-2022) reveals consistent focus on auditory processing mechanisms, particularly potassium channels (Kv3.1, Kv3.3, Kv1.1), nitric oxide signaling, and activity-dependent changes in myelination. Her research spans molecular mechanisms to systems-level auditory processing, with emphasis on sound localization, temporal processing, and recovery from hearing impairment. Key findings include how sound-evoked activity influences myelination, how nitric oxide regulates postsynaptic excitability, and how urocortin 3 aids hearing recovery. Dr. Kopp-Scheinpflug has secured funding from multiple research agencies. She maintains active collaborations with researchers at Lehigh University (Michael Burger Lab), University of Edinburgh (Matthias Hennig Lab), Ben-Gurion University of the Negev (Michal Hershfinkel Lab), and UCL (Dr. Jennifer Linden). Her laboratory currently includes Ezhilarasan Rajaram, Dr. Mihai Stancu, Oskar Kalle Juhani Markkula, Sara Pagella, and Katharine Krueger. Past lab members who have completed their training include Dr. James Sinclair, Dr. Matthew Fischl, Max Bayer, Alkmini Damkou, Alyahyay Mansour, Leander Mrowka, Joseph Kroeger, and Myriam Schmidt-Pauly.

Martha R.C. BhattacharyaView profile

Associate Professor

• Axon Degeneration
• Neurodegenerative Diseases
• Molecular Mechanisms
• +3 more

Martha R.C. Bhattacharya is an Associate Professor in the Department of Neuroscience at the University of Arizona, where she also serves as Faculty Director of the Vertically Integrated Projects (VIP) program. She holds a Ph.D. from UCSF, focusing on mechanotransduction under Dr. David Julius, and completed postdoctoral training at Washington University in St. Louis with Dr. Aaron DiAntonio, studying axon degeneration mechanisms. She joined the University of Arizona in 2017 and received tenure in 2023. Her research investigates molecular mechanisms of axon degeneration using Drosophila and mouse models, with a focus on neurodegenerative diseases such as ALS and Alzheimer’s. Key discoveries include identifying MORN4 and TMEM184B as mediators of axon degeneration. Her work also explores TMEM184B’s role in itch sensation and synaptic dysfunction. Dr. Bhattacharya has received prestigious awards, including the NSF CAREER Award and the Five Star Faculty Award for Teaching Excellence. She leads a dynamic lab mentoring numerous students and has secured NIH R01 and R21 grants. Her educational initiatives, such as the Brain Communication Networks CURE course, emphasize undergraduate research training. Her lab collaborates widely, including with Dr. Sean Sweeney on Drosophila models, and has contributed to translational studies on TMEM184B-associated diseases. Future work includes exploring human stem cell models and advancing therapies for neurodegenerative conditions.