Amin Mottahedin

Amin Mottahedin serves as a Senior Research Fellow at the University of Oxford, working across Foster's lab at the Nuffield Department of Clinical Neurosciences and Buchan’s lab at the Radcliffe Department of Medicine. His research centers on circadian rhythm's impact on stroke-induced brain injury, specifically examining metabolic responses to ischemia and hypoxia.

His academic credentials include:

• MSc in Infection Biology from Uppsala University
• PhD in Medical Sciences from the University of Gothenburg (Mallard's lab)

Dr. Mottahedin's work spans circadian biology, stroke pathophysiology, and mitochondrial metabolism. His doctoral research investigated how infection/inflammation exacerbates hypoxic-ischemic brain injury in developing brains. During his Cambridge postdoc (Murphy's & Krieg's lab), he identified mitochondrial succinate metabolism as a therapeutic target for ischemic stroke. Current efforts employ chemical exchange-sensitive spin-lock MRI to map cerebral glucose metabolism post-stroke, revealing circadian-driven metabolic vulnerabilities.

His recent publication demonstrates innovative imaging approaches to quantify metabolic alterations in stroke models, emphasizing translational potential for time-sensitive interventions.

Key recognitions include:

• International Postdoc Fellowship from Swedish Research Council
• Fellow of the Higher Education Academy (FHEA)

Dr. Mottahedin secured competitive funding through the Swedish Research Council fellowship. No student advising records appear in source materials. He actively contributes to the Buchan Group (Laboratory of Cerebral Ischemia) and Foster's lab while affiliated with Corpus Christi College at Oxford.