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Zheng ZhouView profile

Professor

• Cell Biology
• Molecular Genetics
• Developmental Biology
• +3 more

Zheng Zhou is a Professor at the Verna and Marrs McLean Department of Biochemistry and Molecular Biology at Baylor College of Medicine. He is also a member of the Dan L Duncan Comprehensive Cancer Center. His research focuses on the molecular mechanisms of apoptotic and necrotic cell clearance in Caenorhabditis elegans , particularly the recognition, engulfment, and degradation of dying cells. Zhou holds a BS from Fudan University (Shanghai, China) and a PhD in Biochemistry from Baylor College of Medicine. He completed postdoctoral training at MIT and Baylor College of Medicine. His honors include the Norton Rose Fulbright Faculty Excellence Award (2018) and multiple fellowships, including from the Robert A. Welch Foundation and the Rita Allen Foundation. His research interests include understanding how dying cells expose 'eat-me' signals like phosphatidylserine and how phagocytic receptors (e.g., CED-1) mediate clearance. His lab uses genetic screens, live imaging, and molecular tools to study these processes in C. elegans . Recent work highlights roles of calcium signaling in necrotic cell recognition and autophagy-phagocytosis crosstalk in apoptotic cell degradation. Zhou has mentored numerous students and postdocs, including Omar Peña-Ramos (now a postdoc) and Jonathan Pickett. His lab is funded by grants such as an R35 MRA award and an RO1 supplement supporting research on lipid transfer proteins' roles in cell-cell interactions. Labs/Teams: The Zhou Lab at Baylor College of Medicine focuses on programmed cell death and clearance mechanisms. Collaborations include work on dynamin function and signaling pathways in phagocytosis.

Bahaa MustafaView profile

Assistant Professor

• Cancer Immunotherapy
• Peptide-Based Therapeutics
• Oncolytic Virotherapy
• +3 more

Bahaa Mustafa, Ph.D., is an Assistant Professor in the Department of Pharmaceutical Sciences at the University of Arkansas for Medical Sciences (UAMS) College of Pharmacy. He joined UAMS in May 2022 and became an Associate Member of the Winthrop P. Rockefeller Cancer Institute’s Cancer Therapeutics Program in April 2023. His research focuses on developing biotherapeutics, including peptides and nanobodies, using phage display techniques, targeted liposomal drug delivery for lung cancer, and combining peptide-based immune checkpoint inhibitors with oncolytic virotherapy for colorectal and pancreatic cancers. Dr. Mustafa holds a Ph.D. in Pharmaceutical Sciences from the University of Missouri-Kansas City (2022), a Master of Pharmaceutical Sciences from Petra University (2015), and a Bachelor of Pharmacy from Applied Science University (2010). He teaches Pharmaceutics courses at UAMS, including PSCI 71303 (Pharmaceutics I), PSCI 71404 (Pharmaceutics II), and a non-sterile compounding lab. His research interests span cancer immunotherapy, targeted drug delivery systems, and virotherapy. Recent work includes engineering oncolytic viruses to treat pancreatic cancer and discovering peptides that inhibit immune checkpoints like PD-L1 and CD47. He has contributed to studies on enhancing virotherapy efficacy through stroma targeting and combining therapies to improve tumor resectability. Dr. Mustafa has received several awards, including the 2024 Seeds of Science Award and Travel Grant from the Winthrop P. Rockefeller Cancer Institute, the 2023 Outstanding Dissertation Award, and the 2017 Fulbright Foreign Student Scholarship. His publications highlight innovations in peptide design, oncolytic virus engineering, and drug delivery mechanisms. Dr. Mustafa is affiliated with professional organizations such as the Controlled Release Society, American Association of Pharmaceutical Scientists (AAPS), and American Peptide Society. His lab focuses on translational research bridging peptide therapeutics, virotherapy, and cancer immunology.

Taejoon WonView profile

Assistant Professor

• T Cell Immunology
• Macrophage Immunology
• Myocarditis
• +3 more

Taejoon Won is an Assistant Professor in the Department of Pathobiology at the University of Illinois. His research focuses on immune mechanisms underlying myocarditis, autoimmunity, and the role of programmed death ligand 1 (PD-L1) in inflammatory diseases. He investigates how immune checkpoint inhibitors contribute to cardiovascular adverse events and explores protective roles of specific macrophage subsets in arthritis and myocardial injury. Key research themes include T cell immunology, macrophage biology, and the interplay between cytokines and tissue-resident memory T cells. His work integrates lipidomics, single-cell analysis, and mouse models to study health-disease transitions in innate lymphoid cells and cardiovascular pathologies. Recent articles highlight discoveries such as myocarditis driven by myosin-specific T cells, protective roles of GATA6+ macrophages in pericarditis, and mechanisms of immune checkpoint inhibitor toxicity. Collaborations span cardiology, rheumatology, and immunology, with studies published in Circulation Research , Proceedings of the National Academy of Sciences , and Arthritis and Rheumatology .

Hilary Ann CollerView profile

Professor

• Cellular Quiescence
• Cancer Biology
• Metabolism
• +4 more

Hilary Ann Coller is a Professor at the University of California, Los Angeles (UCLA), jointly appointed in the Department of Molecular, Cell and Developmental Biology and the Department of Biological Chemistry within the College of Letters and Science. She leads a research laboratory focusing on cellular quiescence, metabolism, and their implications in cancer and disease progression. Her research integrates molecular biology, epigenetics, and systems approaches to study: Transcriptional and epigenetic control of quiescence Metabolic adaptations in tumor microenvironments RNA biology and gene regulation networks Development of biomedical technologies (e.g., biosensors) Recent publications (2022–2025) highlight trends in: Cellular quiescence mechanisms and transcription factor dynamics Cancer epigenetics and metabolic reprogramming Innovative tools for metabolomics and drug monitoring Educational research on remote STEM training programs She directs multiple NIH-funded projects, including R01CA221296 (stromal autophagy in melanoma) and R01GM086465 (microRNA mechanisms). Her lab mentors graduate students and postdocs, and she co-leads UCLA’s Bruins-in-Genomics summer initiative for undergraduate computational biology training.

Umamaheswari NatarajanView profile

Researcher

• Cancer Biology
• Epigenetic Modifications in Cancer
• Drug Discovery
• +5 more

Umamaheswari Natarajan is an Associate Research Scientist at Nova Southeastern University's Rumbaugh-Goodwin Institute for Cancer Research within the Barry and Judy Silverman College of Pharmacy. She earned a Ph.D. in Pharmacy from the University of Madras, India, and holds prior degrees including a D.Pharm., M.Sc., and B.Sc. from Indian institutions. Ph.D., University of Madras, India D.Pharm., The Tamil Nadu Dr. M.G.R. Medical University, India M.Sc., Madurai Kamaraj University, India B.Sc., Madurai Kamaraj University, India Dr. Natarajan's research focuses on epigenetic modifications in cancer, particularly identifying biomarkers for tumor aggressiveness and drug resistance, and improving delivery of anti-angiogenesis drugs like F16 and JFD for brain cancers. Her work spans breast, prostate, ovarian, lung, osteosarcoma, glioblastoma, and neuroblastoma studies. She has published 14 peer-reviewed papers and co-authored numerous conference presentations. Her Google Scholar publications reveal a strong emphasis on HDAC inhibitors, MDM2/MDMX regulation, AURKB interactions, and anti-angiogenic therapies. Subfields include neuroblastoma, lung cancer, and ovarian cancer treatment resistance mechanisms. Epigenetic modifications in cancer Drug discovery Angiogenesis Cell cycle regulation Apoptosis Pharmacogenomics Dr. Natarajan actively mentors graduate and undergraduate students in research electives, guiding presentations at conferences like ABRCMC and AACR. Her students have received multiple awards for their work. She also contributes to the Honors College as a course instructor. Current projects involve enhancing brain delivery of anti-angiogenesis drugs and exploring epigenetic biomarkers across cancers. Collaborations include Appu Rathinavelu, Ph.D., in developing research electives.

Douglas B. CowanView profile

Researcher

• Cardiovascular Biology
• Mitochondrial Transplantation
• Ischemia-Reperfusion Injury
• +3 more

Douglas B. Cowan serves as a Staff Scientist in the Cardiology and Vascular Biology Program at Boston Children's Hospital and holds an Associate in Surgery position at Harvard Medical School. His career spans over three decades with foundational training in genetics and molecular biology. B.Sc. in Honors Genetics, University of Western Ontario (1987) M.Sc. and Ph.D., University of Toronto (1989, 1994) Postdoctoral Fellowship, MRC-sponsored at Mount Sinai Hospital and Toronto General Hospital Dr. Cowan's research focuses on mitochondrial transplantation as a novel therapy for ischemia-reperfusion injury, with pioneering work demonstrating that autologous mitochondria injected during reperfusion significantly enhance cardiac recovery. His expertise bridges cardiovascular molecular biology , bioengineering , and translational surgical innovation , particularly in developing rapid mitochondrial isolation techniques and coronary delivery systems. Current investigations explore mitochondrial integration mechanisms in human cardiomyocytes using super-resolution microscopy. His publication portfolio includes 124 works with 4,810 citations and an h-index of 39, dominated by high-impact studies in PLoS ONE , American Journal of Physiology , and Journal of Thoracic and Cardiovascular Surgery . Key trends show evolution from foundational work on endothelial junctions (1999) to mitochondrial therapy dominance since 2009, with recent expansion into stroke applications (2023) and donor heart preservation (2022). Dr. Cowan collaborates extensively with James D. McCully (135+ co-publications), Pedro J. del Nido, and Ingeborg Friehs within the Cardiac Surgery Research Laboratory at Boston Children's Hospital. Current projects include optimizing mitochondrial delivery for pediatric cardiac applications and investigating metabolic mechanisms in fatty acid metabolism post-transplantation.

• Neurobiology
• Molecular Neurobiology
• Cellular Neural circuits
• +5 more

Robert Carrillo is an Associate Professor of Molecular Genetics and Cell Biology and Neuroscience Institute at the University of Chicago, with a joint appointment in the Committee on Neurobiology. He also serves as the Associate Dean for Diversity, Equity, and Inclusion for Basic Science. His research focuses on understanding the molecular mechanisms underlying neural circuit formation and function in Drosophila melanogaster . Dr. Carrillo's educational background includes: BS in Cybernetics from University of California, Los Angeles (2001) PhD in Pharmacology from Yale School of Medicine (2009) Dr. Carrillo's research program investigates how neural circuits are wired during development, with a particular focus on cell surface proteins in the immunoglobulin superfamily (Dprs and DIPs). His lab examines three main aspects of neural circuit formation: Finding the right partner(s) : How neurons identify appropriate synaptic partners through "lock-and-key" mechanisms mediated by cell surface proteins Growing together : How synaptic connections expand to maintain proper activity after initial formation Compensation upon loss of a neighbor : How neurons detect and respond to perturbations in neighboring neurons through synaptic plasticity Analysis of Dr. Carrillo's recent publications reveals a strong focus on Drosophila neural circuit development, particularly examining the roles of Dpr and DIP proteins in motor neuron targeting, synaptic plasticity, and neural wiring. His work combines genetic, biochemical, and functional approaches to uncover fundamental programs of nervous system development, with implications for understanding neurological disorders like autism spectrum disorder. Dr. Carrillo has received numerous scientific awards and honors: NSF CAREER Award (2021) NIH R01 grant as Principal Investigator (2021-2026) NIH K01 grant (2017-2020) Ford Foundation Predoctoral Fellowship (2005-2008) NIH/MARC Fellowship (1998-2001) Dr. Carrillo actively mentors students through various programs including the Neuroscience Early Training Opportunity (NEETO), Diversity Access to Research in Neuroscience (DARN), and Undergraduate Neuroscience Diversity and Outreach (UNDO). His lab has secured significant funding through NIH R01 grants, an NSF CAREER award, and participation in the University of Chicago PREP program as a Co-Principal Investigator. The Carrillo lab has made important contributions to understanding how cell surface proteins mediate neural circuit specificity and plasticity. The Carrillo Lab, located at the University of Chicago, is a vibrant research group studying neural circuit formation in Drosophila. The lab team includes graduate students, postdocs, and technicians working collaboratively on projects related to neural wiring, synaptic plasticity, and circuit function. Dr. Carrillo is also actively involved in diversity and inclusion initiatives in neuroscience education and research.

• Tumor Microenvironment
• Extracellular Vesicles
• Exosomes
• +5 more

Nao Nishida serves as Assistant Professor at Waseda University's Institute for Advanced Study since 2022, following appointments at Tokyo Medical University and Fred Hutchinson Cancer Center. Her research bridges cancer biology and cell-cell communication mechanisms within tumor microenvironments. Her educational background includes: PhD in Agriculture (2014) from Kyoto University Master's in Applied Life Sciences (2011) from Kyoto University Bachelor's in Applied Life Sciences (2009) from Kyoto University Nishida's research centers on extracellular vesicle-mediated tumor-stroma crosstalk, with particular focus on exosome-driven metastasis, tumor-associated macrophage reprogramming, and organotypic culture modeling. She investigates how lipid composition and serine metabolism in cancer-derived EVs regulate microenvironmental remodeling and therapeutic resistance. Her work integrates advanced lipidomics, real-time tissue imaging, and spatial EV distribution analysis to uncover metastatic mechanisms. Analysis of her 17 publications reveals dominant themes in exosome biology (71% of works), cancer microenvironment dynamics (63%), and therapeutic targeting strategies (41%). Recent work increasingly incorporates spatial tissue context (2022-2024) and clinical translation potential. Her awards include: ISEV2025 New Parents Scholarship Japan Society for Promotion of Science Outstanding Researcher Candidate (2021) ISEV2017 Junior Member Scholarship Young Researcher Excellent Presentation Award (2015) Nishida directs multiple active grants including JSPS KAKENHI projects on EV secretion mechanisms (2023-2026) and nutritional stress adaptation (2025-2028), plus Mitsubishi Foundation and Uehara Memorial Foundation awards. She mentors through Waseda's bioscience curriculum while developing organotypic slice platforms for drug screening. Her laboratory utilizes advanced tumor slice culture systems and spatial EV mapping techniques to dissect microenvironmental heterogeneity, with current work focusing on stromal contribution to therapeutic resistance and organ-specific metastatic niches.

Jessica D. RosardaView profile

Assistant Professor

• Cellular Stress
• Signal Transduction
• Chemical Biology
• +5 more

Jessica D. Rosarda PhD is an Assistant Professor in the Department of Anatomy, Physiology and Genetics at the Uniformed Services University (USU) of the Health Sciences School of Medicine in Bethesda, MD. She leads a research laboratory focused on cellular stress mechanisms in military-relevant disorders. PhD in Chemical and Biological Sciences, The Scripps Research Institute (2023) MSc in Pharmacy, University of Florida (2013) BSc in Biology, Washington and Lee University (2010) Dr. Rosarda's research centers on cellular stress response pathways, particularly how cells respond to stress through signaling mechanisms that can either protect or damage tissues. Her work spans chemical biology, molecular medicine, neuroscience, and molecular/cell biology with a focus on proteostasis, unfolded protein response, and stress signaling dynamics in disease contexts. She investigates how imbalances in these pathways contribute to conditions ranging from retinal degeneration to traumatic brain injury, with particular relevance to military medicine. Analysis of Dr. Rosarda's publication record reveals a strong focus on stress response pathways, particularly the unfolded protein response and integrated stress response. Her work demonstrates how perturbations in proteostasis contribute to diverse pathologies including neurodegeneration, amyloidosis, and inflammatory conditions. She employs chemical biology approaches to develop therapeutic strategies targeting these pathways, with several publications on pharmacological modulators of stress responses. Dr. Rosarda maintains an active research program with numerous publications in high-impact journals including Nature Communications, Cell Chemical Biology, and ACS Chemical Biology. Her work frequently involves collaborations with the Wiseman laboratory and other researchers in the fields of proteostasis and stress response. Dr. Rosarda's laboratory at USU focuses on defining stress pathway signaling dynamics in military-relevant disorders, determining the metabolic factors that govern these stress responses, and identifying novel approaches for resolving toxic stress involved in both acute and chronic conditions.

Jane E. CavanaughView profile

Assistant Professor

• Neurobiology of Aging
• Cancer Biology
• Neurodegenerative Diseases
• +3 more

Jane E. Cavanaugh, Ph.D., is an Assistant Professor in the Division of Pharmaceutical Sciences at Duquesne University's School of Pharmacy and an Adjunct Assistant Professor in the Department of Pharmacology at the University of Pittsburgh. She received her Ph.D. in Pharmacology from Pennsylvania State College of Medicine (1997) and a B.A. in Chemistry from Franklin & Marshall College (1992). Her research focuses on the neurobiology and cancer biology of aging, specifically investigating intracellular signaling pathways (e.g., ERK5) that influence cell proliferation and survival in age-related pathologies. Key areas include: Neurodegenerative diseases (Alzheimer's, Parkinson's) Breast cancer mechanisms in aging Interventions using natural compounds (e.g., resveratrol) Motor deficit reversal strategies Her publications (1993–2011) predominantly explore neuroscience and pharmacology, with recurring themes in ERK signaling, neuroprotection, dopaminergic systems, and aging models. Recent work emphasizes therapeutic interventions for neurodegeneration and cancer. Awards & Honors: National Institute on Aging Career Award (K01) Travel Fellowship for Aging Research Graduate Commencement Speaker at Penn State Grants & Funding: She directs multiple projects, including a Duquesne University-funded study on intranasal L-Dopa delivery for CNS disorders and an NIA-supported investigation into ERK5's role in dopaminergic neuron survival. Other grants target breast cancer pathways and age-related motor deficits. Dr. Cavanaugh teaches courses in Human Physiology, Pathology, Pharmacology, and Toxicology for Duquesne's PharmD and graduate programs, and directs a laboratory focused on aging-related signaling pathways.

• Eukaryotic RNA processing
• Post-transcriptional gene regulation
• Spliceosome structure and mechanism
• +6 more

Melissa J. Moore, PhD, is Professor at the University of Massachusetts Chan Medical School, where she holds the Eleanor Eustis Farrington Chair of Cancer Research and serves in the RNA Therapeutics Institute. She also holds appointments in the T. H. Chan School of Medicine (Department of Chemical Biology) and the Morningside Graduate School of Biomedical Sciences (Departments of Biochemistry & Molecular Biotechnology, Interdisciplinary Graduate Program, and Translational Science). Additional affiliations include campus-wide programs in Bioinformatics & Integrative Biology and Chemical Biology. Education: BS in Chemistry/Biology, College of William and Mary PhD in Biological Chemistry, Massachusetts Institute of Technology Research Focus: Melissa Moore’s laboratory investigates post-transcriptional gene regulation in eukaryotes, with emphasis on three interconnected themes: (1) spliceosome structure and catalytic mechanism, (2) nuclear-to-cytoplasmic control of mRNA metabolism, and (3) quality control and clearance of defective ribosomal and messenger RNAs. The group combines biochemistry, single-molecule biophysics, RNA structural biology, and cell biology to dissect these processes at molecular and systems levels. Scientific Awards & Honors: Eleanor Eustis Farrington Chair of Cancer Research Funding & Collaborations: Work is supported by grants from the National Institutes of Health and involves ongoing collaborations with investigators at Brandeis University, MIT, University of Rochester, and other institutions. Rotation projects for graduate students are available in all active research areas. Laboratory & Team: The Moore laboratory is located in the RNA Therapeutics Institute at UMass Chan Medical School, 364 Plantation Street, Worcester, MA. The team employs state-of-the-art single-molecule imaging, mass spectrometry, and high-throughput sequencing to advance understanding of RNA biology and to translate insights into therapeutic RNA technologies.

John S. KaddisView profile

Associate Professor

• Type 1 Diabetes
• Systems Biology
• Bioinformatics
• +5 more

John S. Kaddis, Ph.D. is an Associate Professor in the Department of Diabetes & Cancer Discovery Science within the Arthur Riggs Diabetes & Metabolism Research Institute at City of Hope. With over 15 years of experience at City of Hope, Dr. Kaddis has developed scientific resources for team-based research initiatives focused on type 1 diabetes, creating data systems, analysis tools, and websites used by researchers worldwide. Dr. Kaddis earned his B.S. (Honors) in Biochemistry from California State University Los Angeles, where he completed undergraduate research supported by two National Science Foundation fellowships. He later earned his Ph.D. in Systems Biology and Disease from the University of Southern California. His academic journey includes positions as Assistant Professor since 2015 and Associate Professor since 2022 at City of Hope. Dr. Kaddis's research primarily focuses on the complex mechanisms of type 1 diabetes through systems biology and bioinformatics approaches. His work centers on understanding islet cell biology, beta cell dysfunction, and the immunological aspects of type 1 diabetes. His research has evolved from developing collaborative data resources to conducting cutting-edge investigations into genetic factors in diabetes pathogenesis. He has pioneered the creation of bioinformatics platforms that enable global collaboration in diabetes research. The trends in Dr. Kaddis's publications reveal a progression from foundational work on biobanking and data management toward increasingly sophisticated investigations of genetic and immunological mechanisms in type 1 diabetes. His recent work emphasizes translational applications, including potential therapeutic strategies and predictive models that span diverse ancestral backgrounds. His research bridges basic science with clinical applications through the Human Islet Research Network. Member, American Association for the Advancement of Science (2011-Present) Professional Member, American Diabetes Association (2010-Present) Member, Society of Clinical Research Associates (2003-2012) Lifetime Member, Tri-Beta Honor Society (2000-Present) Dr. Kaddis serves as principal investigator and co-investigator on multiple NIH and JDRF-funded projects, including the Human Islet Research Network Bioinformatics Center and the JDRF nPOD DataShare initiative. His work has established critical infrastructure for collaborative diabetes research, enabling researchers worldwide to access and analyze complex datasets. His laboratory has developed specialized data management systems that facilitate the sharing of islet cell research data across international boundaries while maintaining appropriate security and privacy protections. Dr. Kaddis's athletic pursuits in marathons and triathlons mirror his research approach—requiring stamina and perseverance to achieve long-term goals in diabetes research.

• Mitochondrial Biology
• Aging Research
• Genomic Instability
• +3 more

Sharath Anugula serves as a Guest Researcher in the Department of Cellular and Molecular Medicine at the University of Copenhagen's Faculty of Health and Medical Sciences, specifically within the Molecular Aging Program and Juel-Rasmussen Group. His research is centered at Panum Instituttet in Copenhagen. Dr. Anugula's research interests focus on the intricate relationship between mitochondrial function, genomic stability, and aging processes. His work bridges molecular biology, genetics, and systems approaches to understand fundamental mechanisms of biological aging. He has made significant contributions to understanding how DNA repair mechanisms, particularly REV1-mediated processes, interact with mitochondrial metabolism in aging contexts. His publication record demonstrates expertise in mitochondrial biology, with research examining how metabolic shifts, genomic instability, and nucleotide synthesis pathways influence aging trajectories and disease development. His work often employs both experimental and computational approaches to model complex biological systems. Anugula completed his Ph.D. at the University of Copenhagen in 2021 with research focused on nucleus-mitochondria communication in healthy aging. His scholarly output includes journal articles in Heliyon, Mitochondrion, and Experimental Gerontology, as well as book chapters on aging biology. His collaborative research network includes prominent scientists in aging research, particularly Professor Lene Juel Rasmussen, with whom he has published multiple studies examining the molecular underpinnings of aging processes.

Sandhya GanesanView profile

Assistant Professor

• Host-Pathogen Interaction
• Innate Immunity
• Molecular Biology
• +7 more

Dr. Sandhya Ganesan is an Assistant Professor at the School of Biology , Indian Institute of Science Education and Research Thiruvananthapuram (IISER-TVM) , where she established the Host-Pathogen Interaction Lab in January 2022. Her research focuses on mammalian molecular pathways that detect, respond to, and control bacterial pathogens like Coxiella burnetii , Legionella , and Candida albicans . Academic Rank: Assistant Professor University: Indian Institute of Science Education and Research Thiruvananthapuram (IISER-TVM) School: School of Biology Education: Ph.D. in Biomedical Sciences (Immunology and Microbiology Program), University of Massachusetts Chan Medical School (2014) B. Tech (with distinction) in Industrial Biotechnology, Alagappa College of Technology, Anna University (2007) Research Interests: She investigates host-pathogen interactions , innate immunity , and cell-intrinsic defense mechanisms , particularly how pathogens manipulate host immune surveillance. Her work includes studying inflammasome signaling, SNARE protein functions, and lysosomal biogenesis in pathogen control. Publication Trends: Her publications span immunology , molecular biology , and microbiology , emphasizing inflammasome pathways (e.g., NLRC4, AIM2, NLRP3), SNARE proteins (STX11), and IFNγ-induced host factors. Key subfields include pathogen-containing vacuole composition , lysosomal restriction , and immune dysregulation in disease . Scientific Awards: Milstein Abstract Award (2021) American Association of Immunologists (AAI) Trainee Abstract Award (2020) Professional Activities: She has peer-reviewed for journals like Bio-protocol , Infection and Immunity , and Scientific Reports , and organized the 2020 Intersections Science Fellows Symposium.

• Liver Immunology
• Immune Tolerance
• Transplant Immunology
• +5 more

Associate Professor Patrick Bertolino at the University of Sydney's Central Clinical School leads the Liver Immunology Program at the Centenary Institute. With 20+ years of international recognition in liver immunology , he pioneered discoveries about CD8+ T cell activation and hepatocyte-mediated immune tolerance , including the paradigm-shifting finding that liver-activated T cells are deleted via hepatocyte lysosomes. His work bridges transplantation immunology , chronic hepatitis , and malaria vaccine development using transgenic mouse models . Trained by immunology luminaries: Chantal Rabourdin-Combe (ENS-Lyon), Jacques Miller (WEHI), Barbara Fazekas de St Groth (Centenary Institute) Continuous NHMRC funding since 2000: 2 Program Grants, 6 Project Grants 2010 NHMRC Senior Research Fellowship Research Themes explore how the liver uniquely regulates immunity: Induces transplant tolerance via T cell deletion Exploits immune subversion in hepatitis B/C and malaria Develops gene therapy and mRNA vaccines for liver-targeted immunity Investigates hepatitis C reinfection post-transplant Article Trends show sustained focus on T cell-liver interactions , with recent work on mRNA vaccines (2023), neuroinflammation in GVHD (2024), and long-lived TRM cells in malaria (2025). Publications span high-impact immunology , hepatology , and transplantation journals . Awards include: 2010 NHMRC Senior Research Fellowship 2009 Centenary Institute student paper award 1999 Kevin Lafferty Prize for Autoimmunity 1997 University of Sydney U2000 Fellowship Teaching includes 3rd-year Immunology lectures (since 1998). Led liver immunology group since 2011, mentoring students and postdocs. Collaborates with Centenary Institute and National Hepatitis C Elimination Program .