Anna Ridley

Dr. Anna Ridley serves as a Lecturer in Immunology and Infection at the Blizard Institute within Queen Mary University of London's Faculty of Medicine and Dentistry. She teaches across multiple postgraduate programs including MSc Biomedical Science (Medical Microbiology), MSc Clinical Science (Infection Science), and the Nanchang University Joint Programme in Basic Immunology, while also tutoring Problem Based Learning for MBBS medical students.

Her educational journey includes:

• BSc in Biology from the University of Southampton
• MSc in Medical Immunology from King's College London
• DPhil from the University of Oxford investigating T cell phenotype in inflammatory arthritis
• PGCert in Teaching and Learning in Higher Education with HEA Fellowship

Dr. Ridley's research centers on dissecting T cell and neutrophil roles in inflammatory arthritis pathogenesis. She employs cellular immunology techniques to profile immune responses and screens AI-identified compounds for novel therapeutics, bridging molecular mechanisms with translational applications in autoimmune diseases.

Analysis of her publication record (2011-2021) reveals consistent focus on HLA-B27-associated immune dysregulation in spondyloarthritis, with increasing methodological sophistication from basic binding assays to single-cell genomic approaches. Her work demonstrates evolving emphasis on cellular heterogeneity, clonal dynamics, and AI-driven drug discovery within autoimmune rheumatology.

As an active educator and STEM ambassador, Dr. Ridley supervises postgraduate research projects and collaborates within the Centre for Immunobiology. Her teaching extends beyond campus through school engagement initiatives, reflecting commitment to science communication and student mentorship.

Embedded within the Blizard Institute's Centre for Immunobiology, she contributes to multidisciplinary teams investigating immune-mediated inflammatory diseases. Her laboratory work combines cellular phenotyping with functional assays to unravel neutrophil and T cell contributions to arthritis pathogenesis, supporting the institute's mission in translational biomedical research.